Mice Lacking Inducible Nitric Oxide Synthase Have Improved Left Ventricular Contractile Function and Reduced Apoptotic Cell Death Late After Myocardial Infarction

Contractile Function ◽

Apoptotic Cell ◽

Left Ventricular ◽

Oxide Synthase ◽

Expression of Inducible Nitric Oxide Synthase and Fas/Fas Ligand Correlates with the Incidence of Apoptotic Cell Death in Atheromatous Plaques of Human Coronary Arteries

Nitric Oxide ◽

10.1006/niox.2000.0311 ◽

2000 ◽

Vol 4 (6) ◽

pp. 561-571 ◽

Cited By ~ 27

Author(s):

Teiji Esaki ◽

Toshio Hayashi ◽

Emiko Muto ◽

Hatsuyo Kano ◽

Thakur Navin Kumar ◽

...

Keyword(s):

Nitric Oxide ◽

Cell Death ◽

Nitric Oxide Synthase ◽

Coronary Arteries ◽

Fas Ligand ◽

Apoptotic Cell ◽

Atheromatous Plaques ◽

Oxide Synthase ◽

Inhibition of neutral sphingomyelinase decreases elevated levels of inducible nitric oxide synthase and apoptotic cell death in ocular hypertensive rats

Toxicology and Applied Pharmacology ◽

10.1016/j.taap.2014.08.026 ◽

2014 ◽

Vol 280 (3) ◽

pp. 389-398 ◽

Cited By ~ 7

Author(s):

Mutay Aslan ◽

Goksun Basaranlar ◽

Mustafa Unal ◽

Akif Ciftcioglu ◽

Narin Derin ◽

...

Keyword(s):

Nitric Oxide ◽

Cell Death ◽

Nitric Oxide Synthase ◽

Apoptotic Cell ◽

Apoptotic Cell Death ◽

Hypertensive Rats ◽

Neutral Sphingomyelinase ◽

Oxide Synthase ◽

Borderzone Contractile Dysfunction Is Transiently Attenuated and Left Ventricular Structural Remodeling Is Markedly Reduced Following Reperfused Myocardial Infarction in Inducible Nitric Oxide Synthase Knockout Mice

Journal of the American College of Cardiology ◽

10.1016/j.jacc.2007.07.047 ◽

2007 ◽

Vol 50 (18) ◽

pp. 1799-1807 ◽

Cited By ~ 25

Author(s):

Wesley D. Gilson ◽

Frederick H. Epstein ◽

Zequan Yang ◽

Yaqin Xu ◽

Konkal-Matt R. Prasad ◽

...

Keyword(s):

Nitric Oxide ◽

Myocardial Infarction ◽

Nitric Oxide Synthase ◽

Knockout Mice ◽

Left Ventricular ◽

Contractile Dysfunction ◽

Reperfused Myocardial Infarction ◽

Oxide Synthase ◽

S-Methylisothiourea Inhibits Inducible Nitric Oxide Synthase and Improves Left Ventricular Performance after Acute Myocardial Infarction

Biochemical and Biophysical Research Communications ◽

10.1006/bbrc.1996.1509 ◽

1996 ◽

Vol 227 (2) ◽

pp. 328-333 ◽

Cited By ~ 29

Author(s):

Stephen M. Wildhirt ◽

Hiroshi Suzuki ◽

Wolf-Peter Wolf ◽

Ryszard Dudek ◽

Damian Horstman ◽

...

Keyword(s):

Nitric Oxide ◽

Myocardial Infarction ◽

Acute Myocardial Infarction ◽

Nitric Oxide Synthase ◽

Left Ventricular ◽

Left Ventricular Performance ◽

Ventricular Performance ◽

Oxide Synthase ◽

Enhanced ?-adrenergic response in rat papillary muscle by inhibition of inducible nitric oxide synthase after myocardial infarction

Acta Physiologica ◽

10.1111/j.1748-1716.2007.01684.x ◽

2007 ◽

Vol 190 (2) ◽

pp. 111-117 ◽

Cited By ~ 7

Author(s):

V. D. Pinto ◽

G. J. S. Cutini ◽

C. L. Sartório ◽

A. S. Paigel ◽

D. V. Vassallo ◽

...

