scholarly journals Oscillations in a white blood cell production model with multiple differentiation stages

2019 ◽  
Vol 80 (3) ◽  
pp. 575-600 ◽  
Author(s):  
Franziska Knauer ◽  
Thomas Stiehl ◽  
Anna Marciniak-Czochra
2004 ◽  
Vol 327 (3) ◽  
pp. 201-210 ◽  
Author(s):  
Samuel Bernard ◽  
Jacques Bélair ◽  
Michael C. Mackey

1998 ◽  
Vol 192 (2) ◽  
pp. 167-181 ◽  
Author(s):  
Taryn Hearn ◽  
Caroline Haurie ◽  
Michael C. Mackey

2008 ◽  
Vol 36 (9) ◽  
pp. 1091-1097 ◽  
Author(s):  
Eun Wha Choi ◽  
Hye Cheong Koo ◽  
Il Seob Shin ◽  
Young Jin Chae ◽  
Jong Hwa Lee ◽  
...  

1951 ◽  
Vol 43 (4) ◽  
pp. 525-531 ◽  
Author(s):  
James S. Dinning ◽  
Lou Dewees Payne ◽  
Paul L. Day

2021 ◽  
pp. 62-70
Author(s):  
Mathias Clasen

The effects of horror movies on physical health have received little scientific attention, but there are anecdotal reports of people getting hurt and even dying as a result of horror exposure. Such cases, however, are extremely rare and usually poorly documented. While it is possible to die from fright, the odds of dying from fright induced by a horror movie are not worth worrying about. A little research has investigated potential health benefits of horror exposure. Some research suggests that horror movies may have metabolic benefits, and researchers have found that watching horror movies can increase white blood cell production and blood coagulant, but more research is needed. Indirect evidence does suggest that watching horror movies may increase self-perceived physical health or well-being.


2008 ◽  
Vol 36 (7) ◽  
pp. 807-815 ◽  
Author(s):  
Eun Wha Choi ◽  
Il Seob Shin ◽  
Young Jin Chae ◽  
Hye Cheong Koo ◽  
Jong Hwa Lee ◽  
...  

1996 ◽  
Vol 76 (02) ◽  
pp. 184-186 ◽  
Author(s):  
Kenji lijima ◽  
Fumiyo Murakami ◽  
Yasushi Horie ◽  
Katsumi Nakamura ◽  
Shiro Ikawa ◽  
...  

SummaryA 74-year-old female developed pneumonia following herpes simplex encephalitis. Her white blood cell counts reached 28,400/μl, about 90% of which consisted of granulocytes. The polymorphonuclear (PMN) elastase/α1-arantitrypsin complex levels increased and reached the maximum of 5,019 ng/ml, indicating the release of a large amount of elastase derived from the granulocytes. The mechanism of PMN elastase release was most likely to be granulocyte destruction associated with phagocytosis. The cleavage of fibrinogen and fibrin by PMN elastase, independent of plasmin, was indicated by the presence of the fragments in immunoprecipitated plasma from the patient corresponding to elastase-induced FDP D and DD fragments and the absence of fragments corresponding to plasmin-induced FDP D and DD fragments on SDS-PAGE. These findings suggested that the large amount of PMN elastase released from the excessive numbers of granulocytes in this patient with herpes simplex encephalitis and pneumonia, induced the cleavage of fibrinogen and fibrin without the participation of plasmin.


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