Sellar Tumor Mass: Meningioma Mimicking Pituitary Macroadenoma

2015 ◽  
Vol 11 (3) ◽  
pp. 394-395
Author(s):  
C Poiana
Keyword(s):  
Pituitary Macroadenoma ◽  
Tumor Mass ◽  
Sellar Tumor

scholarly journals The Volume of Tumor Mass and Visual Field Defect in Patients with Pituitary Macroadenoma

2011 ◽  
Vol 25 (1) ◽  
pp. 37 ◽  
Author(s):  
Jung Pil Lee ◽  
In Won Park ◽  
Yun Suk Chung
Keyword(s):  
Visual Field ◽  
Visual Field Defect ◽  
Pituitary Macroadenoma ◽  
Tumor Mass ◽  
Field Defect

Ultrastructural Correlations between Clonal Human Tumor Colonies and in Vivo Neoplasms

1982 ◽  
Vol 40 ◽  
pp. 210-211
Author(s):  
M.J. Murphy ◽  
R.R. Price ◽  
J.C. Sloman
Keyword(s):  
Cultured Cells ◽  
Human Tumor ◽  
Cell Type ◽  
Tumor Mass ◽  
Initial Cell ◽  
Cloning Assay ◽  
Human Tumor Cloning ◽  
The Relationship

The in vitro human tumor cloning assay originally described by Salmon and Hamburger has been applied recently to the investigation of differential anti-tumor drug sensitivities over a broad range of human neoplasms. A major problem in the acceptance of this technique has been the question of the relationship between the cultured cells and the original patient tumor, i.e., whether the colonies that develop derive from the neoplasm or from some other cell type within the initial cell population. A study of the ultrastructural morphology of the cultured cells vs. patient tumor has therefore been undertaken to resolve this question. Direct correlation was assured by division of a common tumor mass at surgical resection, one biopsy being fixed for TEM studies, the second being rapidly transported to the laboratory for culture.

Abstract #803: Cushing Disease Due to Pituitary Macroadenoma: Biochemical Characteristics in a Pediatric Cohort

2015 ◽  
Vol 21 ◽  
pp. 148-149
Author(s):  
Ricardo Correa ◽  
Maria Batsis ◽  
Prashant Chittiboina ◽  
Pooja Raghavan ◽  
Elena Belyavskaya ◽  
...  
Keyword(s):  
Pituitary Macroadenoma ◽  
Cushing Disease ◽  
Biochemical Characteristics

Abstract #507 Panhypopituitarism Secondary to Pituitary Macroadenoma with Optic Chiasm Compression and Possible Apoplexy

2019 ◽  
Vol 25 ◽  
pp. 252
Author(s):  
Olesya Petrenko ◽  
Wilhelmine Wiese-Rometsch
Keyword(s):  
Optic Chiasm ◽  
Pituitary Macroadenoma

Abstract #470 Case of Recurrent Nonfunctioning Pituitary Macroadenoma: Management Issues in an Endocrinology Clinic

2019 ◽  
Vol 25 ◽  
pp. 229-230
Author(s):  
Gabriela Mroueh ◽  
Ramachandra Chemitiganti ◽  
Syed-azhar Hassan
Keyword(s):  
Pituitary Macroadenoma ◽  
Endocrinology Clinic ◽  
Management Issues

Abstract #125 Hypercortisolism in a Patient with a Mediastinal Mass and Pituitary Macroadenoma: A Case Report

2018 ◽  
Vol 24 ◽  
pp. 15-16
Author(s):  
Lima Lawrence ◽  
Alexandra Mikhael ◽  
Khawla Ali ◽  
Varun Kshettry ◽  
Pablo Recinos ◽  
...  
Keyword(s):  
Case Report ◽  
Mediastinal Mass ◽  
Pituitary Macroadenoma

Die Anämie bei malignen Tumorerkrankungen

1989 ◽  
Vol 28 (06) ◽  
pp. 247-254
Author(s):  
E. Aulbert
Keyword(s):  
Malignant Tumors ◽  
Gradient Centrifugation ◽  
Tumor Resection ◽  
Hemoglobin Concentration ◽  
Limiting Factor ◽  
Novikoff Hepatoma ◽  
Tumor Mass ◽  
Lysosomal Fraction ◽  
Proliferation Activity ◽  
Morris Hepatoma

