scholarly journals Therapeutic dilemma in twin reversed arterial perfusion sequence

2019 ◽  
Vol 7 ◽  
pp. 2050313X1983634
Author(s):  
Yoko Aoyagi ◽  
Kentaro Kai ◽  
Masahiro Sumie ◽  
Naoki Fujiyoshi ◽  
Yuichi Furukawa ◽  
...  
Keyword(s):  
Fetal Growth ◽  
Growth Retardation ◽  
Japanese Woman ◽  
Anatomical Location ◽  
Fetal Growth Retardation ◽  
Third Trimester ◽  
Arterial Perfusion ◽  
The Third ◽  
Acardiac Twin ◽  
Twin Reversed Arterial Perfusion

The dissemination of minimally invasive in utero surgery reduced the mortality of twin reversed arterial perfusion sequence, but the mortality of expectantly treated surgical candidates remains high. A 26-year-old, non-parous, Japanese woman at 13 weeks of gestation had been diagnosed with twin reversed arterial perfusion sequence and was judged as a surgical candidate for radiofrequency ablation. However, she did not undergo surgery because of the anatomical location of the acardiac twin. At 18 weeks of gestation, the blood flow to the acardiac twin disappeared spontaneously. The pump twin began to demonstrate fetal growth retardation during the third trimester. The patient delivered a 1891 g female at term. We macroscopically identified the cause of the fetal growth retardation as velamentous insertion of the umbilical cord and microscopically diagnosed the acardiac twin with acardiac acephalus. We should give the same attention to the management of post–twin reversed arterial perfusion sequence as twin reversed arterial perfusion sequence itself.

Working During Pregnancy: Effects on the Fetus

PEDIATRICS ◽  
1982 ◽  
Vol 69 (6) ◽  
pp. 724-727
Author(s):  
Richard L. Naeye ◽  
Ellen C. Peters
Keyword(s):  
Blood Flow ◽  
Fetal Growth ◽  
Growth Retardation ◽  
Late Gestation ◽  
Fetal Growth Retardation ◽  
Maternal Factors ◽  
Third Trimester ◽  
The Third ◽  
Uteroplacental Blood Flow ◽  
Pregnancy Weight

In order to determine whether pregnancy outcome was altered when women were employed outside their homes, 7,722 pregnancies were analyzed. Gestations were not shortened but newborns of women who worked in the third trimester weighed 150 to 400 gm less than newborns of mothers who remained at home. The growth retardation was greatest when women were underweight pregravid and had a low pregnancy weight gain, when they were hypertensive, or when the work required standing. The growth retardation remained after the data were stratified by race, socioeconomic status, and other maternal factors that commonly influence fetal growth. The frequency of large placental infarcts progressively increased when women continued stand-up work into late gestation. Such infarcts reached a peak of 250/1,000 births after the 37th week of gestation in stand-up workers. Low uteroplacental blood flow is a likely explanation for both the fetal growth retardation and the large placental infarcts.

scholarly journals Maternal leucocyte zinc deficiency at start of third trimester as a predictor of fetal growth retardation.

BMJ ◽  
1987 ◽  
Vol 294 (6579) ◽  
pp. 1054-1056 ◽  
Author(s):  
J L Wells ◽  
D K James ◽  
R Luxton ◽  
C A Pennock
Keyword(s):  
Zinc Deficiency ◽  
Fetal Growth ◽  
Growth Retardation ◽  
Fetal Growth Retardation ◽  
Third Trimester

A Prospective Study of Haemostatic Tests at 28 Weeks Gestation as Predictors of Pre-Eclampsia and Growth Retardation

1989 ◽  
Vol 61 (02) ◽  
pp. 243-245 ◽  
Author(s):  
J G Thornton ◽  
B J Molloy ◽  
P S Vinall ◽  
P R Philips ◽  
R Hughes ◽  
...  
Keyword(s):  
Discriminant Analysis ◽  
Prospective Study ◽  
Fetal Growth ◽  
Growth Retardation ◽  
Degradation Products ◽  
Fetal Growth Retardation ◽  
Fibrin Degradation Products ◽  
A Prospective Study ◽  
Primiparous Women ◽  
Significant Model

SummaryA panel of haemostatic tests was perfomed on 400 primiparous women at 28 weeks to test whether one or more could predict the development of pregnancy complications. Fifteen women subsequently developed pre-eclampsia with significant proteinuria and 13 delivered growth retarded infants. There were no significant differences between mothers in the pre-eclampsia group and 22 randomly selected controls. A stepwise logistic discriminant analysis of the data did not produce a significant model. In the growth retarded group only beta thromboglobulin levels were significantly lower than in the controls (p <0.05), although in the logistic discriminant analysis the inclusion of both beta thromboglobulin and fibrin degradation products led to a borderline significant improvement in fit of the model. We conclude that the haemostatic variables studied are not significantly changed at 28 weeks nor clinically useful predictors of either pre-eclampsia or fetal growth retardation.

Uteroplacental hemodynamic disturbances in establishment of fetal growth retardation in streptozocin-induced diabetic rats

Diabetes ◽  
1990 ◽  
Vol 39 (6) ◽  
pp. 743-746 ◽  
Author(s):  
N. C. Chartrel ◽  
M. T. Clabaut ◽  
F. A. Boismare ◽  
J. C. Schrub
Keyword(s):  
Fetal Growth ◽  
Growth Retardation ◽  
Diabetic Rats ◽  
Fetal Growth Retardation
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