scholarly journals Surgical Outcomes of Transmastoid Facial Nerve Decompression for Patients With Traumatic Facial Nerve Paralysis

2021 ◽  
Vol 17 (4) ◽  
pp. 294-300
Author(s):  
Harun Gur ◽  
◽  
Kemal Gorur ◽  
Onur Ismi ◽  
Yusuf Vaysioglu ◽  
...  
CoDAS ◽  
2018 ◽  
Vol 30 (1) ◽  
Author(s):  
David Victor Kumar Irugu ◽  
Anoop Singh ◽  
Sravan CH ◽  
Achyuth Panuganti ◽  
Anand Acharya ◽  
...  

2012 ◽  
Vol 2012 ◽  
pp. 1-5 ◽  
Author(s):  
Sertac Yetiser

Management of traumatic facial nerve disorders is challenging. Facial nerve decompression is indicated if 90–95% loss of function is seen at the very early period on ENoG or if there is axonal degeneration on EMG lately with no sign of recovery. Middle cranial or translabyrinthine approach is selected depending on hearing. The aim of this study is to present retrospective review of 10 patients with sudden onset complete facial paralysis after trauma who underwent total facial nerve decompression. Operation time after injury is ranging between 16 and105 days. Excitation threshold, supramaximal stimulation, and amplitude on the paralytic side were worse than at least %85 of the healthy side. Six of 11 patients had HBG-II, one patient had HBG-I, 3 patients had HBG-III, and one patient had HBG-IV recovery. Stretch, compression injuries with disruption of the endoneurial tubules undetectable at the time of surgery and lack of timely decompression may be associated with suboptimal results in our series.


2019 ◽  
Vol 130 (1) ◽  
pp. 200-205
Author(s):  
Christopher Blake Sullivan ◽  
Daniel Q. Sun ◽  
Vivian L. Zhu ◽  
Marlan R. Hansen ◽  
Bruce J. Gantz

1974 ◽  
Vol 83 (5) ◽  
pp. 582-595 ◽  
Author(s):  
James A. Greer ◽  
D. Thane R. Cody ◽  
Edward H. Lambert ◽  
Louis H. Weiland

An experimental model was employed to establish an endotemporal bone facial nerve paralysis in cats. Twelve facial nerves were initially surgically decompressed— Both bony decompression and sheath decompression—to determine if any harm was done to the nerves by these procedures. Transient harm was found in 3 of the 12 nerves. After the course of the facial paralysis without any decompression was determined, 39 cats underwent either bony or sheath nerve decompression, both immediately after injury and after a delay. The cats were followed clinically and electrophysiologically; the nerve excitability test and the amplitude and latency of muscle response evoked by nerve stimulation were used to evaluate nerve function. While immediate postinjury bony decompression resulted in slightly earlier recovery times, immediate sheath splitting significantly lengthened recovery times and worsened the electrophysiologic test results. Histologic studies confirmed the clinical and electrophysiologic results anatomically. Delayed nerve decompression, either bony or sheath, was not associated with faster recovery rates.


2009 ◽  
Vol 124 (4) ◽  
pp. 402-406 ◽  
Author(s):  
H E Cha ◽  
M K Baek ◽  
J H Yoon ◽  
B K Yoon ◽  
M J Kim ◽  
...  

AbstractObjectives:To evaluate the causes, treatment modalities and recovery rate of paediatric facial nerve paralysis.Materials and methods:We analysed 24 cases of paediatric facial nerve paralysis diagnosed in the otolaryngology department of Gachon University Gil Medical Center between January 2001 and June 2006.Results:The most common cause was idiopathic palsy (16 cases, 66.7 per cent). The most common degree of facial nerve paralysis on first presentation was House–Brackmann grade IV (15 of 24 cases). All cases were treated with steroids. One of the 24 cases was also treated surgically with facial nerve decompression. Twenty-two cases (91.6 per cent) recovered to House–Brackmann grade I or II over the six-month follow-up period.Conclusion:Facial nerve paralysis in children can generally be successfully treated with conservative measures. However, in cases associated with trauma, radiological investigation is required for further evaluation and treatment.


2019 ◽  
Vol 13 (3) ◽  
pp. 44-48
Author(s):  
B K Bhattacharya ◽  
◽  
Subhajit Sarkar ◽  

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