scholarly journals RELATIONS BETWEEN PRE- AND POST-ANAEROBIC OXYGEN CONSUMPTION AND OXYGEN TENSION IN SOME FRESH WATER SNAILS

1953 ◽  
Vol 104 (3) ◽  
pp. 301-312 ◽  
Author(s):  
THEODOR VON BRAND ◽  
BENJAMIN MEHLMAN
Keyword(s):  
Oxygen Consumption ◽  
Fresh Water ◽  
Oxygen Tension

scholarly journals The Control of Respiratory Movements in Crustacea by Oxygen and Carbon Dioxide

1934 ◽  
Vol 11 (1) ◽  
pp. 1-10
Author(s):  
H. MUNRO FOX ◽  
M. L. JOHNSON
Keyword(s):  
Carbon Dioxide ◽  
Oxygen Consumption ◽  
Fresh Water ◽  
Oxygen Tension ◽  
Sea Water ◽  
Respiratory Rhythm ◽  
Marine Species ◽  
Carbon Dioxide Tension ◽  
Respiratory Movements ◽  
Rate Of Movement

1. The respiratory movements of the barnacle Balanus and of the phyllopod Cheirocephalus are not accelerated either by decreased oxygen or by increased carbon dioxide tension in the water. 2. The rate of movement of the scaphognathite of the crayfish Astacus is accelerated by a fall in oxygen but not by a rise in carbon dioxide tension of the environment. 3. The rate of movement of the pleopods of the fresh-water isopod Asellus is accelerated by a fall in oxygen but not by a rise of carbon dioxide tension. 4. In the sea-shore isopod Ligia, submerged in sea water, the rate of pleopod movement is not accelerated by a decrease in oxygen tension below that in equilibrium with air, but a rise in oxygen tension above the latter value slows respiratory movements. Carbon dioxide has no accelerating effect. 5. Both a decrease in oxygen and an increase in carbon dioxide tension accelerate the respiratory pleopod movements of the amphipods Gammarus pulex and G. locusta. 6. Whereas in the fresh-water G. pulex the quickened respiratory rhythm is permanent at each decreased oxygen and increased carbon dioxide tension, in the marine G. locusta these changes in rate of beat are transitory. This is correlated with the greater oxygen consumption of the marine species.

Local blood flow, oxygen tension, and oxygen consumption in the rat spinal cord

1983 ◽  
Vol 58 (4) ◽  
pp. 526-530 ◽  
Author(s):  
Nariyuki Hayashi ◽  
Barth A. Green ◽  
Mayra Gonzalez-Carvajal ◽  
Joseph Mora ◽  
Richard P. Veraa
Keyword(s):  
Spinal Cord ◽  
Blood Flow ◽  
Oxygen Consumption ◽  
Oxygen Tension ◽  
Tissue Oxygen ◽  
Local Blood Flow ◽  
Rat Spinal Cord ◽  
Spinal Cord Blood Flow ◽  
Content Type ◽  
Tissue Oxygen Consumption

✓ Using a reliable and reproducible microelectrode technique, consistent simultaneous measurements of local spinal cord blood flow (SCBF), tissue oxygen tension, and tissue oxygen consumption were made at cervical, thoracic, and lumbar levels in the rat spinal cord. These observations showed that the metabolic state is maintained constant along the cord, despite significant variations in vasculature. The physiological and anatomical aspects of these findings are discussed.

Oxygen consumption in response to chloride uptake in the fresh water field crab

Life Sciences ◽  
1969 ◽  
Vol 8 (13) ◽  
pp. 739-743
Author(s):  
V. Venkata Reddy ◽  
P.R. Aravinda Babu
Keyword(s):  
Oxygen Consumption ◽  
Fresh Water ◽  
Chloride Uptake ◽  
Water Field

Cardiac performance as a function of intracellular oxygen tension in buffer-perfused hearts

2001 ◽  
Vol 281 (6) ◽  
pp. H2463-H2472 ◽  
Author(s):  
Kenneth A. Schenkman
Keyword(s):  
Oxygen Consumption ◽  
Guinea Pig ◽  
Oxygen Tension ◽  
Myocardial Oxygen Consumption ◽  
Arterial Oxygen Tension ◽  
Left Ventricular ◽  
Cardiac Contraction ◽  
Arterial Oxygen ◽  
Adenosine Release ◽  
Lactate Release

Critical intracellular myocardial oxygen tension was determined by optical spectroscopic measurement of myoglobin oxygen saturation in crystalloid-perfused guinea pig hearts. Accurate end-point determinations of the maximally oxygenated and deoxygenated myoglobin were made. Hearts were subjected to a steady decrease in perfusate oxygen tension while left ventricular developed pressure, maximal left ventricular dP/d t, myocardial oxygen consumption, lactate release, and adenosine release were measured as indices of myocardial function. Intracellular myoglobin was found to be only 72% saturated under baseline conditions with an arterial oxygen tension of >600 mmHg at 37°C. Baseline intracellular oxygen tension was 6.3 mmHg. Myocardial oxygen consumption was decreased by 10% when the oxygen tension fell to 5.7 mmHg, and cardiac contraction decreased 10% when oxygen tension was 4.1 mmHg. Adenosine release and, finally, lactate release began to increase at sequentially lower oxygen tensions. The present results indicate that the buffer-perfused guinea pig heart at 37°C has an intracellular oxygen tension just above the threshold for impaired function.

Comparison of buffer and red blood cell perfusion of guinea pig heart oxygenation

2003 ◽  
Vol 285 (5) ◽  
pp. H1819-H1825 ◽  
Author(s):  
Kenneth A. Schenkman ◽  
Daniel A. Beard ◽  
Wayne A. Ciesielski ◽  
Eric O. Feigl
Keyword(s):  
Oxygen Consumption ◽  
Guinea Pig ◽  
Oxygen Saturation ◽  
Red Blood Cells ◽  
Ventricular Function ◽  
Oxygen Tension ◽  
Blood Cells ◽  
Low Oxygen ◽  
Guinea Pig Heart ◽  
Perfused Hearts

Myocardial mean myoglobin oxygen saturation was determined spectroscopically from isolated guinea pig hearts perfused with red blood cells during increasing hypoxia. These experiments were undertaken to compare intracellular myoglobin oxygen saturation in isolated hearts perfused with a modest concentration of red blood cells (5% hematocrit) with intracellular myoglobin saturation previously reported from traditional buffer-perfused hearts. Studies were performed at 37°C with hearts paced at 240 beats/min and a constant perfusion pressure of 80 cmH2O. It was found that during perfusion with a hematocrit of 5%, baseline mean myoglobin saturation was 93% compared with 72% during buffer perfusion. Mean myoglobin saturation, ventricular function, and oxygen consumption remained fairly constant for arterial perfusate oxygen tensions above 100 mmHg and then decreased precipitously below 100 mmHg. In contrast, mean myoglobin saturation, ventricular function, and oxygen consumption began to decrease even at high oxygen tension with buffer perfusion. The present results demonstrate that perfusion with 5% red blood cells in the perfusate increases the baseline mean myoglobin saturation and better preserves cardiac function at low oxygen tension relative to buffer perfusion. These results suggest that caution should be used in extrapolating intracellular oxygen dynamics from buffer-perfused to blood-perfused hearts.

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