scholarly journals Nuclear factor  B inactivation in the rat liver ameliorates short term total warm ischaemia/reperfusion injury

Gut ◽  
2005 ◽  
Vol 54 (6) ◽  
pp. 835-842 ◽  
Author(s):  
H Suetsugu
2007 ◽  
Vol 0 (0) ◽  
pp. 070901081846002-??? ◽  
Author(s):  
Kosho Yamanouchi ◽  
Susumu Eguchi ◽  
Yukio Kamohara ◽  
Katsuhiko Yanaga ◽  
Sadayuki Okudaira ◽  
...  

1997 ◽  
Vol 29 (7) ◽  
pp. 3002-3003 ◽  
Author(s):  
S.C. Weight ◽  
P.N. Furness ◽  
P.R.F. Bell ◽  
M.L. Nicholson

2007 ◽  
Vol 77 (3) ◽  
pp. 506-514 ◽  
Author(s):  
S. Hallstrom ◽  
M. Franz ◽  
H. Gasser ◽  
M. Vodrazka ◽  
S. Semsroth ◽  
...  

2002 ◽  
Vol 36 (2) ◽  
pp. 241-247 ◽  
Author(s):  
Nina E Saxton ◽  
Johanna L Barclay ◽  
Andrew D Clouston ◽  
Jonathan Fawcett

2015 ◽  
Vol 129 (4) ◽  
pp. 345-362 ◽  
Author(s):  
Jordi Gracia-Sancho ◽  
Araní Casillas-Ramírez ◽  
Carmen Peralta

Ischaemia/reperfusion injury is an important cause of liver damage during surgical procedures such as hepatic resection and liver transplantation, and represents the main cause of graft dysfunction post-transplantation. Molecular processes occurring during hepatic ischaemia/reperfusion are diverse, and continuously include new and complex mechanisms. The present review aims to summarize the newest concepts and hypotheses regarding the pathophysiology of liver ischaemia/reperfusion, making clear distinction between situations of cold and warm ischaemia. Moreover, the most updated therapeutic strategies including pharmacological, genetic and surgical interventions, as well as some of the scientific controversies in the field are described.


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