The cellular uptake and lysosomal accumulation of 67Ga-labelled transferrin within tumors of different malignancy were examined using tissue fractionation and immunological techniques. As tumor models the slowly growing Morris hepatoma 5123C, the moderately growing Novikoff hepatoma and the fast and aggressive Yoshida hepatoma AH 130 were investigated. Isolation of subcellular fractions of tumor homogenates was performed by differential centrifugation and density-gradient centrifugation. The intracellular 67Gatransferrin was found to be highly concentrated within the purified lysosomes. The transferrin within the lysosomal fraction was identified by radial immunodiffusion technique using monospecific antiserum. The accumulation of 67Gatransferrin by the tumors resulted in a faster disappearance of 67Ga-transferrin from the blood. This loss of circulating 67Ga-transferrin correlated with the proliferation activity and the spread of the tumors. Since transferrin is indispensible for the utilization of iron by the heme-synthesizing red cell precursors, transferrin concentration in the blood is the limiting factor for the utilization of iron in hemoglobin synthesis. Thus, in a further series of experiments we investigated the development of anemia in tumor-bearing rats. With increasing tumor mass a progressive fall of hemoglobin concentration was found. The anemia was more severe in the faster growing Novikoff hepatoma than in the slowly growing Morris hepatoma. The most significant reduction of hemoglobin concentration was found in the very fast growing Yoshida hepatoma. After total tumor resection hemoglobin concentration and red blood cell count normalized completely within 6-8 weeks. We conclude from these data that the uptake of transferrin by the tumor cells results in a faster disappearance of transferrin from the blood. This loss of circulating transferrin correlates with tumor mass and proliferation activity and is one of the factors responsible for the anemia seen in patients with malignant tumors.

Die Anämie bei malignen Tumorerkrankungen

1989 ◽  
Vol 28 (05) ◽  
pp. 193-200 ◽  
Author(s):  
E. Aulbert
Keyword(s):  
Cellular Uptake ◽  
Tumor Tissue ◽  
Malignant Tumors ◽  
The Other ◽  
Novikoff Hepatoma ◽  
Cellular Accumulation ◽  
Tumor Mass ◽  
Proliferation Activity ◽  
Morris Hepatoma ◽  
Cellular Incorporation

Cellular uptake of 67Ga-labelled transferrin by the tumor tissue was studied in rats with tumors of different malignancy and different tumor mass using the slowly growing Morris hepatoma 5123C, the moderately growing Novikoff hepatoma and the very fast and aggressive Yoshida hepatoma AH130. The cellular accumulation of 67Ga-transferrin was found to correlate with the proliferation activity of the tumor. The 67Ga-transferrin concentration in the very fast growing Yoshida hepatoma was 4.8 times higher than the concentration in the slowly growing Morris hepatoma. The uptake of 67Ga-transferrin by the tumors resulted in a faster disappearance of circulating 67Ga-transferrin from the blood. The rate of disappearance correlated with the proliferation activity and the spread of the tumors. Using tumors of identical size the elimination of 67Ga-transferrin from the blood was much faster in the rats with Yoshida hepatoma than in those with the slowly growing Morris hepatoma. On the other hand, using tumors of different tumor size it could be demonstrated that the rate of disappearance of 67Ga-transferrin from the blood correlated directly with tumor mass. It is concluded that cellular incorporation of transferrin within the tumor cells results in a loss of circulating transferrin, which correlates with tumor mass and proliferation of tumor. This mechanism is supposed to be the cause for the hypotransferrinemia seen in patients with malignant tumors.

Pituitary Macroadenoma Presenting with Complete Nasal Obstruction: A Case Report and Literature Review

2020 ◽  
Author(s):  
Mitchell McDonough ◽  
Andre A. Le Roux ◽  
Christopher J. Chin
Keyword(s):  
Case Report ◽  
Literature Review ◽  
Nasal Obstruction ◽  
Pituitary Macroadenoma
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