scholarly journals Rethrombolysis for Restruck Prosthetic Valve

2004 ◽  
Vol 3 (3) ◽  
pp. 56
Author(s):  
J Ezhilan

One of the complications of prosthetic valve (PV) is thrombotic occlusion of the valve. The incidence is 5/100/yr overall and is more common in mitral position than in aortic position, PV thrombosis presentation can be acute due to thrombi (55%), Subacute due to thrombus and pannus (40%) and Chronic due to pannus alone (5%). It can manifest as congestive cardiac failure, Pulmonary embolism, cardiogenic shock, Thromboembolism or death. Conventionally it was treated with surgical thrombectomy and pannus excision or valve replacement. Later on thrombolysis was tried and various agents like Streplokinase, Urokinase and t- PA have been used successfully to lyse the thrombus.


Circulation ◽  
2020 ◽  
Vol 142 (Suppl_3) ◽  
Author(s):  
Sudhi Tyagi ◽  
Harshal Patil ◽  
Robert Miles ◽  
Elizabeth Siegel ◽  
Salman Allana ◽  
...  

Introduction: Determining the etiology of dyspnea in patients with structural heart disease can be challenging, especially in the current era with high prevalence of COVID-19. Our case highlights the importance of evaluating bioprosthetic valve function in the setting of a change in clinical status. Case: An 82 year old female with surgical coronary revascularization and bioprosthetic aortic valve replacement in 2003 presented with dyspnea. Her evaluation revealed hypoxemia, leukocytosis and a chest x-ray supportive of viral infection when the local prevalence of both COVID-19 and influenza were at their peak. She was admitted to the intensive care unit with impending respiratory failure most likely from an infectious etiology. Echocardiogram revealed an ejection fraction (EF) of 25% and severe prosthetic aortic regurgitation with a pressure half time of 117ms ( Figure ). Six months prior, she had normal EF and normal prosthetic valve function. Interestingly, she lacked a wide pulse pressure, murmur of aortic insufficiency, and other characteristic exam findings of valvular dysfunction. The patient rapidly deteriorated into cardiogenic shock. Following urgent evaluation for transcatheter aortic valve replacement, she had successful valve-in-valve deployment of a 23mm Edwards S3 Ultra valve. Her hemodynamic parameters improved immediately and she was weaned from inotropic support 1 day following valve replacement. Conclusions: Evaluation of prosthetic valve function is integral when a patient’s clinical condition changes. In our patient, depressed EF resulting in an elevated left ventricular end-diastolic pressure likely diminished the regurgitant fraction and the expected aortic insufficiency murmur. Periodic evaluation of prosthetic heart valve function is necessary, particularly when the patient’s clinical condition changes. Acute severe aortic regurgitation with cardiogenic shock is fatal without rapid evaluation and valve replacement.



2009 ◽  
Vol 18 (2) ◽  
pp. 114-118 ◽  
Author(s):  
Ashish Katewa ◽  
Pradeep Vaideeswar ◽  
Jayant V. Khandekar ◽  
Sayed Sajid ◽  
Rahul M. Jawale ◽  
...  


2004 ◽  
Vol 52 (S 1) ◽  
Author(s):  
D Fritzsche ◽  
O Grimmig ◽  
T Eitz ◽  
A Brensing ◽  
K Minami ◽  
...  


2021 ◽  
Vol 65 ◽  
pp. 102314
Author(s):  
Takasumi Goto ◽  
Hiroyuki Nishi ◽  
Mutsunori Kitahara ◽  
Yoshinori Yokono ◽  
Satoshi Sakakibara ◽  
...  


1981 ◽  
Vol 2 (6) ◽  
pp. 290-291 ◽  
Author(s):  
J. B. Myers ◽  
T. O. Morgan ◽  
J. N. Walker


2009 ◽  
Vol 19 (5) ◽  
pp. 530-533 ◽  
Author(s):  
Sigrun R. Hofmann ◽  
Matthias Weise ◽  
Katharina I. Nitzsche

AbstractCongenital arteriovenous malformations are rare causes of congestive cardiac failure in neonates. The most common sites are in the head and liver, but other sites include the thorax, the abdomen and the limbs. The onset of failure is usually not in the immediate neonatal period, but later on in life, albeit that lesions such as the arteriovenous malformation of the vein of Galen, and other arteriovenous malformations in different locations which produce high flow can present early. We describe here the first case, to the best of our knowledge, of prenatal detection of an intrathoracic arteriovenous malformation producing neonatal cardiac failure, which was successfully treated by surgery postnatally.



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