Improvement of Soybean Products Through the Response Mechanism Analysis Using Proteomic Technique

This paper studies the tolerance of electromagnetic relay (EMR) under voltage sag and short interruptions on the basis of response mechanism analysis and the extensive tests. First, it introduces the structure of EMR and proposes response mechanism of EMR under voltage sag. Then, a detailed test plan is presented, including the information of test platform, testing condition, EMRs used in test, list of test, test procedure, and the measured waveforms. Magnitude and duration of the sags are not only the characteristics to be considered to investigate EMR’s tolerance. The other factors, which may have significance influence on tolerance of EMR, are considered here, including point-on-wave (POW), phase angle jump (PAJ), harmonic, magnitude variation in pre- and post-sag segments, two-stage sag, and slow recovery sag. Extensive tests results are presented in the form of voltage-tolerance curves (VTCs). Besides magnitude and duration, POW, PAJ, and two-stage sag have a significant influence on the tolerance of EMR. Other factors only have a tiny impact on the tolerance of EMR. The results show that the magnitude tolerance of EMR is 48–74% of Unom, and duration tolerance is 5–28 ms; they are useful for the technical assessment of EMR’s tolerance to voltage sags and short interruptions, and for the economic assessment of the industry process trip due to its disengagement. Test results also benefit for choosing proper EMR and mitigation device in the complicated operating environment.

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Responses of Platelets to Strains of Streptococcus sanguis: Findings in Healthy Subjects, Bernard-Soulier, Glanzmann’s, and Collagen-Unresponsive Patients

Thrombosis and Haemostasis ◽

10.1055/s-0038-1651098 ◽

1987 ◽

Vol 57 (02) ◽

pp. 222-225 ◽

Cited By ~ 19

Author(s):

A H Soberay ◽

M C Herzberg ◽

J D Rudney ◽

H K Nieuwenhuis ◽

J J Sixma ◽

...

Keyword(s):

Platelet Aggregation ◽

Healthy Subjects ◽

Normal Subjects ◽

Membrane Glycoprotein ◽

Response Mechanism ◽

Platelet Response ◽

Induce Platelet Aggregation ◽

Streptococcus Sanguis ◽

Lag Times ◽

Bernard Soulier Syndrome

SummaryThe ability of endocarditis and dental strains of Streptococcus sanguis to induce platelet aggregation in plasma (PRP) from normal subjects were examined and compared to responses of PRP with known platelet membrane glycoprotein (GP) and response defects. S. sanguis strains differed in their ability to induce normal PRPs to aggregate. Strains that induced PRP aggregation in more than 60% of donors were significantly faster agonists (mean lag times to onset of aggregation less than 6 min) than those strains inducing response in PRPs of fewer than 60% of donors.Platelets from patients with Bernard-Soulier syndrome aggregated in response to strains of S. sanguis. In contrast, platelets from patients with Glanzmann’s thrombasthenia and from a patient with a specific defect in response to collagen were unresponsive to S. sanguis. These observations show that GPIb and V are not essential, but GPIIb-IIIa and GPIa are important in the platelet response mechanism to S. sanguis. Indeed, the data suggests that the platelet interaction mechanisms of S. sanguis and collagen may be similar.

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