scholarly journals Corrigendum: Secretion of Salmonella Pathogenicity Island 1-Encoded Type III Secretion System Effectors by Outer Membrane Vesicles in Salmonella Enterica Serovar Typhimurium

2019 ◽  
Vol 10 ◽  
Author(s):  
Seul I Kim ◽  
Seongok Kim ◽  
Eunsuk Kim ◽  
Seo Yeon Hwang ◽  
Hyunjin Yoon
Keyword(s):  
Outer Membrane ◽  
Type Iii Secretion ◽  
Salmonella Enterica ◽  
Salmonella Enterica Serovar Typhimurium ◽  
Type Iii Secretion System ◽  
Membrane Vesicles ◽  
Secretion System ◽  
Outer Membrane Vesicles ◽  
Salmonella Pathogenicity Island ◽  
Serovar Typhimurium

scholarly journals SigE Is a Chaperone for the Salmonella enterica Serovar Typhimurium Invasion Protein SigD

2001 ◽  
Vol 183 (4) ◽  
pp. 1452-1454 ◽  
Author(s):  
K. Heran Darwin ◽  
Lloyd S. Robinson ◽  
Virginia L. Miller
Keyword(s):  
Hybrid System ◽  
Type Iii Secretion ◽  
Salmonella Enterica ◽  
Salmonella Enterica Serovar Typhimurium ◽  
Type Iii Secretion System ◽  
Secretion System ◽  
Type Iii ◽  
Serovar Typhimurium ◽  
Two Hybrid

ABSTRACT SigD is translocated into eucaryotic cells by a type III secretion system. In this work, evidence that the putative chaperone SigE directly interacts with SigD is presented. A bacterial two-hybrid system demonstrated that SigE can interact with itself and SigD. In addition, SigD was specifically copurified with SigE-His6on a nickel column.

scholarly journals Secretion of the orgC Gene Product by Salmonella enterica Serovar Typhimurium

2003 ◽  
Vol 71 (11) ◽  
pp. 6680-6685 ◽  
Author(s):  
James B. Day ◽  
Catherine A. Lee
Keyword(s):  
Gene Product ◽  
Type Iii Secretion ◽  
Salmonella Enterica ◽  
Virulence Genes ◽  
Salmonella Enterica Serovar Typhimurium ◽  
Type Iii Secretion System ◽  
Pathogenicity Island ◽  
Secretion System ◽  
Serovar Typhimurium ◽  
Transposon Insertions

ABSTRACT HilA activates the transcription of genes on Salmonella pathogenicity island 1 (SPI1), which encodes a type III secretion system (TTSS). Previous studies showed that transposon insertions in orgC, a gene located on SPI1, increase hilA expression. We characterize the orgC gene product and show that it is secreted via the SPI1 TTSS. We propose a model whereby OrgC functions as a secreted repressor of the SPI1 virulence genes.

Sign in / Sign up

Export Citation Format

Share Document