Compressive Optic Neuropathy

2011 ◽  
pp. 21-24
Author(s):  
Matthew J. Thurtell ◽  
Robert L. Tomsak ◽  
Robert B. Daroff

Optic nerve compression results in progressive, and often painless, monocular vision loss. In this chapter, we review the clinical signs and common causes of compressive optic neuropathy. We discuss in more detail the imaging characteristics and management of optic nerve sheath meningioma.

2019 ◽  
pp. 21-24
Author(s):  
Matthew J. Thurtell ◽  
Robert L. Tomsak

Optic nerve compression results in progressive, and often painless, vision loss. In this chapter, we begin by reviewing the clinical features of anterior and posterior compressive optic neuropathy. We next review the common causes of compressive optic neuropathy, which include orbital tumors (e.g., optic nerve sheath meningioma, optic glioma, and capillary hemangioma), orbital infection, orbital inflammation, intracranial tumors (e.g., pituitary macroadenoma, meningioma, and craniopharyngioma), aneurysm, and thyroid eye disease. We then review the workup for compressive optic neuropathy and discuss the various imaging options. Lastly, we discuss the clinical features, imaging findings, management options, and prognosis for visual recovery for patients with optic nerve sheath meningioma.


2012 ◽  
Vol 2012 ◽  
pp. 1-3
Author(s):  
Mohammed M. Ziaei ◽  
Hadi Ziaei

Purpose. To present a unique case of Non-Hodgkin’s-Lymphoma- (NHL) associated compressive optic neuropathy.Method. An 89-year-old male presenting with acute unilateral visual loss and headache.Results. Patient was initially diagnosed with occult giant cell arteritis; however after visual acuity deteriorated despite normal inflammatory markers, an urgent MRI scan revealed an extensive paranasal sinus mass compressing the optic nerve.Conclusion. Paranasal sinus malignancies occasionally present to the ophthalmologist with signs of optic nerve compression and must be included in the differential diagnosis of acute visual loss.


2019 ◽  
Vol 3 (4) ◽  
pp. 436-437
Author(s):  
David Lane ◽  
Kaila Pomeranz ◽  
Shannon Findlay ◽  
Daniel Miller

A 62-year-old woman with a history of metastatic breast cancer and known meningioma presented with unilateral vision loss associated with anisocoria and an afferent pupillary defect. On magnetic resonance imaging we found the cause to be optic nerve compression by a right frontal meningioma. Monocular vision-loss etiologies are anatomically localized to structures anterior to the optic chiasm. This case serves as a reminder that cerebral structures in this location must not be forgotten in the differential.


2013 ◽  
Vol 2013 ◽  
pp. 1-3
Author(s):  
R. Parab ◽  
C. I. Fung ◽  
Gerrit Van Der Merwe

Traumatic optic neuropathy is an uncommon, yet serious, result of facial trauma. The authors present a novel case of a 59-year-old gentleman who presented with an isolated blunt traumatic left optic nerve hematoma causing vision loss. There were no other injuries or fractures to report. This case highlights the importance of early recognition of this rare injury and reviews the current literature and management of traumatic optic neuropathy.


PLoS ONE ◽  
2014 ◽  
Vol 9 (11) ◽  
pp. e112403 ◽  
Author(s):  
Masayuki Hata ◽  
Kazuaki Miyamoto ◽  
Akio Oishi ◽  
Yukiko Makiyama ◽  
Norimoto Gotoh ◽  
...  

2003 ◽  
Vol 128 (2) ◽  
pp. 228-235 ◽  
Author(s):  
Kacker Ashutosh ◽  
Kazim Michael ◽  
Murphy Mark ◽  
Trokel Stephen ◽  
G. Close Lanny

BACKGROUND: Graves' orbitopathy can produce proptosis, compressive optic neuropathy, and extraocular motility abnormalities; symptoms result from an increase in orbital volume due to expansion of intraorbital fat, with or without extraocular muscle involvement. STUDY DESIGN: We conducted a chart review of patients who underwent orbital decompression to treat Graves' orbitopathy. Twenty-two orbits (13 patients) underwent orbital bone decompression, of which 17 orbits (9 patients) underwent the combined medial and lateral “balanced” decompression. RESULTS: All patients had significant improvement with an average decrease in proptosis of 5.9 mm in the balanced decompression group. Restoration of normal optic nerve function was achieved in all patients with compressive optic neuropathy. Diplopia was noted in 4 patients (30.7%) preoperatively. Two patients had new postoperative diplopia (15.35%). CONCLUSION: In our experience, “balanced” decompression results in a reduction of proptosis and improved optic nerve function and has a low incidence of complications.


Orbit ◽  
2019 ◽  
Vol 39 (6) ◽  
pp. 455-455
Author(s):  
Jin Kyun Oh ◽  
Davinder K. Chandhoke ◽  
Roman Shinder

2021 ◽  
pp. 112067212110491
Author(s):  
Ali Nouraeinejad

Radiation-induced optic neuropathy (RION) is a late complication of radiation therapy for brain and skull base tumors. RION leads to the devastating total vision loss in one or both eyes. Therefore, the early detection of RION is vital. Since visual symptoms and clinical signs of RION are not present at early stages of the radiation injury, it is essential to apply a diagnostic test to detect RION as early as possible in order to start therapeutic interventions. The author proposes to apply visual evoked potential (VEP) as a diagnostic test in the interval time after radiation therapy.


2016 ◽  
Vol 7 (4) ◽  
pp. ar.2016.7.0178 ◽  
Author(s):  
Wendy Yen Nee See ◽  
Kala Sumugam ◽  
Visvaraja Subrayan

Background To report an unusual case of compressive optic neuropathy secondary to a large onodi air cell. Method Case report. Results A 50 year-old gentlemen presented to the eye clinic with left eye painless loss of vision for one day. Visual acuity was counting finger in the left eye with a positive relative afferent pupillary defect (RAPD). Dilated left fundus examination revealed a pale optic disc. A computed tomography of orbit and brain showed a large left sphenoid sinus with onodi-cell-like projection on the left superior margin of left optic canal impinging on the left optic nerve. He was referred to the otorhinolaryngology team and subsequently underwent left optic nerve decompression. Post-operatively, his left visual acuity improved to 6/60 with reversal of RAPD. Conclusion There are many causes of optic neuropathy and compressive optic neuropathy due to large onodi air cell is uncommon. Acute unilateral loss of vision heralds from a multitude of sinister causes and junior residents should be vigilant that onodi air cell pneumotisation could be one of them.


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