scholarly journals Charcot and vascular Parkinsonism

2017 ◽  
Vol 75 (3) ◽  
pp. 195-196 ◽  
Author(s):  
Hélio A. G. Teive ◽  
Francisco M. B. Germiniani ◽  
Renato P. Munhoz

ABSTRACT Jean-Martin Charcot (1825-1893), recognized as the founder of Neurology and the first formal teacher of nervous system diseases, died on August 16, 1893, from acute pulmonary edema secondary to myocardial infarction. In his last years, there were several descriptions of his gait and posture disorders, suggesting the diagnosis of “lower-half parkinsonism” due to cerebrovascular disease.

2013 ◽  
Vol 71 (10) ◽  
pp. 815-817 ◽  
Author(s):  
Marleide da Mota Gomes ◽  
Eliasz Engelhardt

Jean-Martin Charcot was a pioneer in a variety of subjects, including nervous system diseases; anatomy; physiology; pathology; and diseases of ageing, joints, and lungs. His medical achievements were mainly based on his anatomopathological proficiency, his observation, and his personal thoroughness that favored the delineation of the nosology of the main neurological diseases, including multiple sclerosis, amyotrophic lateral sclerosis, Parkinson’s disease, peroneal muscular atrophy, and hysteria/epilepsy. The link of this anatomoclinical method with iconographic representations and theatrical lessons, and the rich bibliographical documentations, carried out in a crowded diseased people barn - Salpetrière hospital were the basis of his achievements, which are still discussed 120 years after his death.


1989 ◽  
Vol 7 (2) ◽  
pp. 119-121 ◽  
Author(s):  
David Barash ◽  
Robert A. Silverman ◽  
Paul Gennis ◽  
Nancy Budner ◽  
Marshall Matos ◽  
...  

2019 ◽  
Vol 3 (2) ◽  
pp. 2514183X1988038 ◽  
Author(s):  
Olivier Walusinski

Fulgence Raymond (1844–1910) succeeded Jean-Martin Charcot (1825–1893) to the Chair of Nervous System Diseases. As famous as Charcot remains, Raymond has been forgotten. After a brief biographical account, we will present a few examples of his work still relevant today: hemichorea, Raymond-Cestan syndrome, hereditary spastic paraplegia and acute ascendant paralysis. In each case, his accurate clinical and anatomopathological descriptions are accompanied by aetiological hypotheses that are remarkably prescient with regard to current knowledge. Strongly committed to teaching, he published most of his lessons every year. They remain highly relevant historically, and sometimes for other reasons, as we shall see. We hope to show that Raymond does not deserve to be forgotten.


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