scholarly journals Bihemispheric Subcortical Infarcts in the Middle Cerebral Artery Territory

2011 ◽  
Vol 4 ◽  
pp. CCRep.S7121
Author(s):  
Hiroshi Kataoka ◽  
Masahiro Kumazawa ◽  
Tesseki Izumi ◽  
Satoshi Ueno
Keyword(s):  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Internal Carotid ◽  
Small Vessel Disease ◽  
Cortical Lesions ◽  
Middle Cerebral Artery Territory ◽  
Vessel Disease ◽  
History Of ◽  
Resolution Imaging ◽  
The Brain

Background and purpose Previous studies have suggested embolic mechanisms for bihemispheric subcortical infarcts involving the anterior and posterior circulation. However, the mechanism of bihemispheric subcortical infarcts in middle cerebral artery (MCA) territories remains uncertain. We describe a patient with acute bihemispheric subcortical infarcts in restricted MCA territories suggesting an embolic mechanism. Case description A 62-year-old woman with a history of hypertension and hyperlipidemia suddenly presented with left hemiplegia. Diffusion-weighted and T2-weighted magnetic resolution imaging of the brain showed multiple subcortical high intensity in the MCA territories. There were no acute infarctions in the cerebrum, brain stem, or cerebellum, including cortical lesions. The patient had no carotid, internal carotid artery, or MCA disease. Conclusion Bihemispheric subcortical infarcts in the MCA territory are likely to have a proximal embolic source and such infarcts could be associated with multiple subcortical infarcts due to small vessel disease.

scholarly journals Lenticulostriate artery combined with neuroimaging markers of cerebral small vessel disease differentiate the pathogenesis of recent subcortical infarction

2021 ◽  
pp. 0271678X2199262
Author(s):  
Shuai Jiang ◽  
Tian Cao ◽  
Yuying Yan ◽  
Tang Yang ◽  
Ye Yuan ◽  
...  
Keyword(s):  
Magnetic Resonance ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Small Vessel Disease ◽  
Cerebral Small Vessel Disease ◽  
Small Vessel ◽  
Vessel Disease ◽  
Branch Atheromatous Disease ◽  
Lenticulostriate Artery ◽  
Subcortical Infarction

Recent subcortical infarction (RSI) in the lenticulostriate artery (LSA) territory with a non-stenotic middle cerebral artery is a heterogeneous entity. We aimed to investigate the role of LSA combined with neuroimaging markers of cerebral small vessel disease (CSVD) in differentiating the pathogenic subtypes of RSI by whole-brain vessel-wall magnetic resonance imaging (WB-VWI). Fifty-two RSI patients without relevant middle cerebral artery (MCA) stenosis on magnetic resonance angiography were prospectively enrolled. RSI was dichotomized as branch atheromatous disease (BAD; a culprit plaque located adjacent to the LSA origin) (n = 34) and CSVD-related lacunar infarction (CSVD-related LI; without plaque or plaque located distal to the LSA origin) (n = 18). Logistic regression analysis showed lacunes (odds ratio [OR] 9.68, 95% confidence interval [CI] 1.71–54.72; P = 0.010) and smaller number of LSA branches (OR 0.59, 95% CI 0.36–0.96; P = 0.034) were associated with of BAD, whereas severe deep white matter hyperintensities (DWMH) (OR 0.11, 95% CI 0.02–0.71; P = 0.021) was associated with CSVD-related LI. In conclusion, the LSA branches combined with lacunes and severe DWMH may delineate subtypes of SSI. The WB-VWI technique could be a credible tool for delineating the heterogeneous entity of SSI in the LSA territory.

Embolization of a Superficial Temporal Artery to Middle Cerebral Artery Bypass: Case Report

Neurosurgery ◽  
1983 ◽  
Vol 12 (3) ◽  
pp. 342-345 ◽  
Author(s):  
Frances K. Conley
Keyword(s):  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Internal Carotid ◽  
Artery Bypass ◽  
Superficial Temporal Artery ◽  
Temporal Artery ◽  
Bilateral Carotid ◽  
History Of ◽  
Internal Carotid Arteries ◽  
Bilateral Carotid Artery

Abstract This case history of a man with bilateral carotid artery occlusions presents angiographic documentation of the embolization of a superficial temporal-middle cerebral artery bypass. The embolic source was thrombotic and/or atheromatous debris that had collected in the persistent stump of one of the occluded internal carotid arteries.

