Thyroid stimulating hormone and prolactin secretion after thyrotropin releasing hormone administration to mares: Dose response during anestrus in winter and during estrus in summer

1984 ◽  
Vol 1 (3) ◽  
pp. 263-268 ◽  
Author(s):  
D.L. Thompson ◽  
T.M. Nett
Keyword(s):  
Dose Response ◽  
Thyroid Stimulating Hormone ◽  
Prolactin Secretion ◽  
Thyrotropin Releasing Hormone ◽  
Releasing Hormone ◽  
Hormone Administration ◽  
Stimulating Hormone

scholarly journals The Physiological Role of Thyrotropin-Releasing Hormone in the Regulation of Thyroid-Stimulating Hormone and Prolactin Secretion in the Rat

1978 ◽  
Vol 61 (2) ◽  
pp. 441-448 ◽  
Author(s):  
Arthur R. C. Harris ◽  
Dana Christianson ◽  
M. Susan Smith ◽  
Shih-Lieh Fang ◽  
Lewis E. Braverman ◽  
...  
Keyword(s):  
Physiological Role ◽  
Thyroid Stimulating Hormone ◽  
Prolactin Secretion ◽  
Thyrotropin Releasing Hormone ◽  
Releasing Hormone ◽  
Stimulating Hormone

Thyroid-Stimulating Hormone and Prolactin Responses to Thyrotropin-Releasing Hormone in Common Migraine

Cephalalgia ◽  
1987 ◽  
Vol 7 (4) ◽  
pp. 267-272 ◽  
Author(s):  
Giovanni Murialdo ◽  
Paola Masturzo ◽  
Ugo Filippi ◽  
Diego De Palma ◽  
Daniela Balbi ◽  
...  
Keyword(s):  
Intravenous Administration ◽  
Dose Response ◽  
Thyroid Stimulating Hormone ◽  
Thyrotropin Releasing Hormone ◽  
Healthy Controls ◽  
Response Relationship ◽  
Dose Response Relationship ◽  
Releasing Hormone ◽  
The One ◽  
Stimulating Hormone

Intravenous administration of 50 μg or 200 μg thyrotropin-releasing hormone (TRH) to men with common migraine elicited blunted prolactin (PRL) responses, when compared with healthy controls. The thyroid-stimulating hormone (TSH) response was enhanced after 50 μg TRH in the migraineurs, but not after 200 μg. The physiologic TSH dose-response relationship was abolished in migraine sufferers. The data may be interpreted in the light of dopaminergic and noradrenergic supersensitivity, for PRL and TSH, respectively. The TSH response in migraine differs from the one that occurs in depression.

Thyroid Stimulating Hormone and Growth Hormone Responses to Thyrotropin Releasing Hormone in Anorexia Nervosa

1981 ◽  
Vol 10 (1) ◽  
pp. 51-57 ◽  
Author(s):  
Mark S. Gold ◽  
A. L. C. Pottash ◽  
David M. Martin ◽  
Lawrence B. Finn ◽  
Robert K. Davies
Keyword(s):  
Growth Hormone ◽  
Anorexia Nervosa ◽  
Thyroid Stimulating Hormone ◽  
Thyrotropin Releasing Hormone ◽  
Hormone Release ◽  
Growth Hormone Release ◽  
Hormone Response ◽  
Releasing Hormone ◽  
Hormone Responses ◽  
Stimulating Hormone

Ten female patients who satisfied objective criteria for the diagnosis of anorexia nervosa were given 500 ug of thyrotropin releasing hormone. Thyroid stimulating hormone and growth hormone responses were measured in duplicate by radioimmunoassay. These patients had a low normal Δ thyroid stimulating hormone but a delayed peak response. In addition, these patients had pathological growth hormone release in response to thyrotropin releasing hormone infusion. Both delayed peak thyroid stimulating hormone and growth hormone response to thyrotropin releasing hormone have been reported for patients with hypothalamic disorders.

Some Aspects of Pituitary Function after Neuroadenolysis in Patients with Metastatic Cancer

1981 ◽  
Vol 67 (4) ◽  
pp. 355-359 ◽  
Author(s):  
Nagesh Deshpande ◽  
Guido Moricca ◽  
Franco Saullo ◽  
Luciano Di Martino ◽  
Giok Kwa
Keyword(s):  
Metastatic Cancer ◽  
Thyroid Stimulating Hormone ◽  
Prolactin Secretion ◽  
Pituitary Function ◽  
Releasing Hormone ◽  
Cancer Metastases ◽  
Normal Ranges ◽  
First Time ◽  
Stimulating Hormone

The effects of neuroadenolysis on plasma titres of β-endorphin, β-lipotropin, ACTH, TSH and prolactin have been investigated in five patients with metastatic cancer who responded to the treatment and have been in remission for more than four years and in five others who were undergoing the treatment for the first time for pain due to cancer metastases. β-endorphin, β-lipotropin and ACTH titres were within the normal ranges of values in both categories of patients but post-neuroadenolysis titres of these peptides were higher than those before the treatment. The ability to secrete TSH and prolactin and to respond to thyroid stimulating hormone releasing hormone (TRH) remains intact following the treatment. However, whereas basal TSH titres and response to TRH was lower in the majority of patients, no such effect was observed on prolactin secretion. Plasma titres of prolactin and TSH were below the sensitivity of the method in the five patients who are in remission for more than four years. These preliminary findings suggest that neuroadenolysis probably affects some mechanism(s) associated with the control of β-endorphin, β-lipotropin and ACTH synthesis.

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