scholarly journals COTININE LEVEL AS A BIOCHEMICAL INDICATOR OF THE TOXIC EFFECTS OF PASSIVE SMOKING EXPOSURE ON INFANTS’ ANTHROPOMETRIC MEASURES

2021 ◽  
Vol 21 (1) ◽  
pp. 55-68
Author(s):  
Medhat Saleh ◽  
Ramadan Hamza ◽  
Osama El-Asheer ◽  
Doaa El Shehaby ◽  
Ahmed Ibrahim
Keyword(s):  
Passive Smoking ◽  
Toxic Effects ◽  
Anthropometric Measures ◽  
Cotinine Level ◽  
Biochemical Indicator ◽  
Smoking Exposure

scholarly journals Passive Smoking and Pediatric Dental Caries

2021 ◽  
Vol 39 (1) ◽  
pp. 1-13
Author(s):  
Sirajam Munira ◽  
Md Ziaul Islam ◽  
Md Towfiq Sekander ◽  
Badrul Alam
Keyword(s):  
Dental Caries ◽  
Passive Smoking ◽  
Cross Sectional Study ◽  
Speech Development ◽  
Cross Sectional ◽  
Effective Measure ◽  
Urinary Cotinine ◽  
Cotinine Level ◽  
Smoking Exposure ◽  
The Mean

Background: Dental caries is a chronic dental disorder of children and it can result in substantial morbidity due to pain, dysfunction, poor appearance, and problems of speech development. The study was conducted to assess the association between passive smoking and pediatric dental caries. Methods: The cross-sectional study was conducted among 92 school children aged 5-10 years. Data on exposure to passive smoking at both indoor and outdoor were obtained through face to face interview of the parents with the help of semi-structured questionnaire and checklist. Dental caries was diagnosed by clinical examination and passive smoking exposure was assessed by estimated urinary cotinine level. Results: The study revealed that 58.70% children were male and 41% were female respectively. The mean age of the children was 7.20 years. It was found that 59.8% father of the studied children were smoker where 68.5% children reported that some of their family members were indoor smokers. Of all the children, 62% had dental caries and 85.5% of the children had dental caries who were exposed to passive smoking and had significant association between exposure to passive smoking and dental caries (p<0.001). Dental caries was common among children aged 5-6years who were exposed to passive smok ing. The mean urinary cotinine level was 27.81 ng/ml and maximum children (65.2%) had urinary cotinine level more than 10ng/ml who was exposed to passive smoking. The study found significant positive correlation between urinary cotinine concentrations and the duration of exposure to passive smoking (i=0.692, p<0.001). Conclusion: This study suggests reduction of exposure to passive smoking may be an effective measure for prevention of pediatric dental caries. JOPSOM 2020; 39(1): 1-13

Passive Smoking Exposure

CHEST Journal ◽  
2002 ◽  
Vol 122 (3) ◽  
pp. 1086-1090 ◽  
Author(s):  
Katja Radon ◽  
Kerstin Bu¨sching ◽  
Joachim Heinrich ◽  
H.-E. Wichmann ◽  
Rudolf A. Jo¨rres ◽  
...  
Keyword(s):  
Passive Smoking ◽  
Smoking Exposure

Increased prevalence of asthma and allied diseases among active adolescent tobacco smokers after controlling for passive smoking exposure. A cause for concern?

2004 ◽  
Vol 34 (7) ◽  
pp. 1017-1023 ◽  
Author(s):  
I. Annesi-Maesano ◽  
M. P. Oryszczyn ◽  
C. Raherison ◽  
C. Kopferschmitt ◽  
G. Pauli ◽  
...  
Keyword(s):  
Passive Smoking ◽  
Smoking Exposure

291 EFFECTIVENESS OF CONTROL MEASURES TO REDUCE PASSIVE SMOKING EXPOSURE IN OFFICE BUILDINGS

Epidemiology ◽  
1995 ◽  
Vol 6 (2) ◽  
pp. S55
Author(s):  
K Liu ◽  
L E Alevantis ◽  
S B Hayward ◽  
K B Shah ◽  
E Huang ◽  
...  
Keyword(s):  
Passive Smoking ◽  
Control Measures ◽  
Office Buildings ◽  
Smoking Exposure

Passive smoking exposure and risk of COPD in China – Authors' reply

The Lancet ◽  
2008 ◽  
Vol 371 (9608) ◽  
pp. 201-202
Author(s):  
Peng Yin ◽  
Peymane Adab ◽  
KK Cheng
Keyword(s):  
Passive Smoking ◽  
Smoking Exposure

1067 GENDER DIFFERENCE IN THE RELATIONSHIP BETWEEN LONG TERM PASSIVE SMOKING EXPOSURE AND HDL-CHOLESTEROL IN LATE ADOLESCENCE

