Hydrologic uncertainty processor for probabilistic river stage forecasting: precipitation-dependent model

2001 ◽  
Vol 249 (1-4) ◽  
pp. 46-68 ◽  
Author(s):  
Roman Krzysztofowicz ◽  
Henry D Herr
Keyword(s):  
River Stage ◽  
Dependent Model ◽  
Hydrologic Uncertainty

Hydrologic uncertainty processor for probabilistic river stage forecasting

2000 ◽  
Vol 36 (11) ◽  
pp. 3265-3277 ◽  
Author(s):  
Roman Krzysztofowicz ◽  
Karen S. Kelly
Keyword(s):  
River Stage ◽  
Hydrologic Uncertainty

Communication: Photoinduced Carbon Dioxide Binding with Surface-Functionalized Silicon Quantum Dots

2018 ◽  
Author(s):  
Oscar A. Douglas-Gallardo ◽  
Cristián Gabriel Sánchez ◽  
Esteban Vöhringer-Martinez
Keyword(s):  
Carbon Dioxide ◽  
Quantum Dots ◽  
Atmospheric Carbon Dioxide ◽  
Density Functional ◽  
Tight Binding ◽  
Carbon Dioxide Concentration ◽  
Research Area ◽  
Silicon Quantum Dots ◽  
Dependent Model ◽  
Photoinduced Charge

<div> <div> <div> <p>Nowadays, the search of efficient methods able to reduce the high atmospheric carbon dioxide concentration has turned into a very dynamic research area. Several environmental problems have been closely associated with the high atmospheric level of this greenhouse gas. Here, a novel system based on the use of surface-functionalized silicon quantum dots (sf -SiQDs) is theoretically proposed as a versatile device to bind carbon dioxide. Within this approach, carbon dioxide trapping is modulated by a photoinduced charge redistribution between the capping molecule and the silicon quantum dots (SiQDs). Chemical and electronic properties of the proposed SiQDs have been studied with Density Functional Theory (DFT) and Density Functional Tight-Binding (DFTB) approach along with a Time-Dependent model based on the DFTB (TD-DFTB) framework. To the best of our knowledge, this is the first report that proposes and explores the potential application of a versatile and friendly device based on the use of sf -SiQDs for photochemically activated carbon dioxide fixation. </p> </div> </div> </div>

Towards a Trajectory-Dependent Model of (Human) Rational Agency

2018 ◽  
Author(s):  
Karen Jones
Keyword(s):  
Regulatory Role ◽  
Rational Agency ◽  
Dependent Model ◽  
Reliable Path

This chapter addresses the question, “What is the role and authority of conscious deliberation and judgment in human rational agency?” Anti-rationalists claim that the rationalist account of its role and authority is mistaken: conscious deliberation and judgment plays a relatively small part in our practical lives, can be used in the service of rationalizing bullshit, and is not the only or necessarily the most reliable path of access to our reasons. Against the anti-rationalist, the chapter argues that their critique rests on an analogy between the authority of judgment and the authority of an expert, when the rationalist models judgment’s authority on that of a judge. Against the traditional rationalist, the chapter argues the judge model fails. The chapter explores a third model—the monitor model—which, like rationalism, gives our reflective capacities a significant regulatory role, but accommodates the anti-rationalist emphasis on emotion and fast non-deliberative action.

scholarly journals ZBP1 promotes LPS-induced cell death and IL-1β release via RHIM-mediated interactions with RIPK1

2021 ◽  
Vol 12 (1) ◽  
Author(s):  
Hayley I. Muendlein ◽  
Wilson M. Connolly ◽  
Zoie Magri ◽  
Irina Smirnova ◽  
Vladimir Ilyukha ◽  
...  
Keyword(s):  
Immune Response ◽  
Cell Death ◽  
Complex Formation ◽  
Complex I ◽  
Yersinia Pseudotuberculosis ◽  
Caspase 8 ◽  
Inflammasome Activation ◽  
Inflammatory Signaling ◽  
Complex Ii ◽  
Dependent Model

AbstractInflammation and cell death are closely linked arms of the host immune response to infection, which when carefully balanced ensure host survival. One example of this balance is the tightly regulated transition from TNFR1-associated pro-inflammatory complex I to pro-death complex II. By contrast, here we show that a TRIF-dependent complex containing FADD, RIPK1 and caspase-8 (that we have termed the TRIFosome) mediates cell death in response to Yersinia pseudotuberculosis and LPS. Furthermore, we show that constitutive binding between ZBP1 and RIPK1 is essential for the initiation of TRIFosome interactions, caspase-8-mediated cell death and inflammasome activation, thus positioning ZBP1 as an effector of cell death in the context of bacterial blockade of pro-inflammatory signaling. Additionally, our findings offer an alternative to the TNFR1-dependent model of complex II assembly, by demonstrating pro-death complex formation reliant on TRIF signaling.

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