scholarly journals Challenge Test Results in Patients With Suspected Penicillin Allergy, but No Specific IgE

2011 ◽  
Vol 3 (2) ◽  
pp. 118 ◽  
Author(s):  
Anne Holm ◽  
Holger Mosbech
2018 ◽  
Vol 176 (1) ◽  
pp. 8-14 ◽  
Author(s):  
Noriyuki Yanagida ◽  
Sakura Sato ◽  
Nobuyuki Maruyama ◽  
Kyohei Takahashi ◽  
Ken-ichi Nagakura ◽  
...  

Allergy ◽  
2008 ◽  
Vol 63 (7) ◽  
pp. 897-902 ◽  
Author(s):  
V. Van Kampen ◽  
S. Rabstein ◽  
I. Sander ◽  
R. Merget ◽  
T. Brüning ◽  
...  

2018 ◽  
Vol 462 ◽  
pp. 9-12 ◽  
Author(s):  
Vijaya Knight ◽  
Molly L. Wolf ◽  
Anita Trikha ◽  
Douglas Curran-Everett ◽  
Melissa Hiserote ◽  
...  

1991 ◽  
Vol 21 (2) ◽  
pp. 305-312 ◽  
Author(s):  
John Bancroft ◽  
Ann Cook ◽  
David Davidson ◽  
John Bennie ◽  
Guy Goodwin

SYNOPSISThe neuroendocrine response to L-tryptophan infusion was measured at two stages of the menstrual cycle, premenstrually and postmenstrually, in 13 women with and 13 women without premenstrual depression (the MC and NMC groups respectively). Previous studies have shown that in non-depressed women, this challenge test results in an increase in circulating prolactin and growth hormone. In depressed women both responses are blunted. In this study the growth hormone and cortisol responses were smaller in the MC group than the NMC group on both occasions. The prolactin response was blunted premenstrually compared with postmenstrually in both groups. These findings suggest that women who experience premenstrual depression may have neuroendocrine abnormalities throughout the cycle. The neurotransmitter abnormalities reflected in these altered endocrine responses appear to interact with neuroendocrine changes that normally occur premenstrually resulting in a vulnerability to depression at that phase of the cycle.


2014 ◽  
Vol 2014 ◽  
pp. 1-13 ◽  
Author(s):  
Beata Uziębło-Życzkowska ◽  
Grzegorz Gielerak ◽  
Paweł Siedlecki ◽  
Beata Pająk

Brugada Syndrome (BS) is an inherited channelopathy associated with a high incidence of sudden cardiac death. The paper presents the discovery of new genetic variants ofSCN5Agene which might be associated with the development of a concealed form of Brugada Syndrome. The study involved a group of 59 patients (37 men) with suspected concealed form of Brugada Syndrome. Pharmacological provocation with intravenous ajmaline administration was performed. Six patients with positive test results were subjected to molecular analysis ofSCN5Agene with MSSCP method. Additionally, MSSCP genotyping was performed for samples obtained from the family members with Brugada Syndrome, despite the fact that they had negative ajmaline challenge test results. Genetic examinations of theSCN5Agene at 6 positive patients showed 6 known polymorphisms, 8 new single nucleotide point (SNP) variants located at exons, and 12 new single nucleotide point variants located at introns. Among new SNPs localized inSCN5Agene exons three SNPs affected the protein sequence.


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