scholarly journals A Study of Cecal Ligation and Puncture-Induced Sepsis in Tissue-Specific Tumor Necrosis Factor Receptor 1-Deficient Mice

2019 ◽  
Vol 10 ◽  
Author(s):  
Jolien Vandewalle ◽  
Sophie Steeland ◽  
Sara Van Ryckeghem ◽  
Melanie Eggermont ◽  
Elien Van Wonterghem ◽  
...  
Keyword(s):  
Tumor Necrosis Factor ◽  
Tumor Necrosis ◽  
Cecal Ligation ◽  
Tumor Necrosis Factor Receptor ◽  
Cecal Ligation And Puncture ◽  
Tissue Specific ◽  
Specific Tumor ◽  
Deficient Mice ◽  
Necrosis Factor

scholarly journals Su1033 – Contrasting Effects of Global Versus Tissue-Specific Deletion of Tumor Necrosis Factor Receptor 1(TNFr1) in Murine Colitis

2019 ◽  
Vol 156 (6) ◽  
pp. S-490
Author(s):  
Ying Huang ◽  
Cambrian Y. Liu ◽  
Sharon S. Tam ◽  
Nandini Girish ◽  
Shivesh Punit ◽  
...  
Keyword(s):  
Tumor Necrosis Factor ◽  
Tumor Necrosis ◽  
Tumor Necrosis Factor Receptor ◽  
Tissue Specific ◽  
Murine Colitis ◽  
Necrosis Factor ◽  
Specific Deletion

Induction of CD14 expression inLpsn,Lpsd and tumor necrosis factor receptor-deficient mice

1996 ◽  
Vol 26 (11) ◽  
pp. 2686-2692 ◽  
Author(s):  
Tetsuya Takakuwa ◽  
Hans-Peter Knopf ◽  
Andreas Sing ◽  
Rita Carsetti ◽  
Chris Galanos ◽  
...  
Keyword(s):  
Tumor Necrosis Factor ◽  
Tumor Necrosis ◽  
Tumor Necrosis Factor Receptor ◽  
Deficient Mice ◽  
Necrosis Factor

scholarly journals A Role for Tumor Necrosis Factor Receptor Type 1 in Gut-associated Lymphoid Tissue Development: Genetic Evidence of Synergism with Lymphotoxin β

1998 ◽  
Vol 187 (12) ◽  
pp. 1977-1983 ◽  
Author(s):  
Pandelakis A. Koni ◽  
Richard A. Flavell
Keyword(s):  
Tumor Necrosis Factor ◽  
Lymphoid Tissue ◽  
Tumor Necrosis ◽  
Tumor Necrosis Factor Receptor ◽  
Tumor Necrosis Factor Receptors ◽  
Β Receptor ◽  
Lymphotoxin Α ◽  
Deficient Mice ◽  
Necrosis Factor

Lymphotoxin α (LTα) signals via tumor necrosis factor receptors (TNFRs) as a homotrimer and via lymphotoxin β receptor (LTβR) as a heterotrimeric LTα1β2 complex. LTα-deficient mice lack all lymph nodes (LNs) and Peyer's patches (PPs), and yet LTβ-deficient mice and TNFR-deficient mice have cervical and mesenteric LN. We now show that mice made deficient in both LTβ and TNFR type 1 (TNFR1) lack all LNs, revealing redundancy or synergism between TNFR1 and LTβ, acting presumably via LTβR. A complete lack of only PPs in mice heterozygous for both ltα and ltβ, but not ltα or ltβ alone, suggests a similar two-ligand phenomenon in PP development and may explain the incomplete lack of PPs seen in tnfr1−/− mice.

Accelerated wound healing in tumor necrosis factor receptor p55‐deficient mice with reduced leukocyte infiltration

2002 ◽  
Vol 16 (9) ◽  
pp. 963-974 ◽  
Author(s):  
RYOICHI MORI ◽  
TOSHIKAZU KONDO ◽  
TOHRU OHSHIMA ◽  
YUKO ISHIDA ◽  
NAOFUMI MUKAIDA
Keyword(s):  
Wound Healing ◽  
Tumor Necrosis Factor ◽  
Tumor Necrosis ◽  
Tumor Necrosis Factor Receptor ◽  
Leukocyte Infiltration ◽  
Deficient Mice ◽  
Necrosis Factor
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