Event Centrality, Posttraumatic Cognitions, and the Experience of Posttraumatic Growth

2013 ◽  
Vol 22 (4) ◽  
pp. 379-393 ◽  
Author(s):  
Steven L. Lancaster ◽  
Megan Kloep ◽  
Benjamin F. Rodriguez ◽  
Rebecca Weston
2010 ◽  
Vol 25 (5) ◽  
pp. 817-822 ◽  
Author(s):  
Adriel Boals ◽  
Darnell Schuettler

2021 ◽  
pp. 003022282110515
Author(s):  
Angela P. Ryan ◽  
Jennifer Ripley

Multiple factors have been studied to determine their relationship to posttraumatic growth, with findings suggesting the importance of resiliency (Bensimon, 2012), social support (Tedeschi & Calhoun, 2004), event centrality (Taku et al., 2014), religious coping (Pargament et al., 2006), relationship to the deceased (Oginska–Bulik, 2015), and the circumstance of passing (Feigelman et al., 2009). This study investigated the relationship of these identified factors with posttraumatic growth in a sample of 165 bereaved individuals. Regression results indicated that the overall model predicted scores on posttraumatic growth ( R 2 = .473, R 2 adj = .456, F (5, 159) = 28.51, p < .001), with most robust effects found in social support ( β = .281), religious commitment ( β = .289), and event centrality ( β = .410). This indicates bereavement experiences can result in significant growth in individuals, particularly those who seek social support, identify the event as an essential part of their experience, and identify religion as an important aspect of their routine.


2010 ◽  
Vol 15 (6) ◽  
pp. 518-533 ◽  
Author(s):  
Adriel Boals ◽  
Jennifer M. Steward ◽  
Darnell Schuettler

Author(s):  
Helen Johanßen ◽  
Nikola Schoofs ◽  
Reinhold Kliegl ◽  
Felix Bermpohl ◽  
Dominik Ülsmann ◽  
...  

Abstract Background The centrality of an event indicates the extent to which it becomes a core part of identity and life story. Event centrality (EC) has been shown to have a strong relationship with PTSD symptoms, which seems to be indirectly influenced by negative posttraumatic cognitions (PTC). However, research on this potential mediation and its causal links particularly with clinical samples is limited and essential to derive treatment implications. Methods Pre- and posttreatment data of 103 day-unit patients with PTSD was examined using mediation analyses and structural equation modeling. Results Negative PTC mediated the relationship between EC and PTSD symptoms, partially pre- and completely posttreatment. Within extended longitudinal analyses causal directions of the mediation pathways were not adequately interpretable due to unexpected suppression effects. Conclusions The results suggest that EC may only have an indirect effect on PTSD symptoms through negative PTC. Thus, decreasing negative PTC which are connected to centralized events might be a key element for PTSD treatment. Thereby, transforming the cognitions’ valence to more positive and constructive forms could be crucial rather than mere decentralization. Although suppression effects limited causal inferences, they do not contradict the mediation and further indicate potential interactional terms and a transformation of EC.


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