The Role of Enteroceptive Awareness in Eating Disorders: a Study On a Group of Binge Eaters

Author(s):  
Gian Marco Longo
2017 ◽  
Vol 41 (S1) ◽  
pp. S556-S556
Author(s):  
G.M. Longo ◽  
J. Falcone ◽  
R.M. Martoni ◽  
L. Bellodi ◽  
A. Ogliari ◽  
...  

IntroductionEnteroceptive awareness is defined as the ability to perceive the body states. Such ability is provided by the enteroceptors, organs and structures specifically designated to receive both internal and external stimuli. This capability, especially the perception of satiation, seems to be lacking in individuals suffering from Eating Disorders and there is no procedure to increase this ability.ObjectivesThe primary purpose is to improve patient's enteroceptive awareness. We tested whether patients diagnosed with binge eating disorder (BED), while deprived of eyesight, would better focus on their internal signals, such as the satiation feeling, and therefore eat less and slower.MethodsFor the first time, 29 patients with BED were deprived of vision during a meal. In a standardized procedure, participants ate two different meals, the first one while deprived of eyesight, the second one, a week later, in normal conditions. Both the amount of eaten food and the total time to complete the meal were taken into account during each of the meals. The patients filled in a Visual Analogue Scale (VAS) questionnaire at the end of each meal, in order to evaluate the experience and their internal feelings.ResultsThe results show that patients suffering from BED, when deprived of eyesight, eat less food, take more time to finish the meal and experience the same level of satiation with respect to the normal condition.ConclusionThis procedure could be of great interest for the implementation of specific intervention protocols that are aimed at the recovery of enteroceptive awareness in patients with eating disorders.Disclosure of interestThe authors have not supplied their declaration of competing interest.


CNS Spectrums ◽  
2004 ◽  
Vol 9 (7) ◽  
pp. 523-529 ◽  
Author(s):  
Palmiero Monteleone ◽  
Antonio DiLieto ◽  
Eloisa Castaldo ◽  
Mario Maj

AbstractLeptin is an adipocyte-derived hormone, which is involved predominantly in the long-term regulation of body weight and energy balance by acting as a hunger suppressant signal to the brain. Leptin is also involved in the modulation of reproduction, immune function, physical activity, and some endogenous endocrine axes. Since anorexia nervosa (AN) and bulimia nervosa (BN) are characterized by abnormal eating behaviors, dysregulation of endogenous endocrine axes, alterations of reproductive and immune functions, and increased physical activity, extensive research has been carried out in the last decade in order to ascertain a role of this hormone in the pathophysiology of these syndromes. In this article, we review the available data on leptin physiology in patients with eating disorders. These data support the idea that leptin is not directly involved in the etiology of AN or BN. However, malnutrition-induced alterations in its physiology may contribute to the genesis and/or the maintenance of some clinical manifestations of AN and BN and may have an impact on the prognosis of AN.


2015 ◽  
Vol 23 (5) ◽  
pp. 413-416 ◽  
Author(s):  
Lies Depestele ◽  
Laurence Claes ◽  
Eva Dierckx ◽  
Imke Baetens ◽  
Katrien Schoevaerts ◽  
...  

1987 ◽  
Vol 16 (4) ◽  
pp. 305-309 ◽  
Author(s):  
Jeffrey M. Jonas ◽  
Mark S. Gold

Ten individuals with antidepressant-resistant bulimia were treated with the long-acting opiate antagonist naltrexone. Seven of the ten experienced at least a 75 percent reduction of their bulimic symptoms, and have maintained their improvment on three to five month follow-up. These preliminary data suggest that naltrexone may be of use in bulimia unresponsive to standard antidepressant therapy, and may provide insight into the role of endogenous opioids in the etiology of eating disorders.


2011 ◽  
Vol 16 (1) ◽  
pp. e72-e72 ◽  
Author(s):  
S. Gaetani ◽  
W. H. Kaye ◽  
V. Cuomo ◽  
Daniele Piomelli
Keyword(s):  

1987 ◽  
Vol 62 (8) ◽  
pp. 754-755
Author(s):  
Alexander R. Lucas

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