2012 ◽  
Vol 30 ◽  
pp. e310
Author(s):  
Chi Le-Ha ◽  
Lawrence J. Beilin ◽  
Sally Burrows ◽  
Rae-Chi Huang ◽  
Wendy H. Oddy ◽  
...  
Keyword(s):  
Gender Difference ◽  
Passive Smoking ◽  
Hdl Cholesterol ◽  
Late Adolescence ◽  
Smoking Exposure ◽  
The Relationship

scholarly journals Influence of Time and Frequency of Passive Smoking Exposure on Mucociliary Clearance and the Autonomic Nervous System

2015 ◽  
Vol 61 (4) ◽  
pp. 453-461 ◽  
Author(s):  
A. P. C. Freire ◽  
D. Ramos ◽  
M. R. Leite ◽  
B. S. Silva ◽  
R. M. David ◽  
...  
Keyword(s):  
Nervous System ◽  
Autonomic Nervous System ◽  
Passive Smoking ◽  
Mucociliary Clearance ◽  
Autonomic Nervous ◽  
Smoking Exposure

scholarly journals Health Risk of Environmental Tobacco Smoke (ETS)

2013 ◽  
Author(s):  
Dewan Raja ◽  
Bahar Sultana
Keyword(s):  
Public Health ◽  
Environmental Tobacco Smoke ◽  
Tobacco Smoke ◽  
Passive Smoking ◽  
The Elderly ◽  
Second Hand Smoke ◽  
Potential Health ◽  
Secondhand Smoking ◽  
Smoking Exposure ◽  
Home Area

Environmental tobacco smoke is a preventable cause of significant morbidity and mortality among non-smokers. Reducing exposure to tobacco smoke is an essential community and public health objective. This report documents the substantial evidence characterizing the health risks caused by exposure to passive smoking. Multiple major reviews of evidence have concluded that environmental tobacco smoke is a known human carcinogen and that exposure to passive smoke causes adverse effects like SIDS, congenital birth defects, lead poisoning, and lung cancer. Regrettably, reductions in exposure have been slower among small children than among adults, as growing workplace restriction protects the majority of adults while the homes remain the most important source of exposure for children. The relationship between passive smoking exposure and potential health hazards to all ages of population were researched. We analyzed and systematically reviewed information from multiple literature sources. We found that everyone from the fetus to the elderly is significantly affected by passive smoking. Like firsthand smoking, secondhand smoking is also hazardous. Health education and regulations regarding secondhand smoking in both the office and home area can improve public health. This paper indicates a critical need for second hand smoke reduction interventions especially among vulnerable populations.

scholarly journals Modified effect of active or passive smoking on the association between age and abdominal aortic calcification: a nationally representative cross-sectional study

BMJ Open ◽  
2021 ◽  
Vol 11 (10) ◽  
pp. e047645
Author(s):  
Luyan Lv ◽  
Shixian Wu ◽  
Yungui Yang ◽  
Xiongli Yue
Keyword(s):  
Vascular Calcification ◽  
Passive Smoking ◽  
Cross Sectional Study ◽  
Serum Cotinine ◽  
Cross Sectional ◽  
Age Related ◽  
Abdominal Aortic ◽  
Smoking Exposure ◽  
Nationally Representative ◽  
Never Smokers

ObjectiveThe deleterious effects of smoking on atherosclerosis were well known; however, the interaction among ageing, smoking and atherosclerosis remains unclear. This study tested the hypothesis that the association between age and vascular calcification, a critical mark of atherosclerosis, was modified by smoking.DesignCross-sectional study.SettingA nationally representative sample, the National Health and Nutrition Examination Surveys 2013–2014.ParticipantsThis study included 3140 adults aged 40–80 years with eligible data for abdominal aortic calcification (AAC). Active and passive smoking exposure was identified through self-reports and tobacco metabolites (serum cotinine and urinary 4-methylnitrosamino-3-pyridyl-1-butanol).Primary outcome measuresAAC score was determined using dual-energy X-ray absorptiometry (DXA) scans. OR was estimated using the logistic regression method to assess the association between age and the presence of severe or subclinical AAC stratified by smoking exposure. The survey-weighted Wald test was used to evaluate potential interactions.ResultsAAC was positively associated with age in the general population. After adjustment for age, sex, race/ethnicity and other cardiovascular risk factors, age was significantly associated with the odds of severe AAC (OR for each 5-year increase in age: 1.66, 95% CI 1.48 to 1.87, p<0.001). As expected, the association between age and vascular calcification was especially stronger in smokers than in never smokers (p value for interaction ≤0.014). According to spline fitting, the progression of vascular calcification was significantly increased after 45 years in smokers compared with that after 60 years in never smokers. Quitting smoking may compromise the deleteriousness of the vascellum especially in younger adults. However, the difference in age-related calcification among never smokers with or without secondhand smoke exposure was minor, regardless of the definition by self-report, serum cotinine, or urinary 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanol.ConclusionsSmoking significantly accelerated the progression of age-related subclinical atherosclerosis. Early smoking cessation should be encouraged among young smokers. The effect of passive smoking exposure on arteriosclerosis should be assessed further.

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