scholarly journals Electromyographic diagnosis of ischaemic lesion of muscles of the extremities

2013 ◽  
Vol 0 (1) ◽  
pp. 67
Author(s):  
Oksana Gayko ◽  
Olexey Dolgopolov ◽  
Sergey Strafun
Keyword(s):  
2021 ◽  
Vol 14 (4) ◽  
pp. e236189
Author(s):  
Joana Morais ◽  
Ana Andrade Oliveira ◽  
Inês Burmester ◽  
Olga Pires

The artery of Percheron is a rare anatomical variation that supplies thalamus and the midbrain. A stroke in this area is a rare event. The presentation varies widely, with some bizarre disturbances, like transient episodic loss of consciousness similar to coma, somnolence, cognition and memory impairment and psychosis. We report a case of a patient who presented at the emergency department with a sudden change of consciousness. During the observation, she oscillated reactive state of consciousness with obnubilation similar to coma. The first exams were normal, which include a cranial CT of the brain, and so the patient was kept under observation. The final diagnosis was only possible 24 hours later with cranial CT where an ischaemic lesion on the Percheron territory was identified. This case highlights an unusual clinic and a difficult neuroimaging stroke diagnosis of a rare condition, that is unknown to most of the physicians.


1998 ◽  
Vol 2 (4) ◽  
pp. 315-322 ◽  
Author(s):  
P.S Yam ◽  
J Patterson ◽  
D.I Graham ◽  
T Takasago ◽  
D Dewar ◽  
...  

1981 ◽  
Vol 1 (1) ◽  
pp. 53-60 ◽  
Author(s):  
A. Tamura ◽  
D. I. Graham ◽  
J. McCulloch ◽  
G. M. Teasdale

A procedure for occluding the stem of the proximal middle cerebral artery of the rat is described. The operation is performed under anaesthesia through a small subtemporal craniectomy. After occlusion, 3 animals were perfused with carbon black and 8 with a FAM fixative (40% formaldehyde, glacial acetic acid, and methanol). The findings were compared with sham-operated animals. Carbon black studies demonstrated an area of impaired perfusion corresponding to the territory of the occluded artery in each animal. Neuropathological studies invariably showed that there was ischaemic brain damage in the cortex and basal ganglia. The frontal cortex was involved in every animal, as was the lateral part of the neostriatum; the sensorimotor and auditory cortex were involved in most animals, whereas the occipital cortex and medial striatum were involved only infrequently. The damage produced by ischaemia could be readily distinguished from the small local lesion seen at the surgical site in sham-operated animals. The ability to produce a consistent focal ischaemic lesion in the rodent brain provides a technical approach that is sufficiently reproducible to enable investigation of the pathophysiology of ischaemia using recently developed autoradiographic and neurochemical methods.


1984 ◽  
Vol 118 (2) ◽  
pp. 91-95 ◽  
Author(s):  
R.M. Ludatscher ◽  
M. Hashmonai ◽  
H. Peleg

1981 ◽  
Vol 1 (1) ◽  
pp. 61-69 ◽  
Author(s):  
A. Tamura ◽  
D. I. Graham ◽  
J. McCulloch ◽  
G. M. Teasdale

Local cerebral blood flow has been measured by quantitative autoradiography, employing [14C]iodoantipyrine as tracer, in rats killed half an hour after occlusion of the middle cerebral artery. The results were compared with pattern of local cerebral blood flow (CBF) in sham-operated rats and with neuropathological findings. In every animal there was a profound reduction (to 13% of control levels) in blood flow in the neocortex previously supplied by the occluded artery. The level of blood flow in the areas in which ischaemic brain damage occurred was 0.24 ±0.03 ml g−1 min−1 (mean ± SEM). This level of CBF is considerably greater than that reported following a similar surgical procedure in cats and primates. Moderate reductions in blood flow were also seen outside the territory of the occluded artery and in parts of the opposite hemisphere. Absolute increases in blood flow (hyperaemia) were seen only in the substantia nigra and globus pallidus ipsilateral to the occlusion. It is suggested that this finding and the reductions in blood flow outside the territory of the middle cerebral artery are reflections of alterations in neuronal function and metabolic activity secondary to the ischaemic lesion.


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