Case Study 192 Advanced Optic Nerve Cupping

2013 ◽  
pp. 433-433
Author(s):  
Roger P. Harrie ◽  
Cynthia J. Kendall
Keyword(s):  
Author(s):  
Dr. Harsha S. ◽  
Dr. Mamatha KV.

The optic nerve carries visual information from your eye to your brain. Optic neuritis is when your optic nerve becomes inflamed. Optic neuritis can flare up suddenly from an infection or nerve disease. The inflammation usually causes temporary vision loss that typically happens in only one eye. Those with Optic neuritis sometimes experience pain. As you recover and the inflammation goes away, your vision will likely return. There are no direct references in our classics regarding optic neuritis but can be contemplated as a condition by name Parimlayi Timira. The specific management as such is not cited but a transcendence approach can be done with adopting the treatment which has the ability to pacify the already occurred pathology and prevent the further development of the disease. One such interesting case study on Optic neuritis is elaborated here where in specific treatment modalities (Shodana, Shamana and Kriyakalpas) played role in pacifying the condition.


2013 ◽  
pp. 295-295
Author(s):  
Roger P. Harrie ◽  
Cynthia J. Kendall

2013 ◽  
pp. 365-366
Author(s):  
Roger P. Harrie ◽  
Cynthia J. Kendall

2016 ◽  
Vol 35 (1) ◽  
pp. 13-18 ◽  
Author(s):  
Jamie M. Musgrove ◽  
Cheryl Riley

AbstractSepto-optic dysplasia (SOD) is a rare congenital heterogeneous malformation. SOD was formerly known as de Morsier syndrome, which associated a midline brain defect such as an absent septum pellucidum with optic nerve hypoplasia. The diagnosis of SOD is made when there are two or more characteristics of the classic triad. The triad consists of optic nerve hypoplasia, pituitary hormone abnormalities, and midline brain defects, although it can vary in the severity of clinical presentation and phenotype. The purpose of this article is to review a case and analyze the literature regarding prevalence, etiology, clinical presentation, diagnosis, and management of SOD.


2015 ◽  
Vol 2015 ◽  
pp. 1-3
Author(s):  
A. Bruce Janati ◽  
Naif Saad ALGhasab ◽  
Fazal Haq ◽  
Ahmad Abdullah ◽  
Aboubaker Osman

Introduction. Laurence-Moon-Biedl (LMB) syndrome is a rare autosomal-recessive ciliopathy with manifold symptomatology. The cardinal clinical features include retinitis pigmentosa, obesity, intellectual delay, polydactyly/syndactyly, and hypogenitalism. In this paper, the authors report on three siblings with Laurence-Moon-Biedl syndrome associated with a probable pseudocycloid form of congenital nystagmus.Methods. This was a case study conducted at King Khaled Hospital.Results. The authors assert that the nystagmus in Laurence-Moon-Biedl syndrome is essentially similar to idiopathic motor-defect nystagmus and the nystagmus seen in optic nerve hypoplasia, ocular albinism, and bilateral opacities of the ocular media.Conclusion. The data support the previous hypothesis that there is a common brain stem motor abnormality in sensory-defect and motor-defect nystagmus.


2013 ◽  
pp. 171-172
Author(s):  
Roger P. Harrie ◽  
Cynthia J. Kendall

2013 ◽  
pp. 25-26
Author(s):  
Roger P. Harrie ◽  
Cynthia J. Kendall

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