Keyword(s):

Nitric Oxide ◽

Myocardial Infarction ◽

Nitric Oxide Synthase ◽

Papillary Muscle ◽

Adrenergic Response ◽

Oxide Synthase ◽

Rat Papillary Muscle ◽

INCREASED INDUCIBLE NITRIC OXIDE SYNTHASE EXPRESSION CONTRIBUTES TO MYOCARDIAL DYSFUNCTION AND HIGHER MORTALITY POST MYOCARDIAL INFARCTION IN MICE

Journal of Hypertension ◽

10.1097/00004872-200006001-00241 ◽

2000 ◽

Vol 18 ◽

pp. S71

Author(s):

Q. Feng ◽

X. Lu

Keyword(s):

Nitric Oxide ◽

Myocardial Infarction ◽

Nitric Oxide Synthase ◽

Myocardial Dysfunction ◽

Post Myocardial Infarction ◽

Oxide Synthase ◽

Induction of inducible nitric oxide synthase by Epstein-Barr virus B95-8-derived LMP1 in Balb/3T3 cells promotes stress-induced cell death and impairs LMP1-mediated transformation

Oncogene ◽

10.1038/sj.onc.1205990 ◽

2002 ◽

Vol 21 (52) ◽

pp. 8047-8061 ◽

Cited By ~ 19

Author(s):

Jau-Song Yu ◽

Hsing-Chen Tsai ◽

Chih-Ching Wu ◽

Li-Ping Weng ◽

Hsin-Pai Li ◽

...

Keyword(s):

Nitric Oxide ◽

Cell Death ◽

Nitric Oxide Synthase ◽

Epstein Barr Virus ◽

3T3 Cells ◽

Barr Virus ◽

Epstein Barr ◽

Oxide Synthase ◽

Improvement of cardiac performance after selective modulation of increased inducible Nitric Oxide Synthase (iNOS) formation in myocardial infarction

Journal of the American College of Cardiology ◽

10.1016/s0735-1097(96)81586-5 ◽

1996 ◽

Vol 27 (2) ◽

pp. 183-184

Author(s):

Stephen M. Wildhirt ◽

Hiroshi Suzuki ◽

Ryszard Dudek ◽

Damian Horstman ◽

Richard J. Bing

Keyword(s):

Nitric Oxide ◽

Myocardial Infarction ◽

Nitric Oxide Synthase ◽

Cardiac Performance ◽

Selective Modulation ◽

Oxide Synthase ◽

Antisense RNA to Inducible Nitric Oxide Synthase Reduces Cytokine-Mediated Brain Endothelial Cell Death

Annals of the New York Academy of Sciences ◽

10.1196/annals.1338.037 ◽

2005 ◽

Vol 1042 (1) ◽

pp. 439-447 ◽

Cited By ~ 3

Author(s):

DING-I YANG ◽

SHAWEI CHEN ◽

UTHAYASHANKER R. EZEKIEL ◽

JAN XU ◽

YINGJI WU ◽

...

Keyword(s):

Nitric Oxide ◽

Cell Death ◽

Endothelial Cell ◽

Nitric Oxide Synthase ◽

Antisense Rna ◽

Brain Endothelial Cell ◽

Endothelial Cell Death ◽

Oxide Synthase ◽

Paeoniflorin ameliorates acute myocardial infarction of rats by inhibiting inflammation and inducible nitric oxide synthase signaling pathways

Molecular Medicine Reports ◽

10.3892/mmr.2015.3870 ◽

2015 ◽

Vol 12 (3) ◽

pp. 3937-3943 ◽

Cited By ~ 27

Author(s):

CHANG CHEN ◽

PING DU ◽

JUNJIE WANG

Keyword(s):

Nitric Oxide ◽

Myocardial Infarction ◽

Acute Myocardial Infarction ◽

Nitric Oxide Synthase ◽

Signaling Pathways ◽

Oxide Synthase ◽