Early phase wound healing by primary intention as shown by ultrasonography

1998 ◽  
Vol 7 (5) ◽  
pp. 222-224 ◽  
Author(s):  
I.J. Kenney
Keyword(s):  
2016 ◽  
Vol 105 (7) ◽  
pp. 1828-1839 ◽  
Author(s):  
Ji-Ung Park ◽  
Hyun-Do Jung ◽  
Eun-Ho Song ◽  
Tae-Hyun Choi ◽  
Hyoun-Ee Kim ◽  
...  

1998 ◽  
Vol 22 (5) ◽  
pp. 276-279 ◽  
Author(s):  
Teruo Kiyama ◽  
Maria B. Witte ◽  
Frank J. Thornton ◽  
Adrian Barbul

Author(s):  
Francesca Tatini ◽  
Giada Magni ◽  
Gaetano De Siena ◽  
Roberto Pini ◽  
Stefano Bacci ◽  
...  
Keyword(s):  

2008 ◽  
Vol 86 (Supplement) ◽  
pp. 543
Author(s):  
C Legendre ◽  
M-C Moal ◽  
E Cassuto ◽  
L Rostaing ◽  
F Berthoux ◽  
...  

2015 ◽  
Vol 112 (18) ◽  
pp. E2327-E2336 ◽  
Author(s):  
Tomonori Katsuyama ◽  
Federico Comoglio ◽  
Makiko Seimiya ◽  
Erik Cabuy ◽  
Renato Paro

Regeneration of fragmented Drosophila imaginal discs occurs in an epimorphic manner involving local cell proliferation at the wound site. After disc fragmentation, cells at the wound site activate a restoration program through wound healing, regenerative cell proliferation, and repatterning of the tissue. However, the interplay of signaling cascades driving these early reprogramming steps is not well-understood. Here, we profiled the transcriptome of regenerating cells in the early phase within 24 h after wounding. We found that JAK/STAT signaling becomes activated at the wound site and promotes regenerative cell proliferation in cooperation with Wingless (Wg) signaling. In addition, we showed that the expression of Drosophila insulin-like peptide 8 (dilp8), which encodes a paracrine peptide to delay the onset of pupariation, is controlled by JAK/STAT signaling in early regenerating discs. Our findings suggest that JAK/STAT signaling plays a pivotal role in coordinating regenerative disc growth with organismal developmental timing.


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