scholarly journals Ossifying Fibroma pada mandibula pasien anak

2020 ◽  
Vol 4 (2) ◽  
pp. 21
Author(s):  
Ratih Trikusumadewi Lubis ◽  
Fadhlil Ulum Abdul Rahman ◽  
Muhammad Adri Nurrahim ◽  
Lusi Epsilawati ◽  
Eka Marwansyah Oli'i

Objectives: To view a case report of mandibular ossifying fibroma (OF) in pediatric male. Case Report: A 12 year old child came to RSHS with a panoramic radiograph with the chief complaint of swelling in the right mandible. Panoramic radiograph shows well-defined mixed lesions with radiolucent content and snowflake-like opaque. MDCT shows a superior-inferior and bucco-lingual extension of the lesion. The suspicion of this case leads to Ossifying Fibroma with differential diagnosis of Adenomatoid Odontogenic Tumor (AOT) and Calcifiying Ephitelial Odontogenic Tumor (CEOT). Conclusion: OF cases in children, especially mandibular, are very rare, where the characteristics of the lesion can be well defined through the help of panoramic radiographs and MDCT. OF is a lesion that has benign characteristics with well-defined borders, and the most important lies in the presence of wrapping capsules and mixed internal structures accompanied by snowflake-like calcification.

Author(s):  
ANNE FERNANDES DOS SANTOS ◽  
FRANCISCO DE ASSIS ALMEIDA LIMA-JÚNIOR ◽  
JOHN LENNON SILVA CUNHA ◽  
MARCELO FERNANDES SANTOS DE MELO ◽  
JULIANA BATISTA MELO DA FONTE ◽  
...  

Author(s):  
FERNANDA ARAGÃO FELIX ◽  
RODRIGO PORPINO MAFRA ◽  
LARISSA SANTOS AMARAL ROLIM ◽  
HELLEN BANDEIRA DE PONTES SANTOS ◽  
PATRÍCIA DAVIN GOMES PARENTE ◽  
...  

2021 ◽  
Vol 156 (Supplement_1) ◽  
pp. S88-S89
Author(s):  
A Lazim ◽  
R Kuklani ◽  
D Sundararajan

Abstract Introduction/Objective Adenomatoid odontogenic tumor (AOT) is an uncommon benign odontogenic tumor representing 3 to 7% of all odontogenic tumors. This tumor was first reported as adeno-ameloblastoma by Bernier and Tiecke in 1950 as it was initially assumed to be a type of ameloblastoma. In 1969, Philipsen and Bern proposed the term adenomatoid odontogenic tumor which was subsequently adopted by WHO and became the accepted terminology for this tumor. AOT is classified as a tumor of odontogenic epithelium but occasionally abnormal hard tissues consistent with dentinoid material may also be present as part of the tumor. AOT tends to occur in younger patients and 50% of the cases are diagnosed in teenagers. It occurs twice as commonly in females and frequently involves the anterior maxilla. Radiographically, AOT can appear as a radiolucent or mixed lucent-opaque lesion and may be associated with an impacted tooth. Methods/Case Report We report two unusually large expansile lesions of AOT that presented in the mandible. In the first case, the tumor presented as an expansile radiolucent lesion involving the right posterior mandible in a 32 year old female. In the second case, the tumor presented as an expansile mixed lucent-opaque lesion involving the left anterior mandible in a 21 year old female. The clinical presentation, radiographic and imaging findings, histopathologic features and treatment of these two cases will be discussed. The recommended treatment for AOT is surgical excision. The prognosis is good as this tumor seldom recurs after excision. Results (if a Case Study enter NA) NA Conclusion AOT is considered to be a non-aggressive, non-invasive and slow growing benign neoplasm. It is usually discovered on routine radiographic examination as the lesion is usually small and asymptomatic at the time of diagnosis but occasional cases that are larger in size have been reported in the literature.


2016 ◽  
Vol 2016 ◽  
pp. 1-5 ◽  
Author(s):  
Fatemeh Rezaei ◽  
Hesamedin Nazari ◽  
Babak Izadi

A 46-year-old male patient referred to Department of Oral Medicine, with the primary chief complaint of a painless swelling in the right side of mandibular. A panoramic radiograph revealed a well-defined, multilocular radiolucent bony lesion with thin and straight septa in the right side of mandible extending from distal of canine to mesial of third molar. Histological examination showed a solid proliferation of atypical plasmacytoid cells, which was indicative of plasmacytoma. A systemic workup for the final diagnosis was performed to rule out multiple myeloma.


Author(s):  
Analú Barros De Oliveira ◽  
Túlio Morandin Ferrisse ◽  
Lauro Toyoshi Mizuno ◽  
Delise Pellizzaro ◽  
Marcelo Gonçalves ◽  
...  

2018 ◽  
Vol 2018 ◽  
pp. 1-4
Author(s):  
Zhen Kang ◽  
Xiangde Min ◽  
Liang Wang

Background. Abernethy malformation is a rare splanchnic vascular abnormality characterizing extrahepatic abnormal shunts that is classified into types I and II. Abernethy malformation type I has a female predilection and is associated with a variety of concurrent hepatic benign or malignant tumours while type II with concurrent tumours is very rare in females. Case Report. We report a rare female case of Abernethy malformation type II with concurrent occupying lesion in the right liver, which was successfully transplanted; the occupying lesion was pathologically proven to be nodular hyperplasia. Conclusion. This case might provide further knowledge regarding Abernethy malformation. On imaging, the anatomy of portal vein should be carefully investigated to categorize Abernethy malformation, and a wide variety of differential diagnosis of concurrent occupying lesions should be taken into account.


Author(s):  
NA Belykh ◽  
MA Akhkyamova ◽  
VV Gusev ◽  
OA Lvova

Dopa-responsive dystonia (DRD) is a rare progressive genetically heterogenous disorder with pediatric onset. DRD is 3 times as prevalent in women than in men. This article reports a clinical case of DRD in a young female presenting with paraparesis, foot dystonia (more pronounced in the right foot) and pronounced walking impairment, who was admitted for emergency treatment to a Neurology Unit. Based on the additional tests, which included a levodopa trial and Sanger sequencing, the patient was diagnosed with DRD. Levodopa caused a considerable improvement of the symptoms. The article describes the clinical features of the disease, talks about its differential diagnosis, genetic predisposition and treatment strategy.


Sign in / Sign up

Export Citation Format

Share Document