Therapeutic Management of Right Side Congestive Heart Failure in a Dachshund Bitch

2016 ◽  
Vol 12 (2) ◽  
Author(s):  
D. J. Talukdar ◽  
K. Sharma ◽  
H. Bayan ◽  
F. A. Ahmed ◽  
G. Das ◽  
...  
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Mitral Valve ◽  
Clinical Signs ◽  
Mitral Valve Insufficiency ◽  
Peripheral Region ◽  
Acute Onset ◽  
The Body ◽  
Heartworm Disease ◽  
Valve Insufficiency

Congestive Heart Failure (CHF) is a term that refers to the heart’s inability to pump adequate blood to the body. There are many causes of CHF in dogs viz. mitral valve insufficiency (MVI) or a leaky mitral valve, dilated cardiomyopathy (Haggstrom, 2010) and chronic heartworm disease. Clinical signs vary depending on whether the dog has left- or right-sided heart failure. Right-sided congestive heart failure (RS-CHF) causes poor venous return to the heart, accumulation of fluid in the peritoneum leading to ascites. Fluid also leaks from the veins in the limbs, causing oedema of the peripheral region. In animals, Right-sided congestive heart failure (RS-CHF) has previously been described in dogs (McIntosh and McEntee, 1995), cats and also in ferret (Haggstrom, 2010). The reported canine cases were among young to middle-aged male dogs (Bull mastiff, Siberian husky and dachshund), with acute-onset congestive right-sided heart failure or syncope as the presenting complaints. The present case was diagnosed as right side heart failure in a six years old female Dachshund dog with pleural effusion

scholarly journals Mitral Valve Disease in Thyroid Storm

2021 ◽  
Vol 5 (Supplement_1) ◽  
pp. A941-A942
Author(s):  
Sandra Rocio Rivera Menjura ◽  
Lia G Moyano Rivas ◽  
Camila Parraguez Gamboa ◽  
Cristobal Balmaceda ◽  
Juan P Peralta ◽  
...  
Keyword(s):  
Heart Failure ◽  
Mitral Valve ◽  
Invasive Mechanical Ventilation ◽  
Mitral Valve Insufficiency ◽  
Left Ventricular ◽  
Thyroid Storm ◽  
Multiple Infections ◽  
Mitral Insufficiency ◽  
Type I ◽  
Valve Insufficiency

Abstract Introduction: The cardiovascular effects that thyroid gland causes are widely studied. In fact, there is a known correlation between Graves’ Disease and mitral valve damage. We present the case of a patient admitted with thyroid storm and heart failure associated with severe structural damage of the mitral valve papillary muscle. Case Report: 24 year old woman with hyperthyroidism diagnosed 12 years ago, treated irregularly with thiamazole and propranolol, leaving treatment a year ago, presents dyspnea, class III functional capacity, diarrhea and logic dysphagia of a month of evolution. Heart rate over 170 bpm, respiratory rate 48 rpm and blood pressure 143/84 mmHg. Physical exam positive for exophthalmos, grade III goiter, crackles in both lung bases, pretibial myxedema and fulfilling criteria for a thyroid storm (65 points in Burch-Wartofsky Point Scale). First Lab Results: TSH<0.005µU/mL, free T4>7.7ng/dl and TRAB 37.8UI/L. Chest ray: Global cardiomegaly and pulmonary edema. EKG: Narrow complex supraventricular tachycardia. Thyroid ultrasound: Intrathoracic goiter. Transesophageal echocardiogram: Severe mitral insufficiency (Carpentier Type I and IIIB), right cavities and left ventricular enlargement, preserved right ventricular function and severe pulmonary hypertension (PSAP 71-76 mmHg). First treated with thiamazole, hydrocortisone IV, cholestyramine and sedation, falling time after into ventilatory failure and developing delirium, requiring invasive mechanical ventilation. Tested positive for COVID- 19. Starts preparation with Lugol and undergoes Total Thyroidectomy. After surgery develops severe hypocalcemia secondary to transitory hypoparathyroidism. During hospitalization presents multiple infections including pneumonia (Pseudomonas Aeruginosa), lung aspergillosis, bacteriuria (Enteroccocus Faecium) and candiduria (Candida Albicans and Glabrata), each one treated with multiple antibiotics and vasoactive drugs. Once stable, mitral valve replacement is realized, after which, the patient progresses favorably being discharged with programmed ambulatory controls. Conclusion: We report a case of a patient who was presented with positive thyroid storm criteria associated with heart failure and severe mitral valve insufficiency. The case gets complicated as multiple infections take place, including COVID-19. Fortunately, because of the early and aggressive multidisciplinary management, the patient evolved favorably, overcoming the life-threatening conditions she went through. Key Words: Thyroid storm, mitral valve insufficiency, heart failure. Bibliography: Klein I, Danzi S. Thyroid disease and the heart. Circulation. 2007 Oct 9;116(15):1725-35. doi: 10.1161/CIRCULATIONAHA.106.678326. Erratum in: Circulation. 2008 Jan 22;117(3):e18. PMID: 17923583.

