Defence signalling pathways in cereals

1999 ◽  
Vol 2 (4) ◽  
pp. 295-300 ◽  
Author(s):  
Pietro Piffanelli ◽  
Alessandra Devoto ◽  
Paul Schulze-Lefert
2011 ◽  
Vol 12 (8) ◽  
pp. 746-758 ◽  
Author(s):  
KATY M. CHRISTIANSEN ◽  
YANGNAN GU ◽  
NATALIE RODIBAUGH ◽  
ROGER W. INNES

2006 ◽  
Vol 171 (2) ◽  
pp. 249-269 ◽  
Author(s):  
David Lecourieux ◽  
Raoul Ranjeva ◽  
Alain Pugin

2006 ◽  
Vol 49 (2) ◽  
pp. 302-312 ◽  
Author(s):  
Ziguo Zhang ◽  
Angela Feechan ◽  
Carsten Pedersen ◽  
Mari-Anne Newman ◽  
Jin-long Qiu ◽  
...  

2005 ◽  
Vol 32 (1) ◽  
pp. 1 ◽  
Author(s):  
Louise F. Thatcher ◽  
Jonathan P. Anderson ◽  
Karam B. Singh

To overcome the attack of invading pathogens, a plant’s defence system relies on preformed and induced responses. The induced responses are activated following detection of a pathogen, with the subsequent transmission of signals and orchestrated cellular events aimed at eliminating the pathogen and preventing its spread. Numerous studies are proving that the activated signalling pathways are not simply linear, but rather, form complex networks where considerable cross talk takes place. This review covers the recent application of powerful genetic and genomic approaches to identify key defence signalling pathways in the model plant Arabidopsis thaliana (L.) Heynh. The identification of key regulatory components of these pathways may offer new approaches to increase the defence capabilities of crop plants.


2014 ◽  
Vol 65 (12) ◽  
pp. 3289-3298 ◽  
Author(s):  
Jianing Wei ◽  
Joop J. A. van Loon ◽  
Rieta Gols ◽  
Tila R. Menzel ◽  
Na Li ◽  
...  

2005 ◽  
Vol 41 (5) ◽  
pp. 673-684 ◽  
Author(s):  
Katherine J. Denby ◽  
Laure J.M. Jason ◽  
Shane L. Murray ◽  
Robert L. Last

2013 ◽  
Vol 32 (2) ◽  
pp. 342-356 ◽  
Author(s):  
Leila Eshraghi ◽  
Jonathan P. Anderson ◽  
Nader Aryamanesh ◽  
Jen A. McComb ◽  
Bryan Shearer ◽  
...  

2002 ◽  
Vol 38 ◽  
pp. 9-19 ◽  
Author(s):  
Guy S Salvesen

The ability of metazoan cells to undergo programmed cell death is vital to both the precise development and long-term survival of the mature adult. Cell deaths that result from engagement of this programme end in apoptosis, the ordered dismantling of the cell that results in its 'silent' demise, in which packaged cell fragments are removed by phagocytosis. This co-ordinated demise is mediated by members of a family of cysteine proteases known as caspases, whose activation follows characteristic apoptotic stimuli, and whose substrates include many proteins, the limited cleavage of which causes the characteristic morphology of apoptosis. In vertebrates, a subset of caspases has evolved to participate in the activation of pro-inflammatory cytokines, and thus members of the caspase family participate in one of two very distinct intracellular signalling pathways.


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