EFFECTS OF CHRONIC BACK PAIN ON THE PERCEPTION OF EXPERIMENTAL HEAT PAIN

1990 ◽  
Vol 71 (7) ◽  
pp. 1283 ◽  
Author(s):  
S. LAUTENBACHE
1990 ◽  
Vol 71 (3_suppl) ◽  
pp. 1283-1292 ◽  
Author(s):  
S. Lautenbacher ◽  
G. Galfe ◽  
G. Karlbauer ◽  
A. Möltner ◽  
F. Strian

2021 ◽  
Vol 2 ◽  
Author(s):  
Benjamin Provencher ◽  
Stéphane Northon ◽  
Mathieu Piché

Musculoskeletal injuries lead to sensitization of nociceptors and primary hyperalgesia (hypersensitivity to painful stimuli). This occurs with back injuries, which are associated with acute pain and increased pain sensitivity at the site of injury. In some cases, back pain persists and leads to central sensitization and chronic pain. Thus, reducing primary hyperalgesia to prevent central sensitization may limit the transition from acute to chronic back pain. It has been shown that spinal manipulation (SM) reduces experimental and clinical pain, but the effect of SM on primary hyperalgesia and hypersensitivity to painful stimuli remains unclear. The goal of the present study was to investigate the effect of SM on pain hypersensitivity using a capsaicin-heat pain model. Laser stimulation was used to evoke heat pain and the associated brain activity, which were measured to assess their modulation by SM. Eighty healthy participants were recruited and randomly assigned to one of the four experimental groups: inert cream and no intervention; capsaicin cream and no intervention; capsaicin cream and SM at T7; capsaicin cream and placebo. Inert or capsaicin cream (1%) was applied to the T9 area. SM or placebo were performed 25 min after cream application. A series of laser stimuli were delivered on the area of cream application (1) before cream application, (2) after cream application but before SM or placebo, and (3) after SM or placebo. Capsaicin cream induced a significant increase in laser pain (p < 0.001) and laser-evoked potential amplitude (p < 0.001). However, SM did not decrease the amplification of laser pain or laser-evoked potentials by capsaicin. These results indicate that segmental SM does not reduce pain hypersensitivity and the associated pain-related brain activity in a capsaicin-heat pain model.


2019 ◽  
Vol 24 (5) ◽  
pp. 14-15
Author(s):  
Jay Blaisdell ◽  
James B. Talmage

Abstract Ratings for “non-specific chronic, or chronic reoccurring, back pain” are based on the diagnosis-based impairment method whereby an impairment class, usually representing a range of impairment values within a cell of a grid, is selected by diagnosis and “specific criteria” (key factors). Within the impairment class, the default impairment value then can be modified using non-key factors or “grade modifiers” such as functional history, physical examination, and clinical studies using the net adjustment formula. The diagnosis of “nonspecific chronic, or chronic reoccurring, back pain” can be rated in class 0 and 1; the former has a default value of 0%, and the latter has a default value of 2% before any modifications. The key concept here is that the physician believes that the patient is experiencing pain, yet there are no related objective findings, most notably radiculopathy as distinguished from “nonverifiable radicular complaints.” If the individual is found not to have radiculopathy and the medical record shows that the patient has never had clinically verifiable radiculopathy, then the diagnosis of “intervertebral disk herniation and/or AOMSI [alteration of motion segment integrity] cannot be used.” If the patient is asymptomatic at maximum medical improvement, then impairment Class 0 should be chosen, not Class 1; a final whole person impairment rating of 1% indicates incorrect use of the methodology.


2007 ◽  
Vol 40 (21) ◽  
pp. 14
Author(s):  
Nancy Walsh
Keyword(s):  

1999 ◽  
Vol 11 (3) ◽  
pp. 381-385 ◽  
Author(s):  
Alexander A. Vendrig ◽  
Jan J. L. Derksen ◽  
Hubert R. de Mey

2014 ◽  
Author(s):  
Pilar Peris ◽  
Jordi Blasco ◽  
Josep L Carrasco ◽  
Angels Martinez-Ferrer ◽  
Juan Macho ◽  
...  

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