Neuroanatomical distribution of angiotensin-II-like neuropeptide within the central nervous system of the crab Chasmagnathus; physiological changes triggered by water deprivation

2010 ◽  
Vol 341 (1) ◽  
pp. 181-195 ◽  
Author(s):  
Lia Frenkel ◽  
Beatriz Dimant ◽  
Enrique L. Portiansky ◽  
Hans Imboden ◽  
Héctor Maldonado ◽  
...  
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Angiotensin Ii ◽  
Water Deprivation ◽  
Physiological Changes ◽  
The Central Nervous System

Central site for pressor action of blood-borne angiotensin in rat

1980 ◽  
Vol 239 (3) ◽  
pp. R358-R361 ◽  
Author(s):  
G. D. Fink ◽  
J. R. Haywood ◽  
W. J. Bryan ◽  
W. Packwood ◽  
M. J. Brody
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Angiotensin Ii ◽  
Pressor Response ◽  
Third Ventricle ◽  
Central Component ◽  
The Third ◽  
Electrolytic Lesions ◽  
The Central Nervous System ◽  
The Difference

A previous study demonstrated that the threshold dose of intra-arterial angiotensin II required to induce a pressor response in the rat was significantly lower when the drug was administered into the carotid artery than when administered into the abdominal aorta. This result was interpreted to indicate that part of the increase in arterial pressure produced by low concentrations of blood-borne angiotensin in this species was the result of an effect on structures in the central nervous system selectively accessible via the carotid vascular bed. The purpose of the present study was to establish more precisely the site of the pressor action of angiotensin within the central nervous system. The central component of the pressor effect of angiotensin was quantified as the difference in pressor responses to intracarotid and intra-aortic infusions of angiotensin II (delta c-a). In conscious rats, delta c-a was attenuated by administration of the angiotensin antagonist, saralasin, into the third cerebral ventricle. In rats with chronic electrolytic lesions of the anteroventral third ventricle (AV3V), delta c-a was abolished. Periventricular structures surrounding the third ventricle appear to mediate the central component of the pressor action of blood-borne angiotensin in the rat.

scholarly journals Superoxide Mediates the Actions of Angiotensin II in the Central Nervous System

2002 ◽  
Vol 91 (11) ◽  
pp. 1038-1045 ◽  
Author(s):  
Matthew C. Zimmerman ◽  
Eric Lazartigues ◽  
Julie A. Lang ◽  
Puspha Sinnayah ◽  
Iman M. Ahmad ◽  
...  
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Angiotensin Ii ◽  
The Central Nervous System

Central Nervous System Pressor Responses in Rats Susceptible and Resistant to Sodium Chloride Hypertension

1978 ◽  
Vol 55 (s4) ◽  
pp. 225s-227s ◽  
Author(s):  
T. Ikeda ◽  
L. Tobian ◽  
J. Iwai ◽  
Patricia Goossens
Keyword(s):  
Blood Pressure ◽  
Central Nervous System ◽  
Nervous System ◽  
Sodium Chloride ◽  
Angiotensin Ii ◽  
Hypertonic Saline ◽  
Pressor Effect ◽  
Left Lateral Ventricle ◽  
Pressor Responses ◽  
The Central Nervous System

1. The pressor responses to hypertonic saline and angiotensin II introduced into the left lateral ventricle were both significantly greater in salt-sensitive (S) rats compared with salt-resistant (R) rats, with all rats on a low Na diet. 2. When S rats were given thiazide to nullify the pressor effect of dietary NaCl, their blood pressure averaged only 5 mmHg higher than that of the R rats; nevertheless, these S rats had significantly higher central nervous system pressor responses to angiotensin II and hypertonic saline. 3. Thus, if excessive dietary Na increases blood pressure by way of action on the central nervous system, these heightened pressor responses could partially account for the NaCl hypertension in S rats. Alternatively, depressed central nervous system pressor responses in R rats could partially explain the resistance of R rats to NaCl hypertension.

scholarly journals Effect of Chronic Intermittent Hypoxia on Angiotensin II Receptors in the Central Nervous System

2018 ◽  
Vol 41 (2) ◽  
pp. 130-136
Author(s):  
Barbara J. Morgan ◽  
Nicole Schrimpf ◽  
Morgan Rothman ◽  
Ann Mitzey ◽  
Mark S. Brownfield ◽  
...  
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Angiotensin Ii ◽  
Intermittent Hypoxia ◽  
Angiotensin Ii Receptors ◽  
Chronic Intermittent Hypoxia ◽  
The Central Nervous System

scholarly journals Expression of (pro)renin receptor and angiotensin II type 1 receptor on bone marrow‐related neurons in the central nervous system

2013 ◽  
Vol 27 (S1) ◽  
Author(s):  
Christie Diane Kimball ◽  
Wencheng Li ◽  
Andrea Zsombok ◽  
Andrei Derbenev ◽  
Joseph Francis ◽  
...  
Keyword(s):  
Central Nervous System ◽  
Bone Marrow ◽  
Nervous System ◽  
Angiotensin Ii ◽  
The Central Nervous System

scholarly journals Both Nox2‐ and Nox4‐containing NAD(P)H oxidases are required for the full vasopressor effects of angiotensin‐II (Ang‐II) in the central nervous system

2007 ◽  
Vol 21 (6) ◽  
Author(s):  
Jeffrey Peterson ◽  
Melissa A. Burmeister ◽  
Xin Tian ◽  
John A. Stupinski ◽  
Ram V. Sharma ◽  
...  
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Angiotensin Ii ◽  
Ang Ii ◽  
The Central Nervous System

Maternal physiology

2020 ◽  
pp. 45-68
Author(s):  
Korede Adekanye ◽  
Abrie Theron
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Regional Anaesthesia ◽  
Physiological Changes ◽  
Medical Conditions ◽  
Comprehensive Review ◽  
Anatomy And Physiology ◽  
The Central Nervous System ◽  
Time Periods

During pregnancy, maternal anatomy and physiology changes significantly. It is important to be aware of what is considered ‘normal’ for pregnancy, in order to promptly recognize and treat medical conditions that may subsequently develop in the antenatal, intrapartum, or postnatal time periods. This chapter sets out the anatomical and physiological changes according to each system: starting with a comprehensive review of the cardiovascular and respiratory changes, which have the most significant adaptations, through the renal, gastro-intestinal, haematological, and endocrine changes, and ending with the central nervous system. Altered anatomy and physiology will affect the conduct of general and regional anaesthesia. The difficulties associated with the obstetric airway, maternal oxygenation, and positioning during anaesthesia are clearly described.

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