A case of acute cerebellar ataxia with two possible etiologies: viral cerebellitis and paraneoplastic antibody-positive ovarian cancer

2020 ◽  
Vol 41 (8) ◽  
pp. 2285-2287
Author(s):  
Hyunjin Ju ◽  
Mina Lee ◽  
Kayeong Im ◽  
Yon Hee Kim ◽  
Kyum-Yil Kwon
2020 ◽  
Vol 18 (1) ◽  
pp. 16
Author(s):  
OKKY PRASETYO ◽  
ERNY PRASETYO ◽  
DENNY PRASETYO

<p><em>Acute Cerebellar ataxia is a motor control or coordination disorder that can be caused by several things, most often in children due to viral infections including paramyxovirus, head trauma, stroke and intoxication with varying prognosis. The paramyxovirus causes parotitis is highly neurotrophic with aseptic meningitis, encephalitis, hydrocephalus, decreased blood flow on the cerebellum and vasculitis. Those mechanisms implicated on many clinical symptoms, including cerebellar ataxia. Management is only limited to supportive and rehabilitative because symptoms will improve within 2-3 weeks.</em><em></em></p><p><em> </em></p><p>Keywords : acute cerebellar ataxia, parotitis, clinical evaluation, management</p><p><strong> </strong></p>


2013 ◽  
Vol 44 (05) ◽  
pp. 265-267 ◽  
Author(s):  
Chihiro Yonee ◽  
Yoshihiro Maegaki ◽  
Yuichi Kodama ◽  
Hiroshi Hayami ◽  
Yukitoshi Takahashi ◽  
...  

Author(s):  
I.M.S. Sawhney ◽  
S. Prabhakar ◽  
U.K. Dhand ◽  
J.S. Chopra

1923 ◽  
Vol 19 (4) ◽  
pp. 52-60
Author(s):  
G. V. Pervushin

There are only about 30 descriptions of cases of the so-called "acute ataxia", which usually develops after infectious diseases, in the literature available to me, so every case of this disease deserves our attention. The entire literature on the subject should be divided into two periods: in the first, all cases of acute ataxia, both spinal and cerebellar, were described together, without distinction; the second period begins with the works of Bekhterev and Nonne, who singled out cases of acute cerebellar ataxia into a special group.


2020 ◽  
Vol 13 (2) ◽  
pp. 1006-1012
Author(s):  
Aikaterini Liapi ◽  
Apostolos Sarivalasis

The reported case is a 61-year-old woman, admitted for gradual onset of gait disturbances and dysphonia. The serum immunological panel revealed anti-Yo autoantibodies, suggestive of a paraneoplastic syndrome (PNS). A PET-CT revealed a suspicious left ovarian mass with retroperitoneal nodal involvement, and the histological assessment of surgical samples confirmed a FIGO IIIC high-grade serous ovarian cancer (HGSOC). Deemed inoperable at first, the patient was treated by carboplatin and paclitaxel chemotherapy, after which she refused surgical debulking. At the end of her systemic treatment, the patient only experienced a transient improvement of the cerebellar ataxia. Despite the suboptimal oncological treatment, the patient still presents stable disease and is free of progression 7 years from her diagnosis. This case study illustrates the favorable effect of PNS occurrence on oncological outcome in a patient with advanced HGSOC. The absence of recurrence despite the presence of residual disease after the systemic treatment is unusual and could be related to the PNS.


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