scholarly journals Dioctophyma Renale in Ranch Mink

1998 ◽  
Vol 10 (1) ◽  
pp. 111-113 ◽  
Author(s):  
N. W. Dyer
Keyword(s):  
Ranch Mink

IMMUNOTOXICITY OF THE COMMERCIAL POLYBROMINATED DIPHENYL ETHER MIXTURE DE-71 IN RANCH MINK (MUSTELA VISON)

2007 ◽  
Vol 26 (5) ◽  
pp. 988 ◽  
Author(s):  
Pamela A. Martin ◽  
Greg J. Mayne ◽  
Steven J. Bursian ◽  
Gregg Tomy ◽  
Vince Palace ◽  
...  
Keyword(s):  
Diphenyl Ether ◽  
Mustela Vison ◽  
Polybrominated Diphenyl Ether ◽  
Ether Mixture ◽  
Ranch Mink

Progressive Retinal Degeneration in Ranch Mink

1984 ◽  
Vol 21 (1) ◽  
pp. 18-27 ◽  
Author(s):  
W. J. Hadlow
Keyword(s):  
Visual Acuity ◽  
Retinal Degeneration ◽  
Structural Integrity ◽  
Pigment Epithelium ◽  
Outer Nuclear Layer ◽  
Viral Diseases ◽  
Rods And Cones ◽  
Progressive Degeneration ◽  
Ranch Mink ◽  
Growth And Reproduction

Retinal degeneration was prevalent in a large group of sapphire and pastel mink (Mustela vison) kept for studies on slow viral diseases. Nearly 78% of those two to eight years old were affected. The retinopathy was equally common in both sexes but more frequent in sapphires (85%) than in pastels (63%), and it was severe more often in sapphires than in pastels. By light microscopy, the primary change appeared to be progressive degeneration of fully developed photoreceptors, beginning in their outer segments. In many mink, including some younger ones, the rods and cones and outer nuclear layer had disappeared from all but the far periphery of the fundus. The inner retinal layers were spared until late in the disease, and the pigment epithelium remained essentially unchanged. The cause of the retinopathy was not established. It may represent an abiotrophy in which the structural integrity of the photoreceptors began to wane in many mink after they reached two years of age. Apart from reducing visual acuity, the retinopathy has implications for the photoperiodic control of fur growth and reproduction in this highly light-sensitive carnivore.

scholarly journals Nonsuppurative Meningoencephalitis Associated with Aleutian Mink Disease Parvovirus Infection in Ranch Mink

2000 ◽  
Vol 12 (2) ◽  
pp. 159-162 ◽  
Author(s):  
N. W. Dyer ◽  
B. Ching ◽  
M. E. Bloom
Keyword(s):  
Polymerase Chain Reaction ◽  
Nervous System ◽  
Differential Diagnosis ◽  
Viral Genome ◽  
Brain Lesions ◽  
Chain Reaction ◽  
Mesenteric Lymph ◽  
Neurologic Disorders ◽  
Polymerase Chain ◽  
Ranch Mink

Severe nonsuppurative meningoencephalitis associated with Aleutian mink disease parvovirus (ADV) infection was observed in adult ranch mink. Brain lesions included severe, locally extensive to coalescing lymphoplasmacytic meningoencephalitis with accompanying gliosis, satellitosis, and mild extension of inflammation into the leptomeninges. ADV was identified in mesenteric lymph node, spleen, brain, and liver of affected mink by polymerase chain reaction techniques. Sequences of the ADV isolate (TH5) revealed 2 unique residues in the region of the viral genome that determines pathogenicity. These findings suggest that certain strains of ADV may preferentially cause disease in the nervous system. ADV infection should be considered in the differential diagnosis of neurologic disorders in mink.

scholarly journals Giant-Cell Osteosarcoma in a Female Ranch Mink (Mustela Vision)

1998 ◽  
Vol 10 (3) ◽  
pp. 301-302 ◽  
Author(s):  
I. Mikaelian ◽  
B. Martineau-Doizé ◽  
D. Martineau
Keyword(s):  
Giant Cell ◽  
Ranch Mink

scholarly journals Common Occurrence of Urethral Cysts in Older Female Ranch Mink

1981 ◽  
Vol 18 (5) ◽  
pp. 599-607 ◽  
Author(s):  
W. J. Hadlow ◽  
R. E. Race
Keyword(s):  
Urinary Incontinence ◽  
Clinical Signs ◽  
Mustela Vison ◽  
Viral Diseases ◽  
Common Occurrence ◽  
Urine Retention ◽  
Urethral Wall ◽  
Age Dependent ◽  
Proximal Urethra ◽  
Ranch Mink

Cysts were found often in the proximal urethra of female sapphire and pastel mink, Mustela vison, examined at necropsy during studies on slow viral diseases. Their prevalence, the same in both color phases, was age-dependent. They occurred in less than 2% of females under 2 years old but in more than 60% of those over 7 years old. The cysts varied from ovoid vesicles 3 to 4 mm long to multilocular masses 10 to 15 mm across that greatly distended the proximal urethra and sometimes occluded it. Small cysts were not accompanied by clinical signs, but large ones often caused persistent urinary incontinence and occasionally, urine retention. The cysts contained fluid that varied from water-clear to dull yellow. They arose by expansion of small urethral glands normally present in female mink and destroyed much of the urethral wall by pressure atrophy. Although the cysts became larger and more prevalent as the mink aged, the stimulus that caused them to form was not apparent. They appear to have no counterpart in other animals.

Seasonal changes in aggression, hunger, and curiosity in ranch mink

1969 ◽  
Vol 47 (6) ◽  
pp. 1395-1404 ◽  
Author(s):  
R. R. MacLennan ◽  
E. D. Bailey
Keyword(s):  
Breeding Season ◽  
Seasonal Changes ◽  
Environmental Temperature ◽  
Gonadal Hormones ◽  
Corpora Lutea ◽  
Field Situation ◽  
Body Weights ◽  
Males And Females ◽  
And Storage ◽  
Ranch Mink

Aggression, hunger, and curiosity of ranch mink (Mustela vison) were examined. The animals were divided into socialized animals (those visually isolated at 1 year of age) and unsocialized animals (those visually isolated at 10 weeks of age).Aggression was measured by placing two mink in a special cage and measuring the time until a fight or confrontation occurred. The effects of 0, 24, and 48 hours of food deprivation were measured in summer, fall, and winter. The number of trips made by each mink up a vertical cage to get food was used as an indicator of hunger. Levels of curiosity were determined by measuring the speed and pattern of exploration of a novel open-field situation. The breeding behaviors of experienced and inexperienced male mink were compared.Aggression in mink consisted of at least two offensive threats and, as in other animals, fighting was ritualized to a great extent. All young mink showed some adult aggressive patterns by 20 weeks of age. Aggression in males reached a peak at breeding season (March) and declined throughout the summer and fall, to a low point in November. Socialized males had lower aggressive levels than unsocialized males. Aggression in female mink reached a peak after the breeding season and declined to a low in winter. These cycles were likely caused by gonadal hormones in males and hormones from the corpora lutea in females.Both consumption and storage of food must be considered in determining hunger in mink. Hunger varied inversely with environmental temperature, reaching a peak in the winter and a low point in the summer. Immature animals had lower hunger drives than adult animals. Changes in body weights of mink centered around the breeding season.Curiosity decreased from summer to fall as did aggression. Unsocialized males had higher levels of curiosity than socialized males and females. Immature mink showed incomplete exploratory behavior and had lower curiosity scores than adult animals.

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