Local cytokine production in patients with external genital endometriosis

The article presents the results of the investigation of cytokine production by endometrioid heterotopias in organotypic cultivation in comparison with the cell secretory activity of peritoneal fluid and endometrial tissue in women with external genital endometriosis. The obtained results show systemic changes of regulation of cytokine production in the process of external genital endometriosis development.

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Local cytokine production and reactive oxygen species in the peritoneal fluid of endometriosis patients: prospective controlled study.

Fertility and Sterility ◽

10.1016/s0015-0282(01)02206-3 ◽

2001 ◽

Vol 76 (3) ◽

pp. S65

Author(s):

M.A Bedaiwy ◽

T Falcone ◽

J.M Goldberg ◽

M Attaran ◽

R.K Sharma ◽

...

Keyword(s):

Reactive Oxygen Species ◽

Peritoneal Fluid ◽

Cytokine Production ◽

Reactive Oxygen ◽

Controlled Study ◽

Oxygen Species ◽

Prospective Controlled Study ◽

Local Cytokine

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Expression of Natural Cytotoxicity Receptors on Peritoneal Fluid Natural Killer Cell and Cytokine Production by Peritoneal Fluid Natural Killer Cell in Women with Endometriosis

American Journal of Reproductive Immunology ◽

10.1111/aji.12206 ◽

2014 ◽

Vol 71 (4) ◽

pp. 359-367 ◽

Cited By ~ 31

Author(s):

Ayano Funamizu ◽

Atsushi Fukui ◽

Mai Kamoi ◽

Kohei Fuchinoue ◽

Megumi Yokota ◽

...

Keyword(s):

Natural Killer Cell ◽

Natural Killer ◽

Peritoneal Fluid ◽

Cytokine Production ◽

Killer Cell ◽

Natural Cytotoxicity ◽

Natural Cytotoxicity Receptors

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TCT-157 Increased Local Cytokine Production at Culprit Superficial Femoral Artery Plaques

Journal of the American College of Cardiology ◽

10.1016/j.jacc.2012.08.177 ◽

2012 ◽

Vol 60 (17) ◽

pp. B45-B46

Author(s):

Cameron Donaldson ◽

David Schneider ◽

Daniel Bertges ◽

Julie Adams ◽

Nader Elgharib ◽

...

Keyword(s):

Femoral Artery ◽

Superficial Femoral Artery ◽

Cytokine Production ◽

Local Cytokine

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High levels of interleukin‐8, soluble CD4 and soluble CD8 in bullous pemphigoid blister fluid. The relationship between local cytokine production and lesional T‐cell activities

British Journal of Dermatology ◽

10.1046/j.1365-2133.2000.03894.x ◽

2000 ◽

Vol 143 (6) ◽

pp. 1235-1240 ◽

Cited By ~ 21

Author(s):

C‐C. Sun ◽

J. Wu ◽

T‐T. Wong ◽

L‐F. Wang ◽

M‐T. Chuan

Keyword(s):

T Cell ◽

Bullous Pemphigoid ◽

Cytokine Production ◽

Interleukin 8 ◽

Blister Fluid ◽

Soluble Cd8 ◽

The Relationship ◽

Soluble Cd4 ◽

Local Cytokine

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Access to the Spleen Microenvironment through Lymph Shows Local Cytokine Production, Increased Cell Flux, and Altered Signaling of Immune Cells during Lipopolysaccharide-Induced Acute Inflammation

The Journal of Immunology ◽

10.4049/jimmunol.0902049 ◽

2010 ◽

Vol 184 (8) ◽

pp. 4547-4556 ◽

Cited By ~ 35

Author(s):

Elvira Semaeva ◽

Olav Tenstad ◽

Jørn Skavland ◽

Marianne Enger ◽

Per Ole Iversen ◽

...

Keyword(s):

Immune Cells ◽

Acute Inflammation ◽

Cytokine Production ◽

Local Cytokine

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Exacerbates kidney injury in mice subjected to sepsis: critical impact of used heparin

American Journal of BioMedicine ◽

10.18081/2333-5106/018-323-335 ◽

2018 ◽

Vol 6 (4) ◽

pp. 323-335

Author(s):

Peter Weighardt ◽

Niels Hayashi

Keyword(s):

Oxidative Stress ◽

Acute Kidney Injury ◽

Cytokine Production ◽

Multiorgan Failure ◽

Kidney Injury ◽

Tubular Dysfunction ◽

Cytokine Storm ◽

Peritubular Capillary ◽

Inflammatory Reactions ◽

Local Cytokine

Sepsis is the leading cause of death in critically ill patients, and the incidence of sepsis is increasing causes multiorgan failure, including acute kidney injury (AKI) and patients with both sepsis and AKI have an especially high mortality rate. Several different pathophysiological mechanisms have been proposed for sepsis-induced AKI: vasodilation-induced glomerular hypoperfusion, dysregulated circulation within the peritubular capillary network, inflammatory reactions by systemic cytokine storm or local cytokine production, and tubular dysfunction induced by oxidative stress animal sepsis models have been developed using LPS infusion. Renal dysfunction evaluated by serum creatinine and BUN was found in acute non-survivors (<24 hours) and decreased urine output in subacute non-survivors (24–96 hours). The study show that increased AKI in animal with blood collected heparin (P=0.002) compared to citrate, furthermore; sham mice that received heparin did not develop AKI.

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