The role of facial nerve conduction studies and electromyography in predicting the outcome of Bell's palsy

1997 ◽  
Vol 4 (4) ◽  
pp. 348-351
Author(s):  
B. I. Yamout ◽  
G. Zaytoun ◽  
I. Nuweihed
Keyword(s):  
Facial Nerve ◽  
Nerve Conduction ◽  
Bell’S Palsy ◽  
Nerve Conduction Studies ◽  
Bell's Palsy

scholarly journals Nerve Conduction Study in Bell's Palsy

2017 ◽  
Vol 15 (1) ◽  
pp. 44-45
Author(s):  
Sanjib Kumar Shah ◽  
R. P. Kothari ◽  
S. D. Bassi
Keyword(s):  
Facial Nerve ◽  
Nerve Injury ◽  
Nerve Conduction Study ◽  
Nerve Conduction ◽  
Axonal Injury ◽  
Bell’S Palsy ◽  
Nerve Damage ◽  
The State ◽  
Bell's Palsy ◽  
Normal Side

 Background: Nerve conduction study (NCS) is an important electrophysiological tool that can be used for the measurement of the latency and amplitude of the facial nerve. The NCS helps in predicting the prognosis of patients with Bell's palsy. Evaluation of NCS of patients with Bell's palsy may have contribution in counselling and management of the patient. Objective: The study was aimed to study the state of nerve damage in patients with Bell's palsy. Methods: The latency and amplitude of facial nerve stimulating bilateral Orbicularis Oculi and Orbicularis Oris muscles were assessed and noted in 24 patients of Bell's palsy. Results: 15(62.5%) of the patients had significant axonal injury affecting one side and only 1(4.16%) patient had demyelinating injury. The total of 8(33.33%) patients had insignificant axonal injury of the facial nerve in comparision with the normal side. Conclusions: Nerve conduction study shows predominant axonal type of facial nerve injury in patients with Bell's palsy and hence provides valuable information regarding the prognosis and the state of nerve damage in patients with Bell's palsy.

The role of the facial nerve latency test in the prognosis of Bell's Palsy

1990 ◽  
Vol 100 (10) ◽  
pp. 1083-1085 ◽  
Author(s):  
Antonios Th. Skevas ◽  
Vasilios G. Danielides ◽  
Dimitrios A. Assimakopoulos
Keyword(s):  
Facial Nerve ◽  
Bell’S Palsy ◽  
Bell's Palsy ◽  
Facial Nerve Latency Test

The maximal stimulation and facial nerve conduction latency tests: Predicting the outcome of bell's palsy

1994 ◽  
Vol 104 (S1) ◽  
pp. 1-6 ◽  
Author(s):  
John M. Ruboyianes ◽  
Kedar K. Adour ◽  
David Q. Santos ◽  
Peter G. Von Doersten
Keyword(s):  
Facial Nerve ◽  
Nerve Conduction ◽  
Bell’S Palsy ◽  
Bell's Palsy ◽  
Maximal Stimulation

Distribution of Facial Nerve Conduction Velocities in Patients with Bell’s Palsy

1994 ◽  
pp. 514-516 ◽  
Author(s):  
M. Aoyagi ◽  
O. Saito ◽  
H. Tojima ◽  
H. Maeyama ◽  
Y. Koike
Keyword(s):  
Facial Nerve ◽  
Nerve Conduction ◽  
Bell’S Palsy ◽  
Bell's Palsy ◽  
Conduction Velocities

scholarly journals Radiation Exposure and Bell’s Palsy: A Hypothetical Association

2017 ◽  
Author(s):  
M Khateri ◽  
S Cheraghi ◽  
A Ghadimi ◽  
H Abdollahi
Keyword(s):  
Peripheral Nerve ◽  
Facial Nerve ◽  
Radiation Exposure ◽  
Mobile Phones ◽  
Nerve Palsy ◽  
Bell’S Palsy ◽  
Wireless Devices ◽  
Bell's Palsy ◽  
Psychological Stressors

Bell’s palsy is an idiopathic peripheral nerve palsy involving the facial nerve. It accounts for 60 to 75% of all cases of unilateral facial paralysis. The main mechanisms to induce BP remain unclear, but infection, ischemic condition and immunodeficiency may contribute to the development of Bell’s palsy. Accumulating evidence has shown several factors can trigger the reactivation of latent HSV including psychological stressors, physical stressors and immunosuppression. Ionization and non-ionization radiations are of importance of physical stressors. Some data have shown radiation can reactivate HSVs. Based on preliminary studies showing radiation reactivation of HSVs, we aimed to hypothesize radiation (in both forms of ionization and non-ionization) may cause Bell’s palsy. In the future, the role of radiotherapy, radiofrequency radiation from mobile phones and wireless devices in HSV reactivation and Bell’s palsy should be investigated.

Treatment for facial palsy

1968 ◽  
Vol 6 (7) ◽  
pp. 25-26
Keyword(s):  
Facial Nerve ◽  
Facial Palsy ◽  
Nerve Conduction ◽  
Conduction Block ◽  
Bell’S Palsy ◽  
Bell's Palsy ◽  
Facial Muscles ◽  
Nerve Conduction Block

There is controversy on the management of the idiopathic facial nerve palsies (Bell’s palsy). About two out of every three patients have a nerve conduction block alone and recover completely in less than one month.1 The rest develop denervation of the facial muscles to a varying degree and never recover completely. In addition, contracture of the facial muscles may occur, with severe disfigurement. About 10% of all patients with facial palsy have complete denervation and serious sequelae.2 Using electromyography3 and measurement of the anodal galvanic threshold4 an accurate prognosis can be given within the first week.

The possible contributing factors for the success of steroid therapy in Bell's Palsy: a clinical and electrophysiological study

1994 ◽  
Vol 108 (11) ◽  
pp. 940-943 ◽  
Author(s):  
Tarek S. Shafshak ◽  
Aymen Y. Essa ◽  
Fathy A. Bakey
Keyword(s):  
Facial Nerve ◽  
Electrophysiological Study ◽  
Bell’S Palsy ◽  
Control Group ◽  
Not Given ◽  
Bell's Palsy ◽  
Contributing Factors ◽  
Nerve Recovery ◽  
One Year

AbstractIncomplete recovery from Bell's palsy was observed in some patients even after the intake of corticosteroids. This prospective study was performed on 160 patients with unilaterial nonrecurrent Bell's palsy in order to investigate the role of prednisolone on the prognosis of Bell's palsy. Ninety-three patients were given prednisolone tablets (1 mg/kg body wt/day up to 70 mg) for six successive days, then the dose was reduced gradually over the next four days. The remaining 67 patients were not given prednisolone (control group). Facial nerve recovery was assessed clinically and electrophysiologically for up to one year. The results of this study suggested that the most probable contributing factor for the success of prednisolone in improving the prognosis of Bell's palsy was its early intake (within the first 24 hours following onset).

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