scholarly journals MORPHOFUNCTIONAL FEATURES OF THE MYOCARDIUM IN ITS DIASTOLIC OVERLOAD

2021 ◽  
Vol 17 (2) ◽  
pp. 39-46
Author(s):  
A.A. Balabai ◽  
A.A. Krikunov ◽  
P.M. Semeniv ◽  
E.V. Rudenko ◽  
V.P. Zakharova
Keyword(s):  
Heart Failure ◽  
Mitral Valve ◽  
Left Ventricle ◽  
Blood Volume ◽  
Interstitial Fibrosis ◽  
Mitral Valve Insufficiency ◽  
Diastolic Filling ◽  
Myocardial Oxygen Demand ◽  
Statistical Research ◽  
Valve Insufficiency

Relevance. Adaptation of the heart chambers to their volume overload, which increases with diastolic filling of the left ventricle (LV) or systolic regurgitation in the left atrium (LA) in case of mitral valve insufficiency leads to remodeling of the myocardium of these chambers. Longitudinal hypertrophy of cardiomyocytes (CMCs), realized by building up new sarcomeres at the end of existing myofibrils, is an adaptation mechanism in the early stages, but eventually turns into maladjustment, which leads to heart failure. Hypertrophied CMCs, damaged during decompensation, are replaced by connective tissue due to excessive activation of fibroblasts with deposition of the extracellular matrix, which is also an element of the myocardial remodeling. The progression of heart failure is also associated with a mismatch between blood supply and myocardial oxygen demand, since an increase in the size of the CMCs is accompanied by a rarefaction of the intramural network of microvessels. It is believed that the violation of the ratio of the size of the heart, angiogenesis and cardiac function are the basis for the transition of adaptive compensation of the heart to decompensation with the progression of heart failure. Objective: to study morphological changes in the myocardium of the LV and LA in patients with mitral valve insufficiency. Materials and methods. Macroscopically, the condition of the myocardium was studied on the material of 14 autopsies of patients who died of NdMK insufficiency. History of NdMK – from 3 months. up to 2.4±1.1 years. As a control, the hearts of 3 deaths without cardiopathology were studied. The material for light microscopy was pieces of myocardium from different segments of the left ventricle, as well as from the walls of the left ventricle, obtained during autopsy.Morphological (macroscopic, histological and electron microscopic), morphometric and statistical research methods were used. Results. With LV dilatation associated with chronic mitral valve insufficiency, lengthening of each CMCs provides an increase in the area of ​​the myocardial walls, and, accordingly, the size of the cavity of the corresponding chamber of the heart, which compensates to some extent for the increase in diastolic blood volume in the LV and systolic blood volume in the LA. However, the factor limiting this compensation mechanism is the deficiency of the myocardial microvasculature associated with limited capillary growth. The contradiction between the need for the myocardium to lengthen the CMCs and the inability of capillaries to provide them with oxygen leads to a breakdown in compensation with an increase in fibrotic changes. This is a factor limiting the further increase in the volume of the cavity. Conclusions. Overloading of the myocardium with volume leads to an increase in the length of the CMCs, on average, from 57.3±9.1 µm to 93.7±12.4 µm. The increase in the length of the CMCs is due to the increase in the number of sarcomeres from 43.7±8.4 to 62.5±14.5. The diameter of the CMCs in this case does not increase reliably. Overloading of the heart cavities with volume is often accompanied by desynchronization of the CMCs contraction, which leads to disruption of the integrity of the myocytic "working syncytium" and pronounced interstitial fibrosis.

