Motor and Sensory Aspects of Concomitant Strabismus

1958 ◽  
Vol 8 (1) ◽  
pp. 136-141
Author(s):  
Vera Parry
1992 ◽  
Vol 55 ◽  
pp. 114
Author(s):  
B. Bagolini ◽  
V. Porciatti ◽  
B. Falsini ◽  
K. Dickmann ◽  
G. Porrello ◽  
...  

Scientifica ◽  
2016 ◽  
Vol 2016 ◽  
pp. 1-8 ◽  
Author(s):  
Costantino Schiavi

The role played by the extraocular muscles (EOMs) in the etiology of concomitant infantile strabismus is still debated and it has not yet definitively established if the sensory anomalies in concomitant strabismus are a consequence or a primary cause of the deviation. The commonest theory supposes that most strabismus results from abnormal innervation of the EOMs, but the cause of this dysfunction and its origin, whether central or peripheral, are still unknown. The interaction between sensory factors and innervational factors, that is, esotonus, accommodation, convergence, divergence, and vestibular reflexes in visually immature infants with family predisposition, is suspected to create conditions that prevent binocular alignment from stabilizing and strengthening. Some role in the onset of fixation instability and infantile strabismus could be played by the feedback control of eye movements and by dysfunction of eye muscle proprioception during the critical period of development of the visual sensory system. A possible role in the onset, maintenance, or worsening of the deviation of abnormalities of muscle force which have their clinical equivalent in eye muscle overaction and underaction has been investigated under either isometric or isotonic conditions, and in essence no significant anomalies of muscle force have been found in concomitant strabismus.


2016 ◽  
Vol 6 (1) ◽  
Author(s):  
Shu Min Tang ◽  
Rachel Y. T. Chan ◽  
Shi Bin Lin ◽  
Shi Song Rong ◽  
Henry H. W. Lau ◽  
...  

Author(s):  
Rebecca Ford ◽  
Moneesh Patel

The chapter begins by discussing the anatomy and actions of the extraocular muscles and central control of ocular motility, before covering the key clinical skills, namely patient assessment, assessment of ocular movements, visual acuity testing, tests of stereopsis and binocular single vision, tests of retinal correspondence and suppression, and Hess charts. It then covers the key areas of clinical knowledge, including amblyopia, binocular vision and stereopsis, concomitant strabismus, incomitant strabismus, restrictive ocular motility disorders, complex ocular motility syndromes, vertical deviations , and alphabet patterns, and the key practical skills, namely the principles of strabismus surgery and other procedures in strabismus. The chapter concludes with five case-based discussions, on myopic anisometropia, esotropia, infantile esotropia, orbital floor fracture, and consecutive exotropia.


2020 ◽  
Vol 26 (1) ◽  
pp. 16-20
Author(s):  
Valentina A. Usenko ◽  
Aizhamal I. Berdibaeva

Aim. To study the state of the accommodative ability of amblyopia before and after treatment. Materials and methods. One hundred patients (158 eyes) with friendly alternating and monolateral strabismus were examined. The control group consisted of 10 healthy individuals (20 eyes) with emmetropia: the average age of patients under 3 years of age was 2.4 0.15 years, from 3 years to 7 years, 4.8 0.07 years, and above 7 years, 8.3 0.4 years. The vast majority of patients (84 people, 126 eyes) had hyperopic astigmatism, and 16 patients (32 eyes) had a high degree of hyperopia. Twenty-eight patients (40 eyes), 48 patients (84 eyes), and 20 patients (26 eyes) had amblyopia of the 1st, 2nd, and 3rd degrees, respectively. Along with generally accepted methods for examining eyes, the OAA volume and ZAA were determined as the absolute accommodation margin by the proximetric method (AKA-0.1) before and after hardware treatment to relax and stimulate the ciliary muscle. Results. The results after treatment revealed a 1.52-fold increase in OAA to 14.4 0.23 D; 13.5 0.10 D; 13.1 0.41 D; and ZAA and a significant 2-fold increase in visual acuity (P 0.001). Conclusion. A criterion for the effectiveness of hardware treatment for dysbinocular amblyopia is an increase in OAA and ZAA, accompanied by a 2-fold increase in distance visual acuity (P 0.001).


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