Effects of environmental tobacco smoke on urinary cotinine excretion in nonsmokerss Evidence for passive smoking

1985 ◽  
Vol 76 (5) ◽  
pp. 815
Author(s):  
S. Matsukura
Keyword(s):  
Environmental Tobacco Smoke ◽  
Tobacco Smoke ◽  
Passive Smoking ◽  
Urinary Cotinine

Passive smoking among adolescents in Estonia

1999 ◽  
Vol 18 (4) ◽  
pp. 229-232 ◽  
Author(s):  
Kersti Piirna
Keyword(s):  
Cigarette Smoke ◽  
Environmental Tobacco Smoke ◽  
Tobacco Smoke ◽  
Passive Smoking ◽  
Population Sample ◽  
Age Groups ◽  
Active Smoking ◽  
Adolescent Smokers ◽  
Open Questions ◽  
Considerable Impact

1 The objective ofthis study was to assess prevalence and patterns of passive smoking in Estonia and to describe how environmental tobacco smoke is likely to initiate the desire in adolescents to start active smoking. 2 A population sample of 2170 students within the age groups of 13-14,15-16 and 17-18 of school grades 8, 10 and 12, respectively, at Tallinn High Schools participated in this study. The data w,ere collected using questionnaires which mainly consisted of structured multiple-choice and a few open questions. 3 Every fourth adolescent in Tallinn was either an occasional or a habitual smoker. 4 Nearly half (48%) of the respondents had no daily exposure to cigarette smoke. Almost one third (31%) was exposed daily for less than 1 h, 15% for 1-5 h and 6% for over 5 h a day. 5 The number of adolescent smokers was higher than non-smokers among those daily inhaling tobacco sidestream smoke. 6 In conclusion, environment gives considerable impact to the development of smoking habits among adolescents. Passive smoking as an inducing factor is associated with higher prevalence of smoking among the adolescents studied.

scholarly journals Self-Reported Exposure to ETS (Environmental Tobacco Smoke), Urinary Cotinine, and Oxidative Stress Parameters in Pregnant Women—The Pilot Study

2019 ◽  
Vol 16 (9) ◽  
pp. 1656 ◽  
Author(s):  
Lubica Argalasova ◽  
Ingrid Zitnanova ◽  
Diana Vondrova ◽  
Monika Dvorakova ◽  
Lucia Laubertova ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Oxidative Damage ◽  
Antioxidant Capacity ◽  
Pregnant Women ◽  
Environmental Tobacco Smoke ◽  
Tobacco Smoke ◽  
Passive Smoking ◽  
Oxidative Stress Parameters ◽  
And Oxidative Stress ◽  
Stress Parameters

Background: Exposure to ETS (environmental tobacco smoke) is one of the most toxic environmental exposures. Objective: To investigate the association of ETS with physiological, biochemical, and psychological indicators, as well as with urine antioxidant capacity (AC) and oxidative damage to lipids in a pilot sample of healthy pregnant women. Methods: Exposure to ETS was investigated via a validated questionnaire, and urine cotinine and the marker of oxidative damage to lipids via 8-isoprostane concentrations using an ELISA kit. Urine AC was determined by the spectrophotometric Trolox-equivalent antioxidant capacity (TEAC) method. From a sample of pregnant women (n = 319, average age 30.84 ± 5.09 years) in 80, the levels of cotinine and oxidative stress markers were analyzed. Results: Among the 80 pregnant women, 5% (7.4% confirmed by cotinine) reported being current smokers and 25% reported passive smoking in the household (18.8% confirmed by cotinine). The Kappa was 0.78 for smokers and 0.22 for ETS-exposed nonsmokers. Pregnant women in the ETS-exposed group had significantly reduced AC compared to both the nonsmoker (ETS−) and the smoker groups (p < 0.05). Nonsmokers had significantly lower levels of 8-isoprostane than smokers (p < 0.01) and ETS-exposed nonsmokers (p < 0.05). Correlations between urine levels of cotinine and AC were positive in ETS-exposed nonsmokers. Conclusion: A harmful association of active and passive smoking and oxidative stress parameters among pregnant women has been indicated.

