scholarly journals Vgll2a is required for neural crest cell survival during zebrafish craniofacial development

2011 ◽  
Vol 357 (1) ◽  
pp. 269-281 ◽  
Author(s):  
Christopher W. Johnson ◽  
Laura Hernandez-Lagunas ◽  
Weiguo Feng ◽  
Vida Senkus Melvin ◽  
Trevor Williams ◽  
...  
Keyword(s):  
Neural Crest ◽  
Cell Survival ◽  
Neural Crest Cell ◽  
Craniofacial Development ◽  
Crest Cell

scholarly journals Vgl-2a is Required for Neural Crest Cell Survival During Zebrafish Craniofacial Development

2010 ◽  
Vol 344 (1) ◽  
pp. 447
Author(s):  
Christopher W. Johnson ◽  
Weiguo Feng ◽  
Trevor Williams ◽  
Kristin Artinger
Keyword(s):  
Neural Crest ◽  
Cell Survival ◽  
Neural Crest Cell ◽  
Craniofacial Development ◽  
Crest Cell

scholarly journals The ubiquitin ligase Nedd4 regulates craniofacial development by promoting cranial neural crest cell survival and stem-cell like properties

2013 ◽  
Vol 383 (2) ◽  
pp. 186-200 ◽  
Author(s):  
Sophie Wiszniak ◽  
Samuela Kabbara ◽  
Rachael Lumb ◽  
Michaela Scherer ◽  
Genevieve Secker ◽  
...  
Keyword(s):  
Stem Cell ◽  
Neural Crest ◽  
Cell Survival ◽  
Ubiquitin Ligase ◽  
Neural Crest Cell ◽  
Craniofacial Development ◽  
Cranial Neural Crest ◽  
Cranial Neural Crest Cell ◽  
Crest Cell

scholarly journals Corrigendum to “Vgll2a is required for neural crest cell survival during zebrafish craniofacial development” [Dev. Biol. 357 (2011) 269–281]

2012 ◽  
Vol 363 (1) ◽  
pp. 332
Author(s):  
Christopher W. Johnson ◽  
Laura Hernandez-Lagunas ◽  
Weiguo Feng ◽  
Vida Senkus Melvin ◽  
Trevor Williams ◽  
...  
Keyword(s):  
Neural Crest ◽  
Cell Survival ◽  
Neural Crest Cell ◽  
Craniofacial Development ◽  
Crest Cell

scholarly journals 03-P132 Xenopus HMGA2 is required for neural crest cell survival and migration

2009 ◽  
Vol 126 ◽  
pp. S106
Author(s):  
Simone Macrí ◽  
Marco Onorati ◽  
Guidalberto Manfioletti ◽  
Robert Vignali
Keyword(s):  
Neural Crest ◽  
Cell Survival ◽  
Neural Crest Cell ◽  
And Migration ◽  
Crest Cell

The flavonoids hesperidin and rutin promote neural crest cell survival

2012 ◽  
Vol 350 (2) ◽  
pp. 305-315 ◽  
Author(s):  
Jader Nones ◽  
Ana Paula Costa ◽  
Rodrigo Bainy Leal ◽  
Flávia Carvalho Alcantara Gomes ◽  
Andréa Gonçalves Trentin
Keyword(s):  
Neural Crest ◽  
Cell Survival ◽  
Neural Crest Cell ◽  
Crest Cell

scholarly journals Crispld2 is required for neural crest cell migration and cell viability during zebrafish craniofacial development

genesis ◽  
2015 ◽  
Vol 53 (10) ◽  
pp. 660-667 ◽  
Author(s):  
Eric C. Swindell ◽  
Qiuping Yuan ◽  
Lorena E. Maili ◽  
Bhavna Tandon ◽  
Daniel S. Wagner ◽  
...  
Keyword(s):  
Cell Migration ◽  
Cell Viability ◽  
Neural Crest ◽  
Neural Crest Cell ◽  
Craniofacial Development ◽  
Neural Crest Cell Migration ◽  
Crest Cell

Inactivation of the (β)-catenin gene by Wnt1-Cre-mediated deletion results in dramatic brain malformation and failure of craniofacial development

Development ◽  
2001 ◽  
Vol 128 (8) ◽  
pp. 1253-1264 ◽  
Author(s):  
V. Brault ◽  
R. Moore ◽  
S. Kutsch ◽  
M. Ishibashi ◽  
D.H. Rowitch ◽  
...  
Keyword(s):  
Neural Crest ◽  
Wnt Signaling Pathway ◽  
Neural Crest Cell ◽  
Craniofacial Development ◽  
Central Component ◽  
Regulatory Sequences ◽  
Brain Malformation ◽  
Cranial Ganglia ◽  
Crest Cell

('bgr;)-Catenin is a central component of both the cadherin-catenin cell adhesion complex and the Wnt signaling pathway. We have investigated the role of (β)-catenin during brain morphogenesis, by specifically inactivating the (β)-catenin gene in the region of Wnt1 expression. To achieve this, mice with a conditional ('floxed') allele of (β)-catenin with required exons flanked by loxP recombination sequences were intercrossed with transgenic mice that expressed Cre recombinase under control of Wnt1 regulatory sequences. (β)-catenin gene deletion resulted in dramatic brain malformation and failure of craniofacial development. Absence of part of the midbrain and all of the cerebellum is reminiscent of the conventional Wnt1 knockout (Wnt1(−)(/)(−)), suggesting that Wnt1 acts through (β)-catenin in controlling midbrain-hindbrain development. The craniofacial phenotype, not observed in embryos that lack Wnt1, indicates a role for (β)-catenin in the fate of neural crest cells. Analysis of neural tube explants shows that (β)-catenin is efficiently deleted in migrating neural crest cell precursors. This, together with an increased apoptosis in cells migrating to the cranial ganglia and in areas of prechondrogenic condensations, suggests that removal of (β)-catenin affects neural crest cell survival and/or differentiation. Our results demonstrate the pivotal role of (β)-catenin in morphogenetic processes during brain and craniofacial development.

Bmp5 Regulates Neural Crest Cell Survival and Proliferation via Two Different Signaling Pathways

Stem Cells ◽  
2016 ◽  
Vol 35 (4) ◽  
pp. 1003-1014 ◽  
Author(s):  
Hung-Yu Shih ◽  
Shu-Yuan Hsu ◽  
Pin Ouyang ◽  
Sheng-Jia Lin ◽  
Ting-Yun Chou ◽  
...  
Keyword(s):  
Neural Crest ◽  
Cell Survival ◽  
Signaling Pathways ◽  
Neural Crest Cell ◽  
Crest Cell ◽  
Cell Survival And Proliferation
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