scholarly journals Acute left heart failure with pulmonary edema during resection of pediatric neuroblastoma: case report

2021 ◽  
Author(s):  
Shan Gao ◽  
Youjing Dong
Keyword(s):  
Heart Failure ◽  
Case Report ◽  
Pulmonary Edema ◽  
Left Heart ◽  
Left Heart Failure

Localized Unilateral Pulmonary Edema: An Unusual Presentation of Left Heart Failure

CHEST Journal ◽  
1971 ◽  
Vol 60 (3) ◽  
pp. 277-280 ◽  
Author(s):  
Om P. Bahl ◽  
G. Charles Oliver ◽  
S. David Rockoff ◽  
Brent M. Parker
Keyword(s):  
Heart Failure ◽  
Pulmonary Edema ◽  
Unusual Presentation ◽  
Left Heart ◽  
Left Heart Failure ◽  
Unilateral Pulmonary Edema

Pulmonary vascular effects of serotonin (5-OH-tryptamine) in dogs: its role in causing pulmonary edema

1959 ◽  
Vol 197 (5) ◽  
pp. 955-958 ◽  
Author(s):  
S. A. Kabins ◽  
C. Molina ◽  
L. N. Katz
Keyword(s):  
Heart Failure ◽  
Pulmonary Artery ◽  
Pulmonary Edema ◽  
Left Heart ◽  
Vascular Effects ◽  
Left Heart Failure ◽  
Rapid Injection ◽  
Anesthetized Dogs ◽  
Group 3 ◽  
Group Ii

Rapid injection of serotonin (0.5–6.5 mg) was made into the pulmonary artery in 22 experiments in 12 open-chest anesthetized dogs. Serotonin caused pulmonary arteriolar and venous constriction in the majority of experiments, shown by an increase in the pressure gradients between the pulmonary artery and pulmonary artery wedge pressures, and between the latter and the left atrial pressure, respectively. Three groups of responses were obtained as far as the production of pulmonary edema is concerned. The first group (3 dogs) showed no pulmonary edema. The second ( group II—6 dogs) showed moderate to severe bilateral pulmonary edema without evidence of left heart failure. The third (3 dogs) also showed severe bilateral pulmonary edema but in the presence of left heart failure. In group II, the evidence suggests that an increase in capillary permeability is responsible, at least in part, for the pulmonary edema found. The similarity of results in group II to those obtained in the formation of bilateral pulmonary edema following unilateral starch embolization, suggests that serotonin could be the initiating factor in the neurohumoral mechanisms involved—or one of them.

scholarly journals Pulmonary Congestion Due to Right and Left Heart Output Mismatching: A Case Report and Literature Review

2021 ◽  
Vol 12 ◽  
Author(s):  
Jing Yuan ◽  
Yongjun Li ◽  
Jie Sun
Keyword(s):  
Heart Failure ◽  
Differential Diagnosis ◽  
Case Report ◽  
Cardiopulmonary Bypass ◽  
Clinical Manifestations ◽  
Circulation Model ◽  
Pathophysiological Mechanism ◽  
Pulmonary Congestion ◽  
Left Heart ◽  
Left Heart Failure

We report a new pulmonary circulation model during cardiopulmonary bypass that is able to cause pulmonary congestion but without left heart failure. This kind of congestion is characterized by right and left heart output mismatching. The pathophysiological mechanism, clinical manifestations, diagnosis, differential diagnosis, and treatment of this pulmonary congestion are reviewed and discussed in the following article.

Pickwick syndrome

1982 ◽  
Vol 63 (3) ◽  
pp. 48-51
Author(s):  
V. I. Zhukov
Keyword(s):  
Heart Failure ◽  
Right Heart Failure ◽  
Cor Pulmonale ◽  
Left Heart ◽  
Obesity Hypoventilation Syndrome ◽  
Right Heart ◽  
Left Heart Failure ◽  
Hypoventilation Syndrome ◽  
Obesity Hypoventilation

Obesity, as you know, is often combined with atherosclerosis, hypertension and their characteristic left-heart failure. Least of all, in our opinion, obesity is associated with cor pulmonale and right-heart failure. "Pickwick syndrome", it is also known as "Ioe syndrome", "cardiopulmonary syndrome of patients with obesity", "obesity-hypoventilation syndrome".

Sign in / Sign up

Export Citation Format

Share Document