Natural History of Acute Stroke due to Occlusion of the Middle Cerebral Artery and Intracranial Internal Carotid Artery

2013 ◽  
Vol 24 (4) ◽  
pp. 354-358 ◽  
Author(s):  
María Hernández-Pérez ◽  
Natalia Pérez de la Ossa ◽  
Aitziber Aleu ◽  
Mònica Millán ◽  
Meritxell Gomis ◽  
...  
Keyword(s):  
Carotid Artery ◽  
Natural History ◽  
Internal Carotid Artery ◽  
Acute Stroke ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Internal Carotid ◽  
History Of

scholarly journals A RARE CASE OF RUPTURED ANEURYSM AT THE ORIGIN OF INFERIOR PART OF DUPLICATED MIDDLE CEREBRAL ARTERY

2021 ◽  
Vol 7 (2) ◽  
pp. 146-148
Author(s):  
Achmad Firdaus Sani ◽  
Dedy Kurniawan
Keyword(s):  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Internal Carotid ◽  
Anatomical Variation ◽  
Ruptured Aneurysm ◽  
Endovascular Coiling ◽  
Left Middle Cerebral Artery ◽  
History Of ◽  
Head Computed Tomography ◽  
Left Middle

Duplicated middle cerebral artery (DMCA) is an anomalous vessel arises from the internal carotid artery (ICA). This anatomical variation is rare. Aneurysm with this anatomical variation and unusual form was very rare. Even though this kind of aneurysm is rare, it was often ruptured. In this paper, we report a case of 40-years old female with abrupt decreased of consciousness as a chief complaint, along with severe headache one day earlier, no history of head trauma, and there was nuchal rigidity. She didn’t had history of hypertension before. Head computed tomography showed subarachnoid hemmorrhage (SAH) mostly on the left sylvian fissure with Hunt and Hess scale was 3 and Fisher scale was 2, while the cerebral angiography showed duplication of the left middle cerebral artery in which the inferior part of the MCA duplication has ruptured aneurysm at the origin. Treatment option for this aneurysm is endovascular coiling with preserved of the inferior part of duplicated MCA. Result of this treatment shows a good outcome.

Embolic Hemiplegia following Blunt Thoraco-Cervical Trauma

1978 ◽  
Vol 18 (1) ◽  
pp. 63-65
Author(s):  
S. Sivanesan ◽  
V. Vignaendra
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Middle Cerebral Artery ◽  
Pulmonary Vein ◽  
Cerebral Artery ◽  
Internal Carotid ◽  
Post Mortem ◽  
Cervical Trauma ◽  
The Brain ◽  
Artery Branch

A 21-year-old man became hemiplegic 36 hours after being assaulted on the neck and chest. A carotid angiogram showed middle cerebral artery branch occlusion. He died 5 days after the assault. Post mortem examination showed infarction of the brain. This infarction is attributed to an embolus that could have arisen either in the internal carotid artery or a tributary of the pulmonary vein.

scholarly journals Nasopharyngeal Carcinoma Presenting as a Dual Territory Stroke: The Hyperdense Artery Sign

2011 ◽  
Vol 26 (2) ◽  
pp. 37-38
Author(s):  
Ian C. Bickle
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Cerebral Infarction ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Internal Carotid ◽  
Ct Imaging ◽  
The Body ◽  
Attenuation Value ◽  
The Brain

