The reliability of cause-of-death coding in The Netherlands

2010 ◽  
Vol 25 (8) ◽  
pp. 531-538 ◽  
Author(s):  
Peter Harteloh ◽  
Kim de Bruin ◽  
Jan Kardaun
2001 ◽  
Vol 33 (2) ◽  
pp. 279-303 ◽  
Author(s):  
FRANS VAN POPPEL ◽  
INEZ JOUNG

This article describes the long-term trends in marital status mortality differences in the Netherlands using a unique dataset relating to the period 1850–1970. Poisson regression analysis was applied to calculate relative mortality risks by marital status. For two periods, cause-of-death by marital status could be used. Clear differences in mortality by marital status were observed, with strongly increasing advantages for married men and women and a relative increase in the mortality of widowed compared with non-married people. Excess mortality among single and formerly married men and women was visible in many cause-of-death categories, and this became more widespread during the last decades of the nineteenth century. Hypotheses are formulated that might explain why married men and women underwent a stronger decrease in mortality up until the end of World War II.


2018 ◽  
Vol 146 (12) ◽  
pp. 1478-1494 ◽  
Author(s):  
Y. Ma ◽  
C. R. Horsburgh ◽  
L. F. White ◽  
H. E. Jenkins

AbstractTuberculosis (TB) is the leading global infectious cause of death. Understanding TB transmission is critical to creating policies and monitoring the disease with the end goal of TB elimination. To our knowledge, there has been no systematic review of key transmission parameters for TB. We carried out a systematic review of the published literature to identify studies estimating either of the two key TB transmission parameters: the serial interval (SI) and the reproductive number. We identified five publications that estimated the SI and 56 publications that estimated the reproductive number. The SI estimates from four studies were: 0.57, 1.42, 1.44 and 1.65 years; the fifth paper presented age-specific estimates ranging from 20 to 30 years (for infants <1 year old) to <5 years (for adults). The reproductive number estimates ranged from 0.24 in the Netherlands (during 1933–2007) to 4.3 in China in 2012. We found a limited number of publications and many high TB burden settings were not represented. Certain features of TB dynamics, such as slow transmission, complicated parameter estimation, require novel methods. Additional efforts to estimate these parameters for TB are needed so that we can monitor and evaluate interventions designed to achieve TB elimination.


1998 ◽  
Vol 79 (03) ◽  
pp. 511-516 ◽  
Author(s):  
Rogier Bertina ◽  
Zandra Holmes ◽  
Caroline Spaargaren ◽  
Joannes van Krieken ◽  
Bert Manten ◽  
...  

SummaryTo investigate whether the factor V Leiden mutation increases the risk of fatal pulmonary emboli, we determined the presence of the factor V Leiden mutation in pathology material from two series of autopsies of patients from the Leiden University Hospital, The Netherlands. The first series consisted of consecutive autopsies in which pulmonary emboli were mentioned in the autopsy report; most patients of this series had major underlying disease. The second series consisted of autopsies in patients younger than age 70 in which pulmonary emboli were the sole cause of death and no major acquired risk factor for venous thrombosis was present. Extraction of DNA was done on newly prepared tissue from archival paraffin blocks. In the first series, the presence of factor V Leiden was determined in 44 patients, 1 of whom carried the mutation (2.3 percent; 95% confidence interval 0.06 to 12.0 percent). This prevalence is not different from the general population prevalence in The Netherlands. In the second series, factor V Leiden could be determined in 30 patients of whom 3 carried the mutation (10 percent; 95% confidence interval 2.1 to 26.5 percent), which would lead to a threefold relative risk. A large number of patients with diverse psychiatric diagnoses was present in the second series (eleven). We conclude that in the presence of severe illness, the factor V Leiden mutation plays no additional role in the development of pulmonary emboli. The relative risk of the very rare fatal pulmonary embolus that is the sole cause of death might also be less than the relative risk for deep-vein thrombosis in carriers of the factor V Leiden mutation.


