Exemplar Abstract for Comamonas acidovorans (den Dooren de Jong 1926) Tamaoka et al. 1987 emend. Willems et al. 1991, Delftia acidovorans (den Dooren de Jong 1926) Wen et al. 1999 and Pseudomonas acidovorans den Dooren de Jong 1926 (Approved Lists 1980).

2003 ◽  
Author(s):  
Charles Thomas Parker ◽  
Dorothea Taylor ◽  
George M Garrity
Keyword(s):  
Delftia Acidovorans ◽  
Comamonas Acidovorans ◽  
Pseudomonas Acidovorans

scholarly journals Human infection with Delftia tsuruhatensis isolated from a central venous catheter

2011 ◽  
Vol 60 (2) ◽  
pp. 246-248 ◽  
Author(s):  
Benjamin Preiswerk ◽  
Silvia Ullrich ◽  
Rudolf Speich ◽  
Guido V. Bloemberg ◽  
Michael Hombach
Keyword(s):  
Central Venous Catheter ◽  
Causative Agent ◽  
Venous Catheter ◽  
Human Infection ◽  
Central Venous ◽  
First Report ◽  
Catheter Related Infection ◽  
Delftia Acidovorans ◽  
Comamonas Acidovorans ◽  
Delftia Tsuruhatensis

We present the case of a patient with catheter-related infection caused by Delftia tsuruhatensis, a newly described species closely related to Delftia acidovorans (formerly Comamonas acidovorans). To date, D. tsuruhatensis has not been described as a pathogen. To the best of our knowledge, this is the first report describing D. tsuruhatensis as the causative agent of a human infection.

Exemplar Abstract for Delftia acidovorans (den Dooren de Jong 1926) Wen et al. 1999.

2003 ◽  
Author(s):  
Charles Thomas Parker ◽  
Dorothea Taylor ◽  
George M Garrity
Keyword(s):  
Delftia Acidovorans

Orbita-Infektion mit Delftia acidovorans nach Katzenkratzer

2020 ◽  
Vol 145 (21) ◽  
pp. 1559-1561
Author(s):  
Walter Johannes Smits ◽  
Heinz-Hubert Feucht ◽  
Frank Oellig ◽  
Bernhard Zöllner
Keyword(s):  
16S Rrna ◽  
Delftia Acidovorans ◽  
Klinischer Befund

Zusammenfassung Anamnese und klinischer Befund Die 60-jährige Patientin wurde vor 34 Monaten von ihrer Katze am linken Augenlid gekratzt. Danach kam es zu chronischen, langsam progredienten Hautveränderungen mit sich verstärkenden Kopfschmerzen. Therapieversuche mit Kortison, Pimecrolimus, Pregabalin und Metamizol waren erfolglos. Nach 24 Monaten klagte sie über starken Bulbus-Schmerz am linken Auge, zunehmende Schmerzen bei Augenbewegungen und große Lichtempfindlichkeit. Es zeigten sich granulomatöse Papeln im Bereich des Auges. Untersuchungen und Diagnose Die interdisziplinären Untersuchungsbefunde und klinisch-chemischen Parameter waren unauffällig. Eine Biopsie aus dem Lidbereich erbrachte den Nachweis von Delftia acidovorans mittels bakterieller 16S-rRNA-PCR. Therapie und Verlauf Unter der Therapie mit Piperacillin/Tazobactam 3-mal 4,5 g/d i. v. für 10 Tage kam es zu einer raschen klinischen Besserung, sodass die Patientin nach 11 Tagen entlassen werden konnte. Nach weiteren 10 Monaten war sie rezidiv- und beschwerdefrei. Folgerung D. acidovorans ist bislang nicht als zoonotischer Erreger in Erscheinung getreten, sollte aber bei Verletzungen durch Tiere in die differenzialdiagnostischen Erwägungen einbezogen werden.

scholarly journals Delftia acidovorans: A rare cause of septic pulmonary embolism from catheter-related infection: Case report and literature review

2019 ◽  
Vol 27 ◽  
pp. 100835 ◽  
Author(s):  
Dharti Patel ◽  
Arshad Muhammad Iqbal ◽  
Ateeq Mubarik ◽  
Nirali Vassa ◽  
Rania Godil ◽  
...  
Keyword(s):  
Pulmonary Embolism ◽  
Case Report ◽  
Literature Review ◽  
Catheter Related Infection ◽  
Delftia Acidovorans ◽  
Septic Pulmonary Embolism

Regulation of catabolic enzymes during long-term exposure of Delftia acidovorans MC1 to chlorophenoxy herbicides

Microbiology ◽  
2004 ◽  
Vol 150 (4) ◽  
pp. 1005-1014 ◽  
Author(s):  
Dirk Benndorf ◽  
Ian Davidson ◽  
Wolfgang Babel
Keyword(s):  
Stress Proteins ◽  
Elongation Factor ◽  
Resistance Mechanisms ◽  
Minor Role ◽  
Protein Patterns ◽  
Delftia Acidovorans ◽  
Catabolic Enzymes ◽  
Chlorophenoxy Herbicides ◽  
High Concentrations ◽  
2,4 Dichlorophenoxyacetic Acid

Delftia acidovorans MC1 is able to grow on chlorophenoxy herbicides such as 2,4-dichlorophenoxypropionic acid (2,4-DCPP) and 2,4-dichlorophenoxyacetic acid as sole sources of carbon and energy. High concentrations of the potentially toxic organics inhibit the productive degradation and poison the organism. To discover the target of chlorophenoxy herbicides in D. acidovorans MC1 and to recognize adaptation mechanisms, the response to chlorophenoxy acids at the level of proteins was analysed. The comparison of protein patterns after chemostatic growth on pyruvate and 2,4-DCPP facilitated the discovery of several proteins induced and repressed due to the substrate shifts. Many of the induced enzymes, for example two chlorocatechol 1,2-dioxygenases, are involved in the metabolism of 2,4-DCPP. A stronger induction of some catabolic enzymes (chlorocatechol 1,2-dioxygenase TfdCII, chloromuconate cycloisomerase TfdD) caused by an instant increase in the concentration of 2,4-DCPP resulted in increased rates of productive detoxification and finally in resistance of the cells. Nevertheless, the decrease of the (S)-2,4-DCPP-specific 2-oxoglutarate-dependent dioxygenase in 2D gels reveals a potential bottleneck in 2,4-DCPP degradation. Well-known heat-shock proteins and oxidative-stress proteins play a minor role in adaptation, because apart from DnaK only a weak or no induction of the proteins GroEL, AhpC and SodA was observed. Moreover, the modification of elongation factor Tu (TufA), a strong decrease of asparaginase and the induction of the hypothetical periplasmic protein YceI point to additional resistance mechanisms against chlorophenoxy herbicides.

scholarly journals The Tolerance of Chromium (VI) by Delftia acidovorans

2017 ◽  
Vol 23 (S1) ◽  
pp. 1360-1361
Author(s):  
Hongyan Zuo ◽  
Hailiang Dong
Keyword(s):  
Delftia Acidovorans ◽  
Chromium Vi
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