VENTILATORY THRESHOLD ESTABLISHES THE MAXIMAL CARDIOVASCULAR STEADY-STATE FOR EXERCISING CARDIAC PATIENTS

1999 ◽  
Vol 31 (Supplement) ◽  
pp. S40
Author(s):  
S. Melvin ◽  
W. G. Herbert ◽  
L. Pierson ◽  
P. Griffith ◽  
J. Cook ◽  
...  
2021 ◽  
Vol 28 (Supplement_1) ◽  
Author(s):  
F Anselmi ◽  
L Cavigli ◽  
A Pagliaro ◽  
S Valente ◽  
F Valentini ◽  
...  

Abstract Funding Acknowledgements Type of funding sources: None. Background. Although structured exercise training is strongly recommended in cardiac patients, uncertainties exist about the methods for determining exercise intensity (EI) and their correspondence with effective EI obtained by ventilatory thresholds. We aimed to determine the first (VT1) and second ventilatory threshold (VT2) in cardiac patients, sedentary subjects and athletes comparing VT1 and VT2 with EI defined by recommendations. Methods. We prospectively enrolled 350 subjects (mean age: 50.7 ± 12.9 years; 167 cardiac patients, 150 healthy sedentary subjects, 33 competitive endurance athletes). Each subject underwent ECG, echocardiography, and cardiopulmonary exercise testing. The percentages of peak VO2, peak heart rate (HR), and HR reserve were obtained at VT1 and VT2, and compared with EI definition proposed by the recommendations. Results. VO2 at VT1 corresponded to high rather than moderate EI in 67.1% and in 79.6% of cardiac patients, applying the definition of moderate exercise by the previous recommendations and the 2020 guidelines, respectively. Most of cardiac patients had VO2 values at VT2 corresponding to very-high rather than high EI (59.9% and 50.3%, by previous recommendations and 2020 guidelines, respectively). A better correspondence between ventilatory-thresholds and recommended EI domains was observed in healthy subjects and in athletes (90% and 93.9%, respectively). Conclusions. EI definition based on percentages of peak HR and peak VO2 may misclassify the effective EI and the discrepancy between the individually determined and the recommended EI is particularly relevant in cardiac patients. A ventilatory threshold-based rather than a range-based approach is advisable in order to define an appropriate level of EI. Abstract Figure.


1983 ◽  
Vol 15 (2) ◽  
pp. 91
Author(s):  
H. S. Miller ◽  
W. T. Boone ◽  
T. L. Glover ◽  
P. M. Ribisl

1991 ◽  
Vol 63 (1) ◽  
pp. 55-59 ◽  
Author(s):  
Yoshiharu Yamamoto ◽  
Mitsumasa Miyashita ◽  
Richard L. Hughson ◽  
Shin-ichi Tamura ◽  
Minoru Shinohara ◽  
...  

2006 ◽  
Vol 1 (3) ◽  
pp. 222-232 ◽  
Author(s):  
Oliver Faude ◽  
Tim Meyer ◽  
Wilfried Kindermann

Purpose:The work rate (WR) corresponding to ventilatory threshold (VT) is an appropriate intensity for regenerative and low-intensity training sessions. During incremental ramp exercise, VO2 increase lags behind WR increase. Traditionally, a VO2 time delay (td) of 45 seconds is used to calculate the WR at VT from such tests. Considerable inaccuracies were observed when using this constant td. Therefore, this study aimed at reinvestigating the temporal relationship between VO2 and WR at VT.Methods:20 subjects (VO2peak 49.9 to 72.6 mL · min–1 · kg–1) performed a ramp test in order to determine VT and a subsequent steady-state test during which WR was adjusted to elicit the VO2 corresponding to VT. The difference in WR and heart rate at VT was calculated between the ramp and the steady-state test (WRdiff, HRdiff) as well as the time delay corresponding to WRdiff during ramp exercise.Results:Mean values were td = 85 ± 26 seconds (range 38 to 144), WRdiff = 45 ± 12 W (range 23 to 67), HRdiff = 1 ± 9 beats/min (range –21 to +15). The limits of agreement for the difference between WR at VT during ramp and steady-state exercise were ± 24 W. No signifi cant influence on td, WRdiff, or HRdiff from differences in endurance capacity (VO2peak and VT; P > .10 for all correlations) or ramp increment (P = .26, .49, and .85, respectively) were observed.Conclusion:The wide ranges of td, WRdiff, and HRdiff prevent the derivation of exact training guidelines from single-ramp tests. It is advisable to perform a steady-state test to exactly determine the WR corresponding to VT.


1999 ◽  
Vol 86 (5) ◽  
pp. 1544-1551 ◽  
Author(s):  
Barry W. Scheuermann ◽  
John M. Kowalchuk ◽  
Donald H. Paterson ◽  
David A. Cunningham

The effect of carbonic anhydrase inhibition with acetazolamide (Acz, 10 mg/kg) on the ventilatory response to an abrupt switch into hyperoxia (end-tidal [Formula: see text]= 450 Torr) and hypoxia (end-tidal[Formula: see text] = 50 Torr) was examined in five male subjects [30 ± 3 (SE) yr]. Subjects exercised at a work rate chosen to elicit an O2 uptake equivalent to 80% of the ventilatory threshold. Ventilation (V˙e) was measured breath by breath. Arterial oxyhemoglobin saturation (%[Formula: see text]) was determined by ear oximetry. After the switch into hyperoxia, V˙eremained unchanged from the steady-state exercise prehyperoxic value (60.6 ± 6.5 l/min) during Acz. During control studies (Con),V˙e decreased from the prehyperoxic value (52.4 ± 5.5 l/min) by ∼20% (V˙enadir = 42.4 ± 6.3 l/min) within 20 s after the switch into hyperoxia. V˙e increased during Acz and Con after the switch into hypoxia; the hypoxic ventilatory response was significantly lower after Acz compared with Con [Acz, change (Δ) inV˙e/[Formula: see text]= 1.54 ± 0.10 l ⋅ min−1 ⋅ [Formula: see text] −1; Con, ΔV˙e/[Formula: see text]= 2.22 ± 0.28 l ⋅ min−1 ⋅ [Formula: see text] −1]. The peripheral chemoreceptor contribution to the ventilatory drive after acute Acz-induced carbonic anhydrase inhibition is not apparent in the steady state of moderate-intensity exercise. However, Acz administration did not completely attenuate the peripheral chemoreceptor response to hypoxia.


2004 ◽  
Vol 91 (2-3) ◽  
pp. 224-229 ◽  
Author(s):  
Kazuto Omiya ◽  
Haruki Itoh ◽  
Naomi Harada ◽  
Tomoko Maeda ◽  
Akihiko Tajima ◽  
...  

2007 ◽  
Vol 101 (5) ◽  
pp. 547-554 ◽  
Author(s):  
Dominique Hansen ◽  
Paul Dendale ◽  
Jan Berger ◽  
Romain Meeusen

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