Assessment of the exposure of children to environmental tobacco smoke (ETS) by different methods

1999 ◽  
Vol 18 (4) ◽  
pp. 297-301 ◽  
Author(s):  
Gerhard Scherer ◽  
Irmtrud Meger-Kossien ◽  
Kirsten Riedel ◽  
Thomas Renner ◽  
Michael Meger
Keyword(s):  
Environmental Tobacco Smoke ◽  
Exposure Time ◽  
Tobacco Smoke ◽  
Smoking Status ◽  
Chronic Illnesses ◽  
Muconic Acid ◽  
Urinary Cotinine ◽  
Exhaled Air ◽  
Salivary Cotinine

1 In order to elucidate the role of exposure to environ-mental tobacco smoke (ETS) in various acute and chronic illnesses in children, it is important to assess the degree of exposure by suitable methods. For this purpose, we determined the exposure to ETS in 39 children (4-15 years) and 43 adults (16+ years) by questionnaires, personal diffusion samplers for nicotine, and cotinine measurements in saliva and urine. In addition, the influence of the smoking status and the location of the home (urban or suburban) on the benzene exposure of the children was investigated. 2 On average, the 24 children living in homes with at least one smoker were exposed to ETS for 3.1 h/d. This is significantly longer (P<0.001) than the daily exposure time of the 15 children from nonsmoking homes (0.3 h/d). The nicotine concentrations on the personal samplers worn over 7 days were 0.615 and 0.046 tghn3 for children from smoking and nonsmoking homes, respectively (P <0.001). Average salivary cotinine levels were 1.95 ng/ml in children from smoking homes and 0.11 ng/ml in children from nonsmoking homes (P<0.01). The corresponding urinary cotinine levels were 29.4 and 4.5 ng/mg creatinine (P<0.001). There was no difference in the extent of ETS exposure between children and adults from smoking households. Adults from nonsmoking homes tended to have higher ETS exposure than children from nonsmoking homes. 3 Exposure to benzene, which was determined by means of personal samplers, measurements of benzene in exhaled air and of the urinary benzene metabolite trans, trans-muconic acid, was not significantly related to the smoking status of the home but primarily dependent on the location of the home.

scholarly journals Health Risk of Environmental Tobacco Smoke (ETS)

2013 ◽  
Author(s):  
Dewan Raja ◽  
Bahar Sultana
Keyword(s):  
Public Health ◽  
Environmental Tobacco Smoke ◽  
Tobacco Smoke ◽  
Passive Smoking ◽  
The Elderly ◽  
Second Hand Smoke ◽  
Potential Health ◽  
Secondhand Smoking ◽  
Smoking Exposure ◽  
Home Area

Environmental tobacco smoke is a preventable cause of significant morbidity and mortality among non-smokers. Reducing exposure to tobacco smoke is an essential community and public health objective. This report documents the substantial evidence characterizing the health risks caused by exposure to passive smoking. Multiple major reviews of evidence have concluded that environmental tobacco smoke is a known human carcinogen and that exposure to passive smoke causes adverse effects like SIDS, congenital birth defects, lead poisoning, and lung cancer. Regrettably, reductions in exposure have been slower among small children than among adults, as growing workplace restriction protects the majority of adults while the homes remain the most important source of exposure for children. The relationship between passive smoking exposure and potential health hazards to all ages of population were researched. We analyzed and systematically reviewed information from multiple literature sources. We found that everyone from the fetus to the elderly is significantly affected by passive smoking. Like firsthand smoking, secondhand smoking is also hazardous. Health education and regulations regarding secondhand smoking in both the office and home area can improve public health. This paper indicates a critical need for second hand smoke reduction interventions especially among vulnerable populations.

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