scholarly journals Acute Effects of Air Pollution and Noise from Road Traffic in a Panel of Young Healthy Subjects

Proceedings ◽  
2018 ◽  
Vol 6 (1) ◽  
pp. 10 ◽  
Author(s):  
Hanns Moshammer ◽  
Julian Panholzer ◽  
Lisa Ulbing ◽  
Emanuel Udvarhelyi ◽  
Barbara Ebenbauer ◽  
...  

Twenty-four healthy students walked at least four times for 1 hour under each of the four settings: by a busy road; by a busy road wearing ear plugs; in a park; and in a park but exposed to traffic noise (65 dB) through speakers. Particle mass (smaller than 2.5 and 1 µm, PM1 and PM2.5, (respectively)particle number and noise levels were measured throughout each walk. Lung function and exhaled nitric oxide (NO) were measured before, immediately after, 1 hour after, and approximately 24 h after each walk. Blood pressure and heart-rate variability were measured every 15 min during each walk. Air pollution levels reduced lung function levels; noise levels reduced systolic blood pressure and heart-rate variability.

Author(s):  
Hanns Moshammer ◽  
Julian Panholzer ◽  
Lisa Ulbing ◽  
Emanuel Udvarhelyi ◽  
Barbara Ebenbauer ◽  
...  

Panel studies are an efficient means to assess short-term effects of air pollution and other time-varying environmental exposures. Repeated examinations of volunteers allow for an in-depth analysis of physiological responses supporting the biological interpretation of environmental impacts. Twenty-four healthy students walked for 1 h at a minimum of four separate occasions under each of the following four settings: along a busy road, along a busy road wearing ear plugs, in a park, and in a park but exposed to traffic noise (65 dB) through headphones. Particle mass (PM2.5, PM1), particle number, and noise levels were measured throughout each walk. Lung function and exhaled nitrogen oxide (NO) were measured before, immediately after, 1 h after, and approximately 24 h after each walk. Blood pressure and heart rate variability were measured every 15 min during each walk. Recorded air pollution levels were found to correlate with reduced lung function. The effects were clearly significant for end-expiratory flows and remained visible up to 24 h after exposure. While immediate increases in airway resistance could be interpreted as protective (muscular) responses to particulate air pollution, the persisting effects indicate an induced inflammatory reaction. Noise levels reduced systolic blood pressure and heart rate variability. Maybe due to the small sample size, no effects were visible per specific setting (road vs. park).


2011 ◽  
Vol 111 (1) ◽  
pp. 177-183 ◽  
Author(s):  
Maria Foraster ◽  
Alexandre Deltell ◽  
Xavier Basagaña ◽  
Mercedes Medina-Ramón ◽  
Inmaculada Aguilera ◽  
...  

2016 ◽  
Vol 151 ◽  
pp. 804-813 ◽  
Author(s):  
Wilma Zijlema ◽  
Yutong Cai ◽  
Dany Doiron ◽  
Stéphane Mbatchou ◽  
Isabel Fortier ◽  
...  

2016 ◽  
Vol 2016 (1) ◽  
Author(s):  
Wilma Zijlema* ◽  
Yutong Cai ◽  
Dany Doiron ◽  
Stephane Mbatchou ◽  
Isabel Fortier ◽  
...  

2004 ◽  
Vol 26 (2) ◽  
pp. 193-200 ◽  
Author(s):  
Ubiratan de Paula Santos ◽  
Alfésio Luís Ferreira Braga ◽  
Dante Marcelo Artigas Giorgi ◽  
Luiz Alberto Amador Pereira ◽  
César Jose Grupi ◽  
...  

2008 ◽  
Vol 34 (2) ◽  
pp. 226-231 ◽  
Author(s):  
Goran Belojevic ◽  
Branko Jakovljevic ◽  
Vesna Stojanov ◽  
Katarina Paunovic ◽  
Jelena Ilic

Author(s):  
Kateryna B. Fuks ◽  
Claudia Wigmann ◽  
Hicran Altug ◽  
Tamara Schikowski

The detrimental effects of traffic noise on cognition in children are well documented. Not much is known about the health effects in adults. We investigated the association of residential exposure to road traffic noise and annoyance due to road traffic noise with cognitive function in a cohort of 288 elderly women from the longitudinal Study on the influence of Air pollution on Lung function, Inflammation and Aging (SALIA) in Germany. Residential noise levels—weighted 24-h mean (LDEN) and nighttime noise (LNIGHT)—were modeled for the most exposed facade of dwellings and dichotomized at ≥50 dB(A). Traffic noise annoyance (day and night) was estimated by questionnaire. Cognitive function was assessed using the Consortium to Establish a Registry on Alzheimer’s Disease (CERAD-Plus) Neuropsychological Assessment Battery. The modeled noise levels were associated with impaired total cognition and the constructional praxis domain, independently of air pollution. Self-reported noise annoyance was associated with better performance in semantic memory and constructional praxis domains. This finding should be interpreted with caution since we could not control for potential confounding by hearing loss. Noise levels and annoyance were associated, but their health effects seemed mutually independent.


2020 ◽  
Vol 10 (1) ◽  
Author(s):  
Dong-Hoon Lee ◽  
Sun-Hwa Kim ◽  
Si-Hyuck Kang ◽  
Oh Kyung Kwon ◽  
Jin-Joo Park ◽  
...  

Abstract Air pollution has increasingly been recognized as a major healthcare concern. Air pollution, particularly fine particulate matter (≤ 2.5 μm in aerodynamic diameter [PM2.5]) has demonstrated an increase in adverse cardiovascular events. This study aimed to assess the cardiovascular response to personal exposure to different levels of PM2.5. This prospective cohort study enrolled healthy volunteers aged ≥ 18 years with no cardiovascular disease. Study subjects carried personal exposure monitor of PM2.5, digital thermo-hygrometer for temperature and humidity, 24-h blood pressure monitor, and continuous electrocardiogram monitor. Measurements were repeated twice with an interval of 6–12 months. Statistical models consisted of generalized estimation equations to various repeated measures of each subject. A total of 22 subjects were enrolled in this study between July 2018 and January 2019. Measurement was performed twice in all participants, and a total of 36 data were collected except for insufficient data collection. The mean age of the study population was 41.6 years, and 95% of the subjects were females. No study subjects had hypertension or other cardiovascular diseases. The average systolic blood pressure increased with higher PM2.5 levels with marginal significance (0.22 mmHg [95% confidential intervals − 0.04 to 0.48 mmHg] per 10 μg/m3 of PM2.5). All parameters for heart rate variability significantly decreased with a higher level of PM2.5. In this study, we measured individual personal exposure to PM2.5 by using a portable device. We found that 24-h exposure to high levels of PM2.5 was associated with a significant decrease in heart rate variability, suggesting impaired autonomous nervous function.


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