Microglial SIRPα Deletion Facilitates Synapse Loss in Preclinical Models of Neurodegeneration

Neuroscience Bulletin ◽

10.1007/s12264-021-00795-5 ◽

2021 ◽

Author(s):

Chongyun Wu ◽

Luodan Yang ◽

Hannah Youngblood ◽

Timon Cheng-Yi Liu ◽

Rui Duan

Keyword(s):

Preclinical Models ◽

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Review for "Anti‐Lingo ‐1 antibody ameliorates spatial memory and synapse loss induced by chronic stress"

10.1002/cne.25038/v1/review2 ◽

2020 ◽

Keyword(s):

Spatial Memory ◽

Chronic Stress ◽

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Review for "Fracture healing in collagen‐related preclinical models of Osteogenesis Imperfecta"

10.1002/jbmr.3979/v1/review1 ◽

2019 ◽

Keyword(s):

Osteogenesis Imperfecta ◽

Fracture Healing ◽

Preclinical Models

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Decision letter for "Fracture healing in collagen‐related preclinical models of Osteogenesis Imperfecta"

10.1002/jbmr.3979/v1/decision1 ◽

2019 ◽

Keyword(s):

Osteogenesis Imperfecta ◽

Fracture Healing ◽

Preclinical Models

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Periosteocytic osteolysis in preclinical models

Bone Abstracts ◽

10.1530/boneabs.3.s1.1 ◽

2014 ◽

Author(s):

Alberta Zallone

Keyword(s):

Preclinical Models

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Acetate metabolism in Multiple Myeloma identifies11C-Acetate PET as a novel strategy to image bone disease and response to treatment in preclinical models

Bone Abstracts ◽

10.1530/boneabs.5.p122 ◽

2016 ◽

Author(s):

Francesca Fontana ◽

Xia Ge ◽

Xinming Su ◽

Jingyu Xiang ◽

Simone Cenci ◽

...

Keyword(s):

Multiple Myeloma ◽

Bone Disease ◽

Response To Treatment ◽

Acetate Metabolism ◽

Preclinical Models ◽

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Creation of a Prospective Monocentric Clinical-biological Database and Establishment of Preclinical Models in Pancreatic Adenocarcinoma (PA)

Case Medical Research ◽

10.31525/ct1-nct04136132 ◽

2019 ◽

Author(s):

Keyword(s):

Pancreatic Adenocarcinoma ◽

Biological Database ◽

Preclinical Models

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Faculty Opinions recommendation of Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1102046.558017 ◽

2008 ◽

Author(s):

John Lisman

Keyword(s):

Signaling Pathway ◽

Glutamate Receptor ◽

Amyloid Beta ◽

Amyloid Beta Protein ◽

Synapse Loss ◽

Dependent Signaling Pathway

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Faculty Opinions recommendation of MF63 [2-(6-chloro-1H-phenanthro[9,10-d]imidazol-2-yl)-isophthalonitrile], a selective microsomal prostaglandin E synthase-1 inhibitor, relieves pyresis and pain in preclinical models of inflammation.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1124203.581345 ◽

2008 ◽

Author(s):

Hanns Ulrich Zeilhofer ◽

Ilja Broll

Keyword(s):

Prostaglandin E ◽

Preclinical Models ◽

Prostaglandin E Synthase ◽

Microsomal Prostaglandin E Synthase

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Faculty Opinions recommendation of Treatment with imatinib prevents fibrosis in different preclinical models of systemic sclerosis and induces regression of established fibrosis.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1148053.605164 ◽

2009 ◽

Author(s):

Jaap van Laar

Keyword(s):

Systemic Sclerosis ◽

Preclinical Models

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Faculty Opinions recommendation of Identification of human triple-negative breast cancer subtypes and preclinical models for selection of targeted therapies.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.13405017.14775138 ◽

2011 ◽

Author(s):

Thomas Egelhoff

Keyword(s):

Breast Cancer ◽

Triple Negative Breast Cancer ◽

Targeted Therapies ◽

Triple Negative ◽

Breast Cancer Subtypes ◽

Preclinical Models ◽

Cancer Subtypes ◽

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