Chlamydia and HPV induce centrosome amplification in the host cell through additive mechanisms

2021 ◽  
Author(s):  
Kevin Wang ◽  
Karissa J. Muñoz ◽  
Ming Tan ◽  
Christine Sütterlin
Author(s):  
A. E. Ritchie

The cause of bluecomb disease in turkeys is unknown. Filtration of infective intestinal contents suggests a viral origin. To date, it has not been possible to isolate the etiologic agent in various cell cultures. The purpose of this work was to characterize as many virus-like entities as were recognizable in intestines of both healthy and bluecomb-infected turkeys. By a comparison of the viral populations it was hoped that some insight might be gained into the cause of this disease. Studies of turkey hemorraghic enteritis by Gross and Moore (Avian Dis. 11: 296-307, 1967) have suggested that a bacteriophage-host cell interaction may bear some causal relationship to that disease.


Author(s):  
Frederick A. Murphy ◽  
Alyne K. Harrison ◽  
Sylvia G. Whitfield

The bullet-shaped viruses are currently classified together on the basis of similarities in virion morphology and physical properties. Biologically and ecologically the member viruses are extremely diverse. In searching for further bases for making comparisons of these agents, the nature of host cell infection, both in vivo and in cultured cells, has been explored by thin-section electron microscopy.


2009 ◽  
Vol 47 (05) ◽  
Author(s):  
Z Varga ◽  
Z Greff ◽  
J Pató ◽  
P Bánhegyi ◽  
L Őrfi ◽  
...  

2020 ◽  
Vol 5 (Special) ◽  

The coronavirus illness (COVID-19) is caused by a new recombinant SARS-CoV (SARS-CoV) virus (SARS-CoV-2). Target cell infection by SARS-CoV is mediated by the prickly protein of the coronavirus and host cell receptor, enzyme 2 converting angiotensin (ACE2) [3]. Similarly, a recent study suggests that cellular entry by SARS-CoV-2 is dependent on both ACE2 as well as type II transmembrane axial protease (TMPRSS2) [4]. This means that detection of ACE2 and PRSS2 expression in human tissues can predict potential infected cells and their respective effects in COVID-19 patients [1].


2007 ◽  
Vol 42 (2) ◽  
pp. 273-277
Author(s):  
J. Szarka ◽  
Eszter Szarka ◽  
G. Csilléry

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