scholarly journals Comparative assessment of fetal blood flow and cardiac system status of pregnant women with mitral valve pathology

2019 ◽  
Vol 23 (4) ◽  
pp. 645-651
Author(s):  
A.P. Dnistryanska ◽  
Yu. Konovalenko ◽  
N.A. Godlevskaya ◽  
N.G. Burtyak ◽  
L.S. Baida
Keyword(s):  
Blood Flow ◽  
Mitral Valve ◽  
Pregnant Women ◽  
Mitral Valve Prolapse ◽  
Volume Ratio ◽  
Mitral Valve Insufficiency ◽  
The Body ◽  
Cardiovascular Pathology ◽  
Pregnancy And Childbirth ◽  
Valve Insufficiency

Annotation. The aim of this work is to analyze the course of pregnancy in women with mitral pathology, the task is to identify preclinical criteria for worsening cardiovascular pathology that can lead to the development of complications of pregnancy and functioning of the fetal CVS, to evaluate the effectiveness of preventive measures. The article presents the analysis of changes in ultrasound characteristics of fetoplacental blood flow in women with mitral valve prolapse and mitral valve insufficiency, as well as cardiovascular status in these women. Statistical processing was performed by calculating Student coefficients (+) using the Microsoft Excel PC program. Doppler examination of fetal umbilical cord blood flow during pregnancy was studied. The correlation between the level of maternal blood flow compensation and pathological changes in the fetoplacental complex was established. As a result of our work, the prevalence of the disease was estimated. As of 2018, it was found that of the total number of extragenital pathology in 1676 pregnant women with CVSD; respectively for 2017 — 1699, for 2016 — 1925, 2015 — 1857 in the region. Of these, 331 (2018), 313 (2017), 326 (2016), 306 (2015) had mitral valve defects, including prolapse. A retrospective assessment of pregnancy and childbirth indicates the possibility of complications of pregnancy and childbirth. In the group of patients with mitral valve prolapse and mitral valve insufficiency, compared to the control group, they did not show significant changes in the dynamics of the Doppler metrics of cardiovascular pathology, which indicates the compensation of the cardiovascular system against the background of changes in the valve. At the same time, the tendency to decrease of CF in women with mitral valve prolapse and insufficiency of the mitral valve against the background of increasing pregnancy is revealed, indicates a violation of CVD adaptation to changes inherent in pregnancy, which provoke hemic and hemodynamic changes in the body, which will cause pregnancy and fetal status. FPC status in women with mitral valve prolapse and mitral valve insufficiency worsens in the third trimester of pregnancy, especially indicators such as the ratio of systolic volume ratio (SVR) to diastolic volume ratio (DVR) and IR. The administration of 40 mg of ƅ-blockers for a 2-week period improved the FPC, was safe and effective.

scholarly journals Staged Endovascular Repair of Aortic Coarctation followed by Double Valve Surgery

Aorta ◽  
2021 ◽  
Author(s):  
Dimitrios V. Avgerinos ◽  
Rajeev Dayal ◽  
Charles Mack ◽  
Samuel Lang ◽  
Konstantinos S. Mylonas
Keyword(s):  
Heart Failure ◽  
Mitral Valve ◽  
Endovascular Repair ◽  
Aortic Coarctation ◽  
Coarctation Of The Aorta ◽  
Mitral Valve Insufficiency ◽  
Valve Surgery ◽  
Valve Repair ◽  
Unique Case ◽  
Valve Insufficiency

AbstractWe present a unique case of late diagnosis of coarctation of the aorta in an adult, presenting with congestive heart failure associated with severe aortic and mitral valve insufficiency. To minimize operative risk, staged endovascular repair of the coarctation was initially performed, followed by aortic valve replacement and mitral valve repair. Six months postoperatively, the 41-year-old patient remains completely asymptomatic.

