Transition and Transfer to Adult Care for Adolescents and Young Adults with Advanced Chronic Kidney Disease

2021 ◽  
pp. 793-810
Author(s):  
Lorraine E. Bell ◽  
Dirk Bethe
Author(s):  
Andrea R. Molino ◽  
Judith Jerry-Fluker ◽  
Meredith A. Atkinson ◽  
Susan L. Furth ◽  
Bradley A. Warady ◽  
...  

2015 ◽  
Vol 36 (9) ◽  
pp. 734-742 ◽  
Author(s):  
Stephen R. Hooper ◽  
Nina Laney ◽  
Jerilynn Radcliffe ◽  
Divya Moodalbail ◽  
Erum A. Hartung ◽  
...  

2017 ◽  
Vol 24 (6) ◽  
pp. 405-409 ◽  
Author(s):  
Maria E. Díaz-González de Ferris ◽  
Marta Del Villar-Vilchis ◽  
Ricardo Guerrero ◽  
Victor M. Barajas-Valencia ◽  
Emily B. Vander-Schaaf ◽  
...  

2019 ◽  
pp. 2-3

Impaired phosphate excretion by the kidney leads to Hyperphosphatemia. It is an independent predictor of cardiovascular disease and mortality in patients with advanced chronic kidney disease (stage 4 and 5) particularly in case of dialysis. Phosphate retention develops early in chronic kidney disease (CKD) due to the reduction in the filtered phosphate load. Overt hyperphosphatemia develops when the estimated glomerular filtration rate (eGFR) falls below 25 to 40 mL/min/1.73 m2. Hyperphosphatemia is typically managed with oral phosphate binders in conjunction with dietary phosphate restriction. These drugs aim to decrease serum phosphate by binding ingested phosphorus in the gastrointestinal tract and its transformation to non-absorbable complexes [1].


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