1995 ◽  
Vol 141 (5) ◽  
pp. 466-475 ◽  
Author(s):  
J. P. Mackenbach ◽  
A. E. Kunst ◽  
H. Lautenbach ◽  
F. Bijlsma ◽  
Y. B. Oei

Author(s):  
Pauline van Beek ◽  
Floris Groenendaal ◽  
Lisa Broeders ◽  
Peter H Dijk ◽  
Koen P Dijkman ◽  
...  

ObjectiveIn the Netherlands, the threshold for offering active treatment for spontaneous birth was lowered from 25+0 to 24+0 weeks’ gestation in 2010. This study aimed to evaluate the impact of guideline implementation on survival and causes and timing of death in the years following implementation.DesignNational cohort study, using data from the Netherlands Perinatal Registry.PatientsThe study population included all 3312 stillborn and live born infants with a gestational age (GA) between 240/7 and 266/7 weeks born between January 2011 and December 2017. Infants with the same GA born between January 2007 and December 2009 (N=1400) were used as the reference group.Main outcome measuresSurvival to discharge, as well as cause and timing of death.ResultsAfter guideline implementation, there was a significant increase in neonatal intensive care unit (NICU) admission rate for live born infants born at 24 weeks’ GA (27%–69%, p<0.001), resulting in increased survival to discharge in 24-week live born infants (13%–34%, p<0.001). Top three causes of in-hospital mortality were necrotising enterocolitis (28%), respiratory distress syndrome (19%) and intraventricular haemorrhage (17%). A significant decrease in cause of death either complicated or caused by respiratory insufficiency was seen over time (34% in 2011–2014 to 23% in 2015–2017, p=0.006).ConclusionsImplementation of the 2010 guideline resulted as expected in increased NICU admissions rate and postnatal survival of infants born at 24 weeks’ GA. In the years after implementation, a shift in cause of death was seen from respiratory insufficiency towards necrotising enterocolitis and sepsis.


1928 ◽  
Vol 28 (1) ◽  
pp. 33-54 ◽  
Author(s):  
J. J. van Loghem

The results of an inquiry into colds and slight influenza—held by me with the obliging collaboration of 7000 informants in the Netherlands from September 1925 till June 1926—do not contradict the hypothesis propounded in the introduction, that various respiratory diseases depend upon a disturbance in the thermo-regulation, in consequence of which the body becomes receptive to commensal infection.In the first place a proportionally equal number of colds was reported at the same time from the various parts of the country during the period of observation, while the increase and the decrease of these colds ran parallel to the falling and rising of the temperature of the air. Further, when calculating the figures from large and from small families, it was proved that the members of small families have no lesser chance of infection than the members of large families.This joint result tells against the contagiousness of colds in the period of observation.The outbreak in September stood alone; the preceding period had not been marked by a low average temperature but by a series of cold nights. During the following month the occurrence of colds was accompanied by the mortality from old age and by the joint mortality from bronchitis and pneumonia.An increased mortality from pneumonia in March–April, linked up with a moderate lowering of the temperature of the air, was not accompanied by “old-age mortality” and only moderately by “colds” it went together however with an increased mortality from influenza. The influenza cases of the inquiry too had increased by that period of time. So the influenza showed itself as a parasitic infection, which takes its own epidemic course. Still the possibility remains of the virus, at times of reigning influenza, having also widely spread among the healthy. At any rate the idea that catching cold reduces resistance to influenza or pneumonia is supported by the data.The opinion obtained from the results of the inquiry, that most colds and pneumonia, as well as many cases in which old age has been given as the cause of death, are founded on a disturbance of the thermo-regulation, has drawn attention to physiological oscillations of the temperature of the body in relation to that of the temperature of the air. As the thermo-regulating system is not able to fix the temperature of the body completely, even under normal conditions, but allows a certain poikilothermia, it is clear that by a considerable drop in the temperature of the air these oscillations of the temperature of the body become so large that they may cause a disturbance.The prophylaxis of the diseases from catching cold will have to be directed towards the application of “passive” prophylactics (for preserving and supplying heat by imminent cooling), as well as of “active” prophylactics (raising the production of heat by the body).


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