Anomalous mitral arcade leading to congenital mitral insufficiency with an associated anomaly of the tricuspid valve

1992 ◽  
Vol 2 (4) ◽  
pp. 357-358 ◽  
Author(s):  
Jyoti V. Mandke ◽  
Vikas R. Dharnidharka ◽  
Vasundhara P. Sanzgiri
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Mitral Valve ◽  
Mitral Regurgitation ◽  
Tricuspid Valve ◽  
Acute Onset ◽  
Mitral Insufficiency

SummaryA 21-day-old girl presented with congestive heart failure of acute onset and Doppler evidence of mitral regurgitation, which subsequently proved fatal. Necropsy revealed an anomalous arcade lesion of the mitral valve. The tendinous cords of the tricuspid valve were also abnormal, a finding, to the best of our knowledge, not reported before.

Analysis of certain blood biochemical parameters in relation to oxidative stress in chronic mitral valve insufficiency of dogs with heart failure

2018 ◽  
Author(s):  
M.S. Indhu ◽  
P.S.L. Sesh ◽  
K. Loganathasamy ◽  
K. Jeyaraja ◽  
K. Padmanath ◽  
...  
Keyword(s):  
Heart Failure ◽  
Free Radicals ◽  
Mitral Valve ◽  
Biochemical Parameters ◽  
Hematological Parameters ◽  
Mitral Valve Insufficiency ◽  
Cardiac Damage ◽  
Blood Biochemical ◽  
Valve Insufficiency ◽  
Healthy Dogs

Chronic mitral valve insufficiency (CMVI) is the most common acquired heart disease in dogs. In heart failure, the cellular oxygenation and metabolism are affected, which leads to the production of free radicals. Free radicals damage DNA, lipid and protein molecules in cells. In the present experiment, blood samples were collected from CMVI dogs with heart failure and were compared with the results obtained from healthy dogs. A significant increase in the levels of xanthine oxidase, AST, LDH and CK and decrease in the activity of catalase were noticed in CMVI dogs when compared to healthy dogs, which revealed overall cardiac and skeletal muscle damage in CMVI dogs. Results of biochemical parameters revealed an increase in urea level and decrease in sodium, potassium, and calcium levels in CMVI dogs as compared to control dogs, all of which indicate cardiac damage in dogs. Study on hematological parameters revealed a significant decrease in Hb, PCV, RBC and platelet counts and an increase in total WBC counts and percentage of neutrophils, decrease in percentage of the lymphocyte and monocyte in CMVI dogs than control. These results indicate secondary phenomenon to heart failure. The present research data indicates the usefulness of these biomarkers in the diagnosis and prognosis of CMVI with heart failure in dogs.

A Complex Mitral Valve Reconstruction: A Case Report

2005 ◽  
Vol 6 (2) ◽  
pp. 27
Author(s):  
Dimitrios Buklas ◽  
Massimo Massetti ◽  
Eric Saloux ◽  
Eugenio Neri ◽  
Olivier LePage ◽  
...  
Keyword(s):  
Mitral Valve ◽  
Surgical Techniques ◽  
Mitral Valve Insufficiency ◽  
Left Ventricular ◽  
Mitral Valve Reconstruction ◽  
Mitral Repair ◽  
Valve Reconstruction ◽  
Valve Insufficiency ◽  
Valve Function ◽  
Valvular Function

Several techniques are currently in use for mitral valve reconstruction. We report a mitral repair case in which the use of a combination of different surgical techniques resulted in the necessary correction. A 47-year-old woman underwent surgical intervention to treat severe mitral valve insufficiency due to A1/A2/A3 and P2 prolapsed valve tissue. A combination of quadrangular resection, sliding leaflet, single chordal transposition, "flip-over" leaflet, and ring annuloplasty techniques were applied, and postsurgical correct valve function was documented by results of a left ventricular saline filling test and transesophageal echocardiography control. Complex mitral valve repairing techniques can be combined to reestablish valvular function.

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