scholarly journals Correction to: Neck cooling induces blood pressure increase and peripheral vasoconstriction in healthy persons

Author(s):  
Julia Koehn ◽  
Ruihao Wang ◽  
Carmen de Rojas Leal ◽  
Bernd Kallmünzer ◽  
Klemens Winder ◽  
...  

A Correction to this paper has been published: https://doi.org/10.1007/s10072-021-05355-3

2020 ◽  
Vol 41 (9) ◽  
pp. 2521-2529
Author(s):  
Julia Koehn ◽  
Ruihao Wang ◽  
Carmen de Rojas Leal ◽  
Bernd Kallmünzer ◽  
Klemens Winder ◽  
...  

2005 ◽  
Vol 23 (12) ◽  
pp. 2269-2276 ◽  
Author(s):  
Alberto Zanchetti ◽  
Rita Facchetti ◽  
Gian Carlo Cesana ◽  
Maria Grazia Modena ◽  
Anna Pirrelli ◽  
...  

PEDIATRICS ◽  
1990 ◽  
Vol 86 (6) ◽  
pp. 1006-1007
Author(s):  
EDWARD H. PERRY ◽  
HENRIETTA S. BADA ◽  
JOHN D. DAY ◽  
SHELDON B. KORONES ◽  
KRISTOPHER L. ARHEART ◽  
...  

In Reply.— We appreciate the interest and comments of Drs Puccio and Soliani regarding our article "Blood Pressure Increase, Birth Weight Dependent Stability Boundary and Intraventricular Hemorrhage."1 In response, we address the following points: 1. Although mean blood pressure (BP) values greater than 100 mm Hg were observed in some of our patients, these were quite rare. The mean BP was found to be less than 60 mm Hg 99% of the time. Thus, unless one is recording BPs through long periods and sampling quite often, the brief spikes likely would not be observed.


Author(s):  
Achmad Fachrul Megananda ◽  
Tjipto Suwandi

Introduction: Noise is an unwanted sound heard by the hearers, which can trigger health problems if it continues to be exposed to a certain intensity. One of the health problems that can arise due to noise is the blood pressure increase. This study aims to analyze the influence of noise intensity and age to the blood pressure increase. Methods: This study was an observational study completed with cross-sectional design and analytics. The independent variables in this study were the noise intensity and age, while the dependent variable was the blood pressure. The sample collection of this study applied the total sampling method with 46 employees as respondents, i.e., 29 employees of the heavy-duty shop and 17 employees of the EHS Department in PT. Vale Indonesia. The data analysis was conducted by using the logistic regression statistical test with α-value of 0.05. Results: the noise intensity affected the increase of both systolic blood pressure (significance/p-value=0.00) and diastolic blood pressure (significance/p-value=0.01) with an odds ratio of the increase of systolic blood pressure (Exp (B) =9.75) and the increase of diastolic blood pressure (Exp (B)=5.76). Furthermore, the variable of age does not influence the increase of both systolic (significance/p-value=0.57) and diastolic (significance/p-value=0.41) blood pressures. Conclusion:  the rise of the blood pressure of the employees is affected by the noise intensity factor in the workplace. Keywords: noise intensity, age, blood pressure


Author(s):  
Maciej Abakumow ◽  
◽  
Krzysztof Kowalczuk ◽  

Abstract: Apart from protection from very high altitude or influence of increased gravitational accelerations protective suits sometimes are used for another applications like supporting kinesitherapy. Because of some safety considerations connected with possible cardiovascular system overload and dangerous blood pressure increase we tested if these concerns are valid. Main aim ot presented research performed with participation of healthy volunteers was to confirm that use of High Altitude Protection (HAP) suit is safe in terms of increased cardiovascular.


Hypertension ◽  
2014 ◽  
Vol 64 (suppl_1) ◽  
Author(s):  
Cristiane Aoqui Aoqui ◽  
Stefan Chmielewski ◽  
Uwe Heemann ◽  
Marcus Baumann

Background: Overweight is associated with a high prevalence of hypertension. The mechanisms linking overweight to blood pressure increase remain unclear. We hypothesized that vascular Rho-kinase activation contributes to blood pressure increase in overweight by involving TNF-α and TLR4. Methods: C57/BL6 mice fed a high-fat diet for 2 weeks were used to induce overweight associated blood pressure increase. Additional treatment in overweight and normal weight mice contained Rho-kinase inhibitors (fasudil and pravastatin; n=7/all groups), etanercept and TNFR1 and TLR4 null-mice. Microvascular studies were performed in a wire myograph and arterial blood pressure measured with a carotid catheter. Rho-kinase activity was determined in small mesenteric arteries of all groups. Inflammatory ligands such as TNF-alpha and free fatty acids were determined. Effects of TNF-alpha and TLR4 ligand LPS and palmitic acid on Rho-kinase activity and were determined ex vivo in mesenteric arteries and in in vivo. Results: Overweight mice had higher blood pressure (Delta: 9±2 mmHg) and vasoconstriction as normal weight mice. Small mesenteric arteries of overweight mice had a 50% higher Rho-kinase activity as normal weight mice. Ex vivo treatment with the Rho-kinase inhibitor Y-27632 reversed vasoconstriction of mesenteric arteries of overweight mice to constriction level in normal weight mice. In vivo treatment with the Rho-kinase inhibitor fasudil or pravastatin along with high-fat diet abolished the overweight associated blood pressure increase and enhanced vasoconstriction. TNF-alpha and TLR4 ligand free fatty acid were enhanced in overweight mice. TNF-alpha and TLR4 ligand LPS and palmitic acid increased Rho-kinase activity in mesenteric arteries. Use of etenercept, TNFR1 and TLR4 null-mice in the overweight model prevented blood pressure increase, vasoconstriction and Rho-kinase activation. All described effects were independent of adiposity. Conclusion: These results indicated that in diet-induced overweight vascular Rho-kinase activation is a key element of increased blood pressure and vasoconstriction. Potential activator of Rho-kinase are mediated by inflammatory factors including TLR4 ligands and TNF-alpha.


2020 ◽  
Vol 237 (04) ◽  
pp. 454-457
Author(s):  
Iris Wyssmüller ◽  
Karen Schaal ◽  
Sebastian Wolf ◽  
Martin Zinkernagel

Abstract Background It has previously been shown that the process of anti-vascular endothelial growth factor (VEGF) injections can lead to a significant increase of blood pressure. The aim of this study was to investigate whether this blood pressure increase was reproducible with repeated anti-VEGF injections. Patients and Methods Patients with a systolic blood pressure of ≥ 180 mmHg during previous injections who were scheduled for further injections were asked to participate in this study. Systolic as well as diastolic blood pressure was measured before, during, and after the intravitreal injection process. Results Thirty-nine patients (21 females, 18 males) with a mean age of 75 years (range 34 – 94 years) were included in this extension of the FEAR study. At first, clinical systolic blood pressure rose from an average of 157.3 ± 5.9 mmHg to 175 ± 6.7 mmHg at the time of the injection process (p < 0.01). Diastolic blood pressure rose from an average of 75.72 ± 4.2 mmHg to 84.44 ± 7.3 mmHg (p < 0.13) at the time of the injection process. Overall, the majority of the participants (56%, N = 22) had a systolic blood pressure of ≥ 180 mmHg. Conclusions Our results show that the blood pressure increase occurs persistently during the injection process in some patients. Repeated episodes of severe hypertension may predispose patients to cardiovascular events, especially those with concomitant cardiovascular risk factors.


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