scholarly journals High expression of RelA/p65 is associated with activation of nuclear factor-κB-dependent signaling in pancreatic cancer and marks a patient population with poor prognosis

2007 ◽  
Vol 97 (4) ◽  
pp. 523-530 ◽  
Author(s):  
W Weichert ◽  
M Boehm ◽  
V Gekeler ◽  
M Bahra ◽  
J Langrehr ◽  
...  
Keyword(s):  
Pancreatic Cancer ◽  
Patient Population ◽  
Poor Prognosis ◽  
Nuclear Factor Κb ◽  
High Expression ◽  
Nuclear Factor

Nuclear Expression of Glioma-Associated Oncogene Homolog 1 and Nuclear Factor-κB Is Associated with a Poor Prognosis of Pancreatic Cancer

Oncology ◽  
2013 ◽  
Vol 85 (2) ◽  
pp. 86-94 ◽  
Author(s):  
Shih-Hung Yang ◽  
Chih-Hung Hsu ◽  
Jen-Chieh Lee ◽  
Yu-Wen Tien ◽  
Sung-Hsin Kuo ◽  
...  
Keyword(s):  
Pancreatic Cancer ◽  
Poor Prognosis ◽  
Nuclear Factor Κb ◽  
Nuclear Factor ◽  
Nuclear Expression

A genetic variant conferred high expression of CAV2 promotes pancreatic cancer progression and associates with poor prognosis

2021 ◽  
Vol 151 ◽  
pp. 94-105
Author(s):  
Ying Zhu ◽  
Jianbo Tian ◽  
Xiating Peng ◽  
Xiaoyang Wang ◽  
Nan Yang ◽  
...  
Keyword(s):  
Pancreatic Cancer ◽  
Cancer Progression ◽  
Poor Prognosis ◽  
Genetic Variant ◽  
High Expression

Abstract 1766: High expression of ARHGEF2 is associated with poor prognosis via c-Myc signaling in patients with pancreatic cancer

2019 ◽  
Author(s):  
Yosuke Nakao ◽  
Shigeki Nakagawa ◽  
Yo-ichi Yamashita ◽  
Rumi Itoyama ◽  
Toshihiko Yusa ◽  
...  
Keyword(s):  
Pancreatic Cancer ◽  
Poor Prognosis ◽  
High Expression

scholarly journals Macrophages in pancreatic cancer: Starting things off on the wrong track

2013 ◽  
Vol 202 (3) ◽  
pp. 403-405
Author(s):  
Xavier Deschênes-Simard ◽  
Yusuke Mizukami ◽  
Nabeel Bardeesy
Keyword(s):  
Pancreatic Cancer ◽  
Chronic Inflammation ◽  
Acinar Cells ◽  
Nuclear Factor Κb ◽  
Pancreatic Acinar Cells ◽  
Nuclear Factor ◽  
Oncogenic Transformation ◽  
Transcriptional Program ◽  
Cell Biol ◽  
Inflammatory Macrophages

Chronic inflammation drives initiation and progression of many malignancies, including pancreatic cancer. In this issue, Liou et al. (2013. J. Cell Biol. http://dx.doi.org/10.1083/jcb.201301001) report that inflammatory macrophages are major players in the earliest stages of pancreatic cancer. They show that paracrine signals from the macrophages activate the nuclear factor κB transcriptional program in normal pancreatic acinar cells, resulting in acinar–ductal metaplasia, a dedifferentiated state that is poised for oncogenic transformation.

Combination Paclitaxel and Inhibitor of Nuclear Factor κB Activation Improves Therapeutic Outcome for Model Mice With Peritoneal Dissemination of Pancreatic Cancer

Pancreas ◽  
2011 ◽  
Vol 40 (4) ◽  
pp. 600-607 ◽  
Author(s):  
Yuki Fujiwara ◽  
Kenei Furukawa ◽  
Yohta Shimada ◽  
Tomonori Iida ◽  
Hiroaki Shiba ◽  
...  
Keyword(s):  
Pancreatic Cancer ◽  
Peritoneal Dissemination ◽  
Nuclear Factor Κb ◽  
Therapeutic Outcome ◽  
Nuclear Factor
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