Model of Impedance Changes in Unmyelinated Nerve Fibers

2019 ◽  
Vol 66 (2) ◽  
pp. 471-484 ◽  
Author(s):  
Ilya Tarotin ◽  
Kirill Aristovich ◽  
David Holder

1987 ◽  
Vol 67 (3) ◽  
pp. A276-A276
Author(s):  
Robert R. Myers ◽  
Michael W. Kalichman ◽  
Henry C. Powell


1973 ◽  
Vol 76 (3) ◽  
pp. 1004-1007 ◽  
Author(s):  
A. V. Zeveke ◽  
V. I. Myaderov ◽  
V. A. Utkin ◽  
V. L. Shaposhnikov


Pain ◽  
1982 ◽  
Vol 14 (2) ◽  
pp. 198 ◽  
Author(s):  
B. R. Fink ◽  
A. M. Cairns


1996 ◽  
Vol 91 (2) ◽  
pp. 145-154 ◽  
Author(s):  
Takashi Kanda ◽  
Hiroshi Tsukagoshi ◽  
Masaya Oda ◽  
Kazuhito Miyamoto ◽  
Hitoshi Tanabe


1979 ◽  
Vol 88 (6_suppl2) ◽  
pp. 1-17 ◽  
Author(s):  
Robert S. Kimura ◽  
Carol Y. Ota ◽  
Tadahiko Takahashi

Nerve fiber synapses were observed on the small myelinated and unmyelinated spiral ganglion cells of the human cochlea. The nerve fibers penetrated the junctions of the ensheathing satellite cells and myelin lamellae, and partly invaginated into the perikarya to establish axosomatic synapses. One small myelinated neuron with a nerve fiber synapse demonstrated multipolar cell processes. However, most small neurons with identical ultrastructural characteristics did not show these synapses. Axodendritic synapses were also seen on the dendritic processes of the small neurons and between varicose nerves and unmyelinated nerve fibers some distance from the small neurons. While these observations conform in some aspects with the concept of parasympathetic nerve fibers and neurons in the cochlea, they are also compatible with the idea that the small neurons may have an auditory function influenced by the synaptic contacts of efferent fibers from the olivocochlear bundle.



2015 ◽  
Vol 75 (1) ◽  
pp. 53-60 ◽  
Author(s):  
Mathilde Duchesne ◽  
Laurent Magy ◽  
Laurence Richard ◽  
Pierre Ingrand ◽  
Jean-Philippe Neau ◽  
...  


2021 ◽  
Vol 15 ◽  
Author(s):  
Rui Wu ◽  
He Lv ◽  
Hui Wang ◽  
Zhaoxia Wang ◽  
Yun Yuan

ObjectivesMitofusin 2 and ganglioside-induced differentiation-associated protein 1 are two main mitochondrial dynamics-related proteins. Dysfunction of these two proteins leads to different subtypes of Charcot–Marie–Tooth disease type 2A (CMT2A) and CMT2K. This study aims to report the pathological difference between CMT2A and CMT2K in a large cohort.MethodsThirty patients with molecularly confirmed CMT2A and nine with CMT2K were identified by next-generation sequencing. Sural nerve biopsies were performed in 29 patients.ResultsThe patients with both diseases showed length-dependent neuropathy with distal weakness, sensory loss, and no deep tendon reflex. Optic neuropathy appeared in 3/30 (10%) patients with CMT2A. Tendon contracture appeared in 4/9 (50.0%) patients with CMT2K. Sural biopsy revealed the loss of both myelinated and unmyelinated nerve fibers. Closely packed, irregularly oriented neurofilaments were observed in axons of unmyelinated nerve fibers in both diseases. Another important finding was the ubiquitous presence of smaller, rounded, and fragmented mitochondria in CMT2A and elongated mitochondria in CMT2K in the myelinated and unmyelinated axons.ConclusionThis study confirmed large diversity in phenotypes between CMT2A and CMT2K. Mitochondrial dynamics-related variations can induce different mitochondrial morphological changes and neurofilament accumulation in axons.



1970 ◽  
Vol 48 (4) ◽  
pp. 837-839 ◽  
Author(s):  
Haukur Melax ◽  
Thomas S. Leeson

The sinoatrial node of the rat is composed of loosely interwoven modified cardiac muscle cells, which are lined up in groups to form somewhat irregular layers or plates. A generous amount of atrial specific granules is found in their cytoplasm. Between the nodal cells there is a loose connective tissue containing numerous bundles of unit fibrils of collagen and unmyelinated nerve fibers. In the atrioventricular node the modified cardiac cells are larger than those of the sinoatrial node, and are arranged in small groups. They contain a large amount of glycogen. Unmyelinated nerve fibers are extremely numerous in the atrioventricular node and myelinated nerve fibers are also present.





1973 ◽  
Vol 29 (1) ◽  
pp. 56-59 ◽  
Author(s):  
J.-M. Peyronnard ◽  
A. J. Aguayo ◽  
G. M. Bray


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