scholarly journals The calcium effect on the thermopower, critical temperature and charge-carrier system parameters in the Y0.75-xCaxPr0.25Ba2Cu3Oy HTSC-system

2020 ◽  
Vol 1 (4) ◽  
pp. 142-149
Author(s):  
Vitaliy E. Gasumyants
2018 ◽  
Vol 173 ◽  
pp. 03015
Author(s):  
Kirill Kulikov ◽  
Yury Shukrinov ◽  
Majed Nashaat ◽  
Akinobu Irie

The influence of the charge imbalance effect on a system of intrinsic Josephson junctions of high temperature superconductors under external electromagnetic radiation is investigated. We find that the charge imbalance is responsible for the slope of the Shapiro step in the IV-characteristic. The nonperiodic boundary conditions shift the Shapiro step from the canonical position which is determined by the frequency of the external radiation. We also find how the system parameters influence the shift of the Shapiro step.


1999 ◽  
Vol 13 (09n10) ◽  
pp. 1151-1156 ◽  
Author(s):  
A. Lascialfari ◽  
P. Ghigna ◽  
S. De Gennaro

The charge carrier density plays a crucial role in the transition between normal and superconducting state in the copper-oxide superconductors. In Nd 1.85 Ce 0.15 CuO 4+δ sample, for charge carrier density n = nc≃ 0.07 the superconductivity sets in. In order to investigate the magnetic properties around nc, a set of magnetic susceptibility measurements in the temperature range 3-260K for samples with different charge carriers concentrations were realized. Magnetic susceptibility curves of Nd 1.85 Ce 0.15 CuO 4+δ revealed novel behavior well above the critical temperature T c: at fixed T ≥45 K , the susceptibility χ, reported as a function of the charge carrier density n, shows a peak around n=0.06 and drops to significantly lower values for n greater than 0.07, the value at which the samples become superconducting. The effective magnetic moment μeff shows the same behavior. The comparison of the present experimental results with recent transport ones gives evidence of a possible interaction between charge carriers above T c. The possibility that this effect could be due to a bipolarons-like mechanism of charge carriers interaction is discussed.


1979 ◽  
Vol 46 ◽  
pp. 65-74 ◽  
Author(s):  
A.C. Fabian ◽  
J.E. Pringle ◽  
J.A.J. Whelan ◽  
J.A. Bailey

Abstract.Recent photometric and spectroscopic observations of the dwarf nova system Z Cha are discussed. Methods for constraining the system parameters are applied and the disc emissivity is deduced as a function of radius. Indications are found that the disc shrinks in size with increasing time after outburst.


2001 ◽  
Vol 120 (5) ◽  
pp. A142-A142
Author(s):  
J GASKEY ◽  
E SEIDEL

1977 ◽  
Vol 37 (02) ◽  
pp. 243-252
Author(s):  
Yi-Hsiang Chen ◽  
E. B Reeve

SummaryTo shed some light on the homeostatic regulation of plasma fibrinogen, metabolic studies were made in healthy females, and in normal, thyroidectomized, and thyroxine-treated rabbits. In females, compared with normal males, plasma fibrinogen concentration, plasma and interstitial fibrinogen decreased consequent to an increased fractional catabolic rate and a normal fibrinogen synthesis rate. The interstitial/plasma fibrinogen ratio remained unchanged. In normal rabbits, with increasing body weight fractional catabolic rate and catabolic rate decreased, while fibrinogen concentration and plasma fibrinogen remained constant owing to a simultaneous decrease in fibrinogen synthesis. In addition, fractional transcapillary transfer rate and transcapillary flux also decreased resulting in a shrinkage of interstitial fibrinogen. Thyroidectomy and thyroxine-injection markedly altered fibrinogen metabolism: thyroid hormone accelerated fibrinogen catabolism but also stimulated synthesis. The net result was an increase in plasma fibrinogen and fibrinogen concentration. The interstitial/plasma fibrinogen ratio decreased in thyroxine-treated, and increased in thyroidectomized animals. This study defines the variations of the fibrinogen system parameters in these physiologic and pathologic conditions, and illustrates some patterns of alterations in fibrinogen metabolism.


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