scholarly journals Cystitis Cystica, Cystitis Glandularis, and Brunn's Nests in a Feline Urinary Bladder

1981 ◽  
Vol 18 (1) ◽  
pp. 113-116 ◽  
Author(s):  
J. F. Zachary
Keyword(s):  
Urinary Bladder ◽  
Cystitis Glandularis ◽  
Cystitis Cystica

scholarly journals Cystitis glandularis forming a tumorous lesion in the urinary bladder: A rare appearance of disease

2008 ◽  
Vol 24 (4) ◽  
pp. 558 ◽  
Author(s):  
Kazuyoshi Shigehara ◽  
Tohru Miyagi ◽  
Takao Nakashima ◽  
Masayoshi Shimamura
Keyword(s):  
Urinary Bladder ◽  
Cystitis Glandularis ◽  
Tumorous Lesion

Polyps and Masses of the Pediatric Urinary Bladder: A 21-Year Pathology Review

2011 ◽  
Vol 14 (6) ◽  
pp. 438-444 ◽  
Author(s):  
Alison R. Huppmann ◽  
Bruce R. Pawel
Keyword(s):  
Urinary Bladder ◽  
Pediatric Population ◽  
Low Grade ◽  
Nephrogenic Adenoma ◽  
Patient Demographics ◽  
Urothelial Tumors ◽  
Histologic Types ◽  
Cystitis Cystica ◽  
Pathology Review ◽  
Made In

Although not uncommon in adults, bladder tumors are rare in children. In addition, the histologic types of tumors seen in the pediatric population differ from those seen in adults. Although rhabdomyosarcoma is the most common pediatric bladder tumor, many other benign, malignant, and reactive lesions can be encountered. All may present clinically as a mass or polyp in the bladder. This study was designed to describe the pathology and patient demographics of pediatric bladder masses, because there are few studies describing these entities. Retrospectively reviewing our experience over a 21-year period, we identified 98 specimens from 65 patients with polyps or masses in the urinary bladder. As expected, the most frequent diagnosis was rhabdomyosarcoma. This was followed by fibroepithelial polyp and a variety of additional nonurothelial tumors. Only 7 urothelial tumors were identified, including 1 low-grade papillary urothelial carcinoma. Inflammatory lesions, such as cystitis cystica and nephrogenic adenoma, were invariably associated with an irritating factor when a history was provided. Our findings emphasize that diagnoses made in the pediatric urinary bladder are distinct from those in adults, although a wide variety of lesions may still be seen.

scholarly journals Divergent pathway of intestinal metaplasia and cystitis glandularis of the urinary bladder

2006 ◽  
Vol 19 (11) ◽  
pp. 1395-1401 ◽  
Author(s):  
Ming-Tse Sung ◽  
Antonio Lopez-Beltran ◽  
John N Eble ◽  
Gregory T MacLennan ◽  
Puay-Hoon Tan ◽  
...  
Keyword(s):  
Urinary Bladder ◽  
Intestinal Metaplasia ◽  
Cystitis Glandularis

scholarly journals Identification and Validation of Gene Expression Patterns in Cystitis Glandularis Patients and Controls

2017 ◽  
Vol 22 (6) ◽  
pp. 743-750 ◽  
Author(s):  
Chen-long Chu ◽  
Chen-hui Zhao ◽  
Zhi-wei Zhang ◽  
Ming-wei Wang ◽  
Zhao-hui Zhang ◽  
...  
Keyword(s):  
Gene Expression ◽  
Urinary Bladder ◽  
Signaling Pathways ◽  
Expression Patterns ◽  
Gene Set Enrichment Analysis ◽  
Enzyme Linked Immunosorbent Assay ◽  
Sequencing Analysis ◽  
Healthy Controls ◽  
Expression Levels ◽  
Cystitis Glandularis

Our aim was to investigate differences in gene expression in bladder tissues between cystitis glandularis (CG) patients and healthy controls. Subsequent RNA was isolated from urinary bladder samples from CG patients and healthy controls, followed by RNA sequencing analysis. There were 4263 differentially expressed genes in urinary bladder between CG patients and controls, and 8 genes were verified with real-time PCR, Western blot, and enzyme-linked immunosorbent assay (ELISA) analysis. Gene set enrichment analysis (GSEA) revealed that 25 signaling pathways were upregulated in CG patients, and 17 signaling pathways were found upregulated in healthy controls. The mRNA expression levels of the indicated genes, including CCND1, CCNA1, EGFR, AR, CX3CL1, CXCL6, and CXCL1, were significantly increased in urinary bladder from CG and bladder cancer (BC) patients compared with healthy controls, while TP53 was decreased. CX3CL1, CXCL6, and CXCL1 concentrations in peripheral blood from CG and BC patients were significantly increased compared with healthy controls. The protein expression levels of CCND1, EGFR, and AR were significantly increased in urinary bladder from CG and BC patients compared with healthy controls. In conclusion, the gene expression profile of CG patients has established a foundation to study the gene mechanism of CG and BC progression.

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