This 63 year-old chinese female, with both diabetes and hypertension, underwent CT imaging of the brain after presenting with a progressive left sided hemiplegia.  The ‘hyperdense artery sign’ is a generic description that can be evident in any artery of the body on unenhanced CT, occurring due to the presence of intraluminal thrombosis (Figure 1).  It is a well-established sign, most commonly described in CT imaging of the brain, where it is visualised in the vast majority of cases in the middle cerebral artery in the context of an acute cerebral infarction.1   It occurs uncommonly elsewhere, with the internal carotid artery (ICA) and basilar artery being other clinically significant sites. The ‘hyperdense ICA’ sign has been reported to be a reliable and highly specific marker of thromboembolic occlusion of the internal carotid artery.2  The ‘hyperdense artery sign’ is related to the attenuation value of intraluminal thrombus.  The CT attenuation value (Hounsfield unit or HU) of normal blood is dependent on the haematocrit, ranging from 20 to 30 HU.  As the process of thrombus retraction occurs, its water content decreases, increasing the concentration of haemoglobin within the clot.  As a result this raises the attenuation value of the thrombus to 50–80 H. So the term ‘hyperdense’ is given.3 In this case, it proved to be the presenting symptom for an undiagnosed nasopharyngeal tumour, the thrombus likely developing as a complication of the surrounding tumour within the nasopharyngeal recess.  The resultant outcome was a dual territory cerebral infarction of the anterior and middle cerebral artery territories, both supplied by branches of the internal carotid artery (Figures 2a & 2b).  

Response of canine internal carotid system to acetylcholine

1991 ◽  
Vol 261 (5) ◽  
pp. H1392-H1396
Author(s):  
G. Dieguez ◽  
E. Nava-Hernandez ◽  
J. Valle ◽  
A. L. Garcia-Villalon ◽  
J. L. Garcia ◽  
...  
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Internal Carotid ◽  
Arterial System ◽  
Low Concentrations ◽  
Cervical Portion ◽  
The Brain

The reactivity of the canine internal carotid system to acetylcholine (10(-8)-10(-4) M) was studied isometrically with 4-mm cylindrical segments from cervical and cavernous portions of the internal carotid artery and from the middle cerebral artery. Under control conditions, the cervical portion relaxed to every dose, the cavernous portion relaxed at low concentrations (10(-8)-10(-6) M) and contracted at higher concentrations (10(-5)-10(-4) M), whereas the middle cerebral artery contracted to every dose of acetylcholine. These responses were blocked by atropine (10(-6) M). Without endothelium, the cervical portion exhibited a lower relaxation, the cavernous portion contracted, and the middle cerebral artery was practically unresponsive to acetylcholine. These responses were also blocked by atropine. It suggests that the reactivity of the internal carotid system to acetylcholine 1) is endothelium dependent and 2) changes as it courses toward the brain, and this could be related to different embryological origin of the components of this arterial system.

Protective effects of methyl prednisolone and dimethyl sulfoxide in experimental middle cerebral artery embolectomy

1978 ◽  
Vol 49 (4) ◽  
pp. 508-516 ◽  
Author(s):  
Ranjit K. Laha ◽  
Manuel Dujovny ◽  
Pedro J. Barrionuevo ◽  
Samuel C. DeCastro ◽  
Harold R. Hellstrom ◽  
...  
Keyword(s):  
Middle Cerebral Artery ◽  
Dimethyl Sulfoxide ◽  
Cerebral Artery ◽  
Internal Carotid ◽  
Protective Effects ◽  
Arterial Embolism ◽  
Content Type ◽  
Methyl Prednisolone ◽  
Children And Young Adults ◽  
The Brain

✓ Acute arterial embolism continues to be a major cause of stroke morbidity in children and young adults. Potential therapy modalities include medical management and/or cerebral revascularization. The canine middle cerebral artery (MCA) was embolized by means of a pliable cylinder, 8 mm long by 1.6 mm in diameter, via the internal carotid artery. Control and experimental embolectomies were performed 6 hours following embolization. The experimental animals were treated with either dimethyl sulfoxide (DMSO) or methyl prednisolone. In the control animals, the average area of infarction in the brain was 1.45 cu cm. The animals treated with methyl prednisolone (2 mg/kg) or DMSO (2 gm/kg) showed no infarction of the brain, whereas methyl prednisolone (30 mg/kg) did not prevent infarction.

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