scholarly journals Effect of ultra-disperse powder оf zirconate-titanate lead on morphological and functional status of kidney studied experimentally

2007 ◽  
Vol 6 (1) ◽  
pp. 73-77
Author(s):  
N. M. Shevtsova ◽  
V. V. Novitsky ◽  
Ye. A. Stepovaya ◽  
S. V. Nizkodubova
Keyword(s):  
Toxic Effect ◽  
Functional Status ◽  
Chemical Elements ◽  
Disperse Powder ◽  
Single Element ◽  
Zirconate Titanate ◽  
Rat Experiments ◽  
Titanate Lead

Rat experiments revealed that daily inhalation of ultra-disperse powder (UDP) оf zirconate-titanate lead in a dose of 15 mg/m3 re-sult in increased contents of single element of UPD in kidneys and cause changes of morphological and functional status of the or-gan. According to accumulation degree in kidneys, the studied chemical elements are situated in the following sequence: Pb > Ti > Cr > Mn. The presented results are indicative of moderately expressed toxic effect of UDP of circonate-titanate lead on morphological and functional status of rat's kidney, been subjected to its inhalation influence.

Thermal expansion behavior and estimated total polarizations of lead zirconate titanate–lead nickel niobate ceramics

2010 ◽  
Vol 64 (18) ◽  
pp. 1960-1963 ◽  
Author(s):  
Muangjai Unruan ◽  
Anurak Prasatkhetragarn ◽  
Yongyut Laosiritaworn ◽  
Supon Ananta ◽  
Orawan Khamman ◽  
...  
Keyword(s):  
Thermal Expansion ◽  
Lead Zirconate Titanate ◽  
Lead Zirconate ◽  
Thermal Expansion Behavior ◽  
Zirconate Titanate ◽  
Expansion Behavior ◽  
Lead Nickel ◽  
Titanate Lead

Dielectric and ferroelectric properties of lead zirconate titanate-lead nickel niobate ceramics under compressive stress

2009 ◽  
Vol 105 (8) ◽  
pp. 084111 ◽  
Author(s):  
M. Unruan ◽  
A. Prasartketrakarn ◽  
A. Ngamjarurojana ◽  
Y. Laosiritaworn ◽  
S. Ananta ◽  
...  
Keyword(s):  
Compressive Stress ◽  
Lead Zirconate Titanate ◽  
Ferroelectric Properties ◽  
Lead Zirconate ◽  
Zirconate Titanate ◽  
Lead Nickel ◽  
Titanate Lead

Piezoelectric and pyroelectric behavior of lead zirconate titanate–lead sulfide–glass nanocomposites

1999 ◽  
Vol 14 (5) ◽  
pp. 1796-1800
Author(s):  
T. K. Kundu ◽  
D. Chakravorty ◽  
L. E. Cross
Keyword(s):  
Lead Zirconate Titanate ◽  
Lead Sulfide ◽  
Sol Gel ◽  
Lead Zirconate ◽  
Base Glass ◽  
Zirconate Titanate ◽  
Saturation Polarization ◽  
Gel Technique ◽  
Sulfide Glass ◽  
Titanate Lead

Composites containing micron-sized lead zirconate titanate and nanometer-sized lead sulfide in a glass matrix were prepared using a sol-gel technique. The PbS size ranged from 3.5 to 15.3 nm. It was possible to pole some of these nanocomposites. The measured values of saturation polarization, remanent polarization, and coercive field showed an increase as the PbS particle size was enhanced. The composites exhibited improvement in the piezoelectric and pyroelectric behavior as compared to that of the base glass-ceramic.

Schottky Barrier Heights of Tantalum Oxide, Barium Strontium Titanate, Lead Zirconate Titanate and Strontium Bismuth Tantalate

1998 ◽  
Vol 541 ◽  
Author(s):  
J Robertson ◽  
C W Chen
Keyword(s):  
Schottky Barrier ◽  
Strontium Titanate ◽  
Lead Zirconate Titanate ◽  
Barium Strontium Titanate ◽  
Lead Zirconate ◽  
Strontium Bismuth Tantalate ◽  
Barrier Heights ◽  
Barium Strontium ◽  
Zirconate Titanate ◽  
Titanate Lead

AbstractSchottky barrier heights of various metals on tantalum pentoxide, barium strontium titanate, lead zirconate-titanate and strontium bismuth tantalate have been calculated as a function of metal work function. These oxides have a dimensionless Schottky barrier pinning factor, S, of 0.28 – 0.4 and not close to 1, because S is controlled by the Ti-O type bonds not Sr-O type bonds, as assumed previously. Band offsets on silicon are asymmetric with much smaller offset at the conduction band, so that Ta2O5 and barium strontium titanate (BST) are relatively poor barriers to electrons on Si.

scholarly journals Inhibition of human monocyte adhesion by interleukin-4

Blood ◽  
1991 ◽  
Vol 77 (12) ◽  
pp. 2739-2745
Author(s):  
MJ Elliott ◽  
JR Gamble ◽  
LS Park ◽  
MA Vadas ◽  
AF Lopez
Keyword(s):  
T Cell ◽  
Toxic Effect ◽  
Functional Status ◽  
Human Monocyte ◽  
Surface Expression ◽  
Interleukin 4 ◽  
Cytokine Receptors ◽  
Common Mechanism ◽  
Monocyte Adhesion ◽  
Inhibitory Effects

The adhesion of monocytes to vascular surfaces is central to inflammation and atherogenesis; however, very little is known about regulatory factors that can prevent these processes. Here we report the inhibition of human monocyte adhesion to human endothelial layers and plastic by interleukin-4 (IL-4), a T-cell-derived glycoprotein with pleiotropic activities. The inhibitory effects of IL-4 were seen with basal and cytokine-stimulated monocyte adhesion, were apparent at low concentration, and were abolished by inactivating IL-4. No direct toxic effect of IL-4 on monocytes was detected. Inhibition of adhesion was accompanied by small increases in monocyte surface expression of the leukocyte-functional antigen group of adhesion structures, suggesting that absolute levels of expression may be less important than the functional status of such molecules in the regulation of monocyte adhesion. In addition, inhibition by IL-4 of cytokine-stimulated monocyte adhesion was not associated with changes in the surface expression of cytokine receptors. These results suggest a role for IL-4 in the regulation of monocyte adhesion, and may provide for a common mechanism for the inhibitory effects of IL-4 on monocyte function.

scholarly journals Inhibition of human monocyte adhesion by interleukin-4

Blood ◽  
1991 ◽  
Vol 77 (12) ◽  
pp. 2739-2745 ◽  
Author(s):  
MJ Elliott ◽  
JR Gamble ◽  
LS Park ◽  
MA Vadas ◽  
AF Lopez
Keyword(s):  
T Cell ◽  
Toxic Effect ◽  
Functional Status ◽  
Human Monocyte ◽  
Surface Expression ◽  
Interleukin 4 ◽  
Cytokine Receptors ◽  
Common Mechanism ◽  
Monocyte Adhesion ◽  
Inhibitory Effects

Abstract The adhesion of monocytes to vascular surfaces is central to inflammation and atherogenesis; however, very little is known about regulatory factors that can prevent these processes. Here we report the inhibition of human monocyte adhesion to human endothelial layers and plastic by interleukin-4 (IL-4), a T-cell-derived glycoprotein with pleiotropic activities. The inhibitory effects of IL-4 were seen with basal and cytokine-stimulated monocyte adhesion, were apparent at low concentration, and were abolished by inactivating IL-4. No direct toxic effect of IL-4 on monocytes was detected. Inhibition of adhesion was accompanied by small increases in monocyte surface expression of the leukocyte-functional antigen group of adhesion structures, suggesting that absolute levels of expression may be less important than the functional status of such molecules in the regulation of monocyte adhesion. In addition, inhibition by IL-4 of cytokine-stimulated monocyte adhesion was not associated with changes in the surface expression of cytokine receptors. These results suggest a role for IL-4 in the regulation of monocyte adhesion, and may provide for a common mechanism for the inhibitory effects of IL-4 on monocyte function.

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