scholarly journals Tricuspid regurgitation after endomyocardial biopsy

2021 ◽  
Vol 54 (6) ◽  
pp. 238
Author(s):  
Chia-Ju Tsai ◽  
I-Chen Chen ◽  
Chung-Yi Chang
Keyword(s):  
Tricuspid Regurgitation ◽  
Endomyocardial Biopsy

Repair of Early Posttransplant Endomyocardial Biopsy-Related Tricuspid Regurgitation in a Child

2015 ◽  
Vol 6 (2) ◽  
pp. 295-297 ◽  
Author(s):  
F. Bennett Pearce ◽  
Waldemar F. Carlo ◽  
Hayden J. Zaccagni ◽  
Robert J. Dabal ◽  
James K. Kirklin
Keyword(s):  
Tricuspid Regurgitation ◽  
Endomyocardial Biopsy

Use of a Long Sheath for Endomyocardial Biopsy Post Heart Transplantation Reduces the Incidence and Severity of Tricuspid Regurgitation

2010 ◽  
Vol 19 ◽  
pp. S83
Author(s):  
S. McKenzie ◽  
M. Brown ◽  
D. Platts ◽  
J. Maddicks-Laws ◽  
G. Javorsky
Keyword(s):  
Heart Transplantation ◽  
Tricuspid Regurgitation ◽  
Endomyocardial Biopsy

The clover technique in endomyocardial biopsy-induced tricuspid regurgitation

2016 ◽  
Vol 203 ◽  
pp. 731-732 ◽  
Author(s):  
M. Pozzi ◽  
I. Pavlakovic ◽  
A. Dondas ◽  
L. Sebbag ◽  
P. Boissonnat ◽  
...  
Keyword(s):  
Tricuspid Regurgitation ◽  
Endomyocardial Biopsy

Studies on Ultrastructure and Cytochemistry of Right Ventricular Endomyocardial Biopsy

1990 ◽  
Vol 48 (3) ◽  
pp. 256-257
Author(s):  
Xia Mingyu ◽  
Ma Wengshu ◽  
Wu Xiangh ◽  
Chen Dong
Keyword(s):  
Light Microscopy ◽  
Right Ventricular ◽  
Dilated Cardiomyopathy ◽  
Endomyocardial Biopsy ◽  
Heart Diseases ◽  
Ultrastructural Localization ◽  
Controlled Study ◽  
Myocardial Biopsy ◽  
Morphological Assessment ◽  
Rheumatic Heart

This paper describes morphological and cytochemistry changes of endomyocardial biopsy in 94 patients. The samples of myoicardium were taken from 32 patients with dilated cardiomyopathy, and sdudied with light and electron microscop. The cytochemical studies in some of these patients were performed at histological and ultrastructure level. This paper also reported the result of myocardial biopsy in 33 patients with serious dysrythmia.The result of this controlled study indicates that morphological assessment in both cardiomyopathy and congenital or rheumatic heart diseases showed no special changes. In patients of dilated cardiomyopathy, the decreased activity of myosin ATPase was secondary to cardial failure. The change of succinate dehydrogenase (SDHase) was not significant with light microscopy. But ultrastructural localization of SDHase activity is valuable. Its activity was found to be localized in endomembrane and ridge of the mitochondria, the activity of this enzyme was decrease, normal, or increase. SDHase activity was more intense in cardial myocytes well-functioning, or ultrastructurally